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Bnip3L is induced by p53 under hypoxia, and its knockdown promotes tumor growth
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“Decreased sensitivity to hypoxia in vivo contributes to tumor growth following Bnip3L silencing in U2OS xenografts Discussion We further examined the U2OS-s control, U-11, and pooled U2OS (p3LKD) tumors with Bnip3L silencing to gain insights A high frequency of p53 mutations is associated with a growth advantage in de…”
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Noxa, a BH3-Only Member of the Bcl-2 Family and Candidate Mediator of p53-Induced Apoptosis
Oda, E., Ohki, R., Murasawa, H., Nemoto, J., Shibue, T., Yamashita, T., Tokino, T., Taniguchi, T., & Tanaka, †. N. (2000) · Science · doi:10.1126/science.288.5468.1053 · Cited by 2,112
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Self-citationp53- and Drug-Induced Apoptotic Responses Mediated by BH3-Only Proteins Puma and Noxa
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Cellular Adaptation to Hypoxia through Hypoxia Inducible Factors and beyond
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A Global Map of p53 Transcription-Factor Binding Sites in the Human Genome
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Metadata
- Authors
- Fei, P., Wang, W., Kim, S.-H., Wang, S., Burns, T. F., Sax, J. K., Buzzai, M., Dicker, D. T., McKenna, W. G., Bernhard, E. J., & El-Deiry, W. S.
- Venue
- Cancer Cell
- Publisher
- Elsevier BV
- Published
- 2004-12
- Volume
- 6
- Issue
- 6
- Pages
- 597-609
- ISSN
- 1535-6108 (ISSN-L)
- OpenAlex
- W2067471739
- Cited by
- 231
- References
- 9
- FWCI
- 5.14
- Type
- journal article
- Language
- en
- Open access
- bronze
- Licence
- www.elsevier.com/tdm/userlicense/1.0/ (tdm)
- www.elsevier.com/open-access/userlicense/1.0/ (vor, 3150 d)
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- Fei, P., Wang, W., Kim, S.-H., Wang, S., Burns, T. F., Sax, J. K., Buzzai, M., Dicker, D. T., McKenna, W. G., Bernhard, E. J., & El-Deiry, W. S. (2004). Bnip3L is induced by p53 under hypoxia, and its knockdown promotes tumor growth. Cancer Cell. https://doi.org/10.1016/j.ccr.2004.10.012
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