Student or educator? Sign up with your school email and get 6 months of Pro free.
CoChat
Sign Up
Cited by 39

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Peroxisome Proliferator-Activated Receptor γ Is Essential for the Resolution of Staphylococcus aureus Skin Infections

Thurlow, Lance R., Joshi, Gauri S., Richardson, Anthony R.·2018·Cell Host & Microbe
DOI 10.1016/j.chom.2018.07.001
Open this paper in CoChat — free account

Overview

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Integrity signals

Registry

Crossref · 10.1016/j.chom.2018.07.001

Cites

2 of 2 references verified0 citation errors · 0 not checkable

Statistics

Verification

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Citations

Cites

Show all 2 references

Research works printed but not linked: 35

  1. Abdullah, 2012, Lack of PPARγ in myeloid cells confers resistance to Listeria monocytogenes infection, PLoS One, 7, e37349, 10.1371/journal.pone.0037349 · DOI
  2. Almeida, 2012, PPARγ expression and function in mycobacterial infection: roles in lipid metabolism, immunity, and bacterial killing, PPAR Res., 2012, 1, 10.1155/2012/383829 · DOI
  3. Araújo, 2016, A PPARγ agonist enhances bacterial clearance through neutrophil extracellular trap formation and improves survival in sepsis, Shock, 45, 393, 10.1097/SHK.0000000000000520 · DOI
  4. Bedi, 2016, Enhanced clearance of Pseudomonas aeruginosa by peroxisome proliferator-activated receptor gamma, Infect. Immun., 84, 1975, 10.1128/IAI.00164-16 · DOI
  5. Byles, 2013, The TSC-mTOR pathway regulates macrophage polarization, Nat. Commun., 4, 2834, 10.1038/ncomms3834 · DOI · Paper page
  6. Diep, 2008, Contribution of Panton-Valentine leukocidin in community-associated methicillin-resistant Staphylococcus aureus pathogenesis, PLoS One, 3, e3198, 10.1371/journal.pone.0003198 · DOI · Paper page
  7. Duthie, 1952, Staphylococcal coagulase; mode of action and antigenicity, J. Gen. Microbiol., 6, 95
  8. Eini, 2013, Oxygen deprivation affects the antimicrobial action of LL-37 as determined by microplate real-time kinetic measurements under anaerobic conditions, Anaerobe, 22, 20, 10.1016/j.anaerobe.2013.04.014 · DOI
  9. Eisele, 2013, Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence, Cell Host Microbe, 14, 171, 10.1016/j.chom.2013.07.010 · DOI · Paper page
  10. Fuller, 2011, Identification of a lactate-quinone oxidoreductase in Staphylococcus aureus that is essential for virulence, Front. Cell. Infect. Microbiol., 1, 19, 10.3389/fcimb.2011.00019 · DOI
  11. Gallardo-Soler, 2008, Arginase I induction by modified lipoproteins in macrophages: a peroxisome proliferator-activated receptor-gamma/delta-mediated effect that links lipid metabolism and immunity, Mol. Endocrinol., 22, 1394, 10.1210/me.2007-0525 · DOI
  12. Grosser, 2016, Regulatory requirements for Staphylococcus aureus nitric oxide resistance, J. Bacteriol., 10.1128/JB.00229-16 · DOI
  13. Hammer, 2013, Two heme-dependent terminal oxidases power Staphylococcus aureus organ-specific colonization of the vertebrate host, MBio, 4, 10.1128/mBio.00241-13 · DOI · Paper page
  14. Joshi, 2011, Arginine catabolic mobile element encoded speG abrogates the unique hypersensitivity of Staphylococcus aureus to exogenous polyamines, Mol. Microbiol., 82, 9, 10.1111/j.1365-2958.2011.07809.x · DOI
  15. Linsalata, 2006, Peroxisome proliferator-activated receptor gamma and spermidine/spermine N1-acetyltransferase gene expressions are significantly correlated in human colorectal cancer, BMC Cancer, 6, 191, 10.1186/1471-2407-6-191 · DOI
  16. Malur, 2009, Deletion of PPAR gamma in alveolar macrophages is associated with a Th-1 pulmonary inflammatory response, J. Immunol., 182, 5816, 10.4049/jimmunol.0803504 · DOI
  17. Mandell, 1975, Catalase, superoxide dismutase, and virulence of Staphylococcus aureus. In vitro and in vivo studies with emphasis on staphylococcal–leukocyte interaction, J. Clin. Invest., 55, 561, 10.1172/JCI107963 · DOI · Paper page
  18. McDougal, 2003, Pulsed-field gel electrophoresis typing of oxacillin-resistant Staphylococcus aureus isolates from the United States: establishing a national database, J. Clin. Microbiol., 41, 5113, 10.1128/JCM.41.11.5113-5120.2003 · DOI · Paper page
  19. Mölne, 2000, Role of neutrophil leukocytes in cutaneous infection caused by Staphylococcus aureus, Infect. Immun., 68, 6162, 10.1128/IAI.68.11.6162-6167.2000 · DOI
  20. Odegaard, 2007, Macrophage-specific PPARgamma controls alternative activation and improves insulin resistance, Nature, 447, 1116, 10.1038/nature05894 · DOI · Paper page
  21. Penas, 2015, Treatment in vitro with PPARα and PPARγ ligands drives M1-to-M2 polarization of macrophages from T. cruzi-infected mice, Biochim. Biophys. Acta, 1852, 893, 10.1016/j.bbadis.2014.12.019 · DOI
  22. Peschel, 2001, Staphylococcus aureus resistance to human defensins and evasion of neutrophil killing via the novel virulence factor MprF is based on modification of membrane lipids with l-lysine, J. Exp. Med., 193, 1067, 10.1084/jem.193.9.1067 · DOI · Paper page
  23. Peschel, 1999, Inactivation of the dlt operon in Staphylococcus aureus confers sensitivity to defensins, protegrins, and other antimicrobial peptides, J. Biol. Chem., 274, 8405, 10.1074/jbc.274.13.8405 · DOI · Paper page
  24. Popovich, 2008, Are community-associated methicillin-resistant Staphylococcus aureus (MRSA) strains replacing traditional nosocomial MRSA strains?, Clin. Infect. Dis., 46, 787, 10.1086/528716 · DOI · Paper page
  25. Remels, 2009, PPARgamma inhibits NF-kappaB-dependent transcriptional activation in skeletal muscle, Am. J. Physiol. Endocrinol. Metab., 297, E174, 10.1152/ajpendo.90632.2008 · DOI · Paper page
  26. Ricote, 1998, The peroxisome proliferator-activated receptor-gamma is a negative regulator of macrophage activation, Nature, 391, 79, 10.1038/34178 · DOI · Paper page
  27. Rigamonti, 2008, Regulation of macrophage functions by PPAR-alpha, PPAR-gamma, and LXRs in mice and men, Arterioscler. Thromb. Vasc. Biol., 28, 1050, 10.1161/ATVBAHA.107.158998 · DOI · Paper page
  28. Rosenberger, 2003, Phagocyte sabotage: disruption of macrophage signalling by bacterial pathogens, Nat. Rev. Mol. Cell Biol., 4, 385, 10.1038/nrm1104 · DOI · Paper page
  29. Talan, 2011, Comparison of Staphylococcus aureus from skin and soft-tissue infections in US emergency department patients, 2004 and 2008, Clin. Infect. Dis., 53, 144, 10.1093/cid/cir308 · DOI · Paper page
  30. Thurlow, 2011, Staphylococcus aureus biofilms prevent macrophage phagocytosis and attenuate inflammation in vivo, J. Immunol., 186, 6585, 10.4049/jimmunol.1002794 · DOI · Paper page
  31. Thurlow, 2013, Functional modularity of the arginine catabolic mobile element contributes to the success of USA300 methicillin-resistant Staphylococcus aureus, Cell Host Microbe, 13, 100, 10.1016/j.chom.2012.11.012 · DOI · Paper page
  32. Thurlow, 2012, Virulence strategies of the dominant USA300 lineage of community-associated methicillin-resistant Staphylococcus aureus (CA-MRSA), FEMS Immunol. Med. Microbiol., 65, 5, 10.1111/j.1574-695X.2012.00937.x · DOI · Paper page
  33. Vitko, 2016, Expanded glucose import capability affords Staphylococcus aureus optimized glycolytic flux during infection, MBio, 7, 10.1128/mBio.00296-16 · DOI · Paper page
  34. Vitko, 2015, Glycolytic dependency of high-level nitric oxide resistance and virulence in Staphylococcus aureus, MBio, 6, 10.1128/mBio.00045-15 · DOI · Paper page
  35. Xavier, 2013, PPARγ-mediated increase in glucose availability sustains chronic Brucella abortus infection in alternatively activated macrophages, Cell Host Microbe, 14, 159, 10.1016/j.chom.2013.07.009 · DOI · Paper page

Cited by

See all 39

Metadata

Authors
Thurlow, L. R., Joshi, G. S., & Richardson, A. R.
ORCID
Thurlow, Lance R.
Venue
Cell Host & Microbe
Publisher
Elsevier BV
Published
2018-08
Volume
24
Issue
2
Pages
261-270.e4
ISSN
1931-3128
DOI
10.1016/j.chom.2018.07.001
Cited by
39
References
2
Language
en
Licence
Funders
NIH (R01AI093613, R21AI111707)

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review

How to cite

Cite CoChat for the checks on this page, and the paper for its own findings.

This page
CoChat. (2026). Citation check: Peroxisome Proliferator-Activated Receptor γ Is Essential for the Resolution of Staphylococcus aureus Skin Infections. https://app.cochat.ai/papers/10.1016/j.chom.2018.07.001
The paper
Thurlow, L. R., Joshi, G. S., & Richardson, A. R. (2018). Peroxisome Proliferator-Activated Receptor γ Is Essential for the Resolution of Staphylococcus aureus Skin Infections. Cell Host & Microbe. https://doi.org/10.1016/j.chom.2018.07.001

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.