The DOI matches its Crossref record; 111 of the 111 references it cites were found in a registry or an index.
The role of osteopontin in inflammatory processes
Open accessAbstract
Osteopontin (OPN) is a matricellular protein that mediates diverse biological functions. OPN is involved in normal physiological processes and is implicated in the pathogenesis of a variety of disease states, including atherosclerosis, glomerulonephritis, cancer, and several chronic inflammatory diseases. Through interactions with several integrins, OPN mediates cell migration, adhesion, and survival in many cell types. OPN also functions as a Th1 cytokine, promotes cell-mediated immune responses, and plays a role in chronic inflammatory and autoimmune diseases. Besides its function in inflammation, OPN is also a regulator of biomineralization and a potent inhibitor of vascular calcification.
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Cites
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The multiple functions and mechanisms of osteopontin
Icer, M. A., & Gezmen-Karadag, M. (2018) · Clinical Biochemistry · doi:10.1016/j.clinbiochem.2018.07.003 · Cited by 606
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Kahles, F., Findeisen, H. M., & Bruemmer, D. (2014) · Molecular Metabolism · doi:10.1016/j.molmet.2014.03.004 · Cited by 408
Current trends in inflammatory and immunomodulatory mediators in sepsis
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Obesity accelerates T cell senescence in murine visceral adipose tissue
Shirakawa, K., Yan, X., Shinmura, K., Endo, J., Kataoka, M., Katsumata, Y., Yamamoto, T., Anzai, A., Isobe, S., Yoshida, N., et al. (2016) · Journal of Clinical Investigation · doi:10.1172/jci88606 · Cited by 271
Body fluid biomarkers in multiple sclerosis
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Metadata
- Authors
- Lund, S. A., Giachelli, C. M., & Scatena, M.
- Venue
- Journal of Cell Communication and Signaling
- Publisher
- Wiley
- Published
- 2009-10-02
- Volume
- 3
- Issue
- 3-4
- Pages
- 311-322
- ISSN
- 1873-9601 (ISSN-L)
- 1873-961X
- OpenAlex
- W2117062936
- Cited by
- 516
- References
- 111
- FWCI
- 8.88
- Type
- journal article
- Language
- en
- Open access
- hybrid
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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