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Necrostatin-1 and necroptosis inhibition: Pathophysiology and therapeutic implications
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Clinical features of patients infected with 2019 novel coronavirus in Wuhan, China
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Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
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Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase
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Tumor biomarkers for diagnosis, prognosis and targeted therapy
Zhou, Y., Tao, L., Qiu, J., Xu, J., Yang, X., Zhang, Y., Tian, X., Guan, X., Cen, X., & Zhao, Y. (2024) · Signal Transduction and Targeted Therapy · doi:10.1038/s41392-024-01823-2 · Cited by 720
Therapeutic potential of PANoptosis: innate sensors, inflammasomes, and RIPKs in PANoptosomes
Pandeya, A., & Kanneganti, T.-D. (2024) · Trends in Molecular Medicine · doi:10.1016/j.molmed.2023.10.001 · Cited by 303
Cell deaths: Involvement in the pathogenesis and intervention therapy of COVID-19
Li, X., Zhang, Z., Wang, Z., Gutiérrez-Castrellón, P., & Shi, H. (2022) · Signal Transduction and Targeted Therapy · doi:10.1038/s41392-022-01043-6 · Cited by 67
Fan, Z., Ma, H., Li, Y., Wu, Y., Wang, J., Xiong, L., Fang, Z., & Zhang, X. (2022) · Frontiers in Pharmacology · doi:10.3389/fphar.2022.884821 · Cited by 13
Metadata
- Authors
- Cao, L., & Mu, W.
- Venue
- Pharmacological Research
- Publisher
- Elsevier BV
- Published
- 2021-01
- Volume
- 163
- Pages
- 105297
- ISSN
- 1043-6618 (ISSN-L)
- OpenAlex
- W3103916466
- Cited by
- 266
- References
- 198
- FWCI
- 8.02
- Type
- journal article
- Language
- en
- Open access
- green
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- Cao, L., & Mu, W. (2021). Necrostatin-1 and necroptosis inhibition: Pathophysiology and therapeutic implications. Pharmacological Research. https://doi.org/10.1016/j.phrs.2020.105297
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
