The DOI matches its Crossref record; 101 of the 101 references it cites were found in a registry or an index.
Drug Therapy for Alcohol Dependence
Abstract
Alcohol dependence is a chronic disorder that results from a variety of genetic, psychosocial, and environmental factors.1 As defined by the American Psychiatric Association in the Diagnostic and Statistical Manual of Mental Disorders, it is characterized by increased tolerance of the effects of alcohol, impaired control over drinking, and continued drinking despite adverse consequences (Table 1).2 Alcohol dependence affects nearly 10 percent of the population and results in social problems, considerable morbidity and mortality, and high health care costs.3,4 Alcohol dependence is treated by medical, psychological, and social interventions that reduce or eliminate the desire to drink and the . . .
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“Disulfiram blocks the enzyme aldehyde dehydrogenase (ALDH).”
From Disulfiram Efficacy in the Treatment of Alcohol Dependence: A Meta-Analysis (2014) · cites this paper as [8]
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“Substance dependence disorders have proven amenable to intervention, and effective treatment may indeed serve to reduce suicide risk (Fleming et al., 1997; Howard, 2001; Johnson et al., 2003; Sindelar and Fiellin, 2001; Swift, 1999).”
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Kosten, T. A. (2011) · Pharmacology Biochemistry and Behavior · doi:10.1016/j.pbb.2011.02.026 · Cited by 22
“Pharmacological treatments may help obtain this goal (Anton, 2001; Swift, 1999).”
BackgroundPreclinical evaluation of avermectins as novel therapeutic agents for alcohol use disorders
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“Psychosocial intervention alone has very little impact on controlling behavioral symptoms of AUDs, with relapse rated as high as 70% since patient resume to drinking within a year (Finney et al. 1996; Swift 1999).”
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“Each of 4-CQL, DICAQ, 3-FQL, and 4-FQL binds the mu opioid receptor with varying affinities and 4-CQL reduces the anti-nociceptive effects of morphine (de Paulis et al., 2004; Swift, 1999), an action similar to that of opioid antagonists demonstrated to reduce alcohol craving and relapse in alcohol dependence (Swift,…”
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Current and Future Drug Therapies for Alcohol Dependence
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“Current pharmacotherapy for alcohol abuse has had only limited success (Anton et al., 1995; Litten and Allen, 1998; Swift, 1999).”
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“Historically, however, pharmacotherapies were focused mainly on relieving the symptomatology of alcohol withdrawal syndrome; only recently have medications to help achieve and maintain abstinence and control alcohol craving been developed.(72,73) To date, three studies have assessed the efficacy of GHB on craving for…”
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“…enzyme aldehyde dehydrogenase; thus, in the presence of alcohol, elevated concentrations of its metabolite acetaldehyde (Figure 1) will result in the unpleasant disulfiram-ethanol reaction consisting primarily of tachycardia, flushing, nausea, and vomiting (Swift, 1999; Zindel and Kranzler, 2014).”
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“Disulfiram Although disulfiram was approved by the FDA in 1951 for the treatment of alcohol dependence (De Sousa, 2010), it is not often prescribed clinically (Swift, 1999), and up to 80% of patients discontinue disulfiram treatment (Fuller et al., 1986).”
BackgroundThe development of acamprosate as a treatment against alcohol relapse
Kufahl, P. R., Watterson, L. R., & Olive, M. F. (2014) · Expert Opinion on Drug Discovery · doi:10.1517/17460441.2014.960840 · Cited by 19
PARADIGM TO TEST A DRUG-INDUCED AVERSION TO ETHANOL
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FDA-approved disulfiram as a novel treatment for aggressive leukemia
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“The drug targets the enzyme acetaldehyde dehydrogenase, inhibiting the normal break-down of acetaldehyde downstream of alcohol catabolism by the liver, which causes a ‘hangover’ effect immediately following alcohol consumption [9].”
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“Taking all these studies together, they have suggested “new vistas” (Kranzler, 2000) on the treatment of alcoholism, getting over the “scientific frustation” (Johnson, 2000) due to previous trials showing the lack of efficacy of serotonergic drugs in alcohol-dependent patients (Swift, 1999).”
BackgroundComparison of Cyanamide and Disulfiram in Effects on Liver Function
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“However, it is disturbing that approximately 70% of patients relapse within the first year of treatment [5,6].”
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“Food and Drug Administration has approved disulfiram, naltrexone, acamprosate, and topiramate for treatment of alcohol dependence.(104) There is growing evidence that patients with alcohol dependence who carry a particular variant of an opioid receptor gene are more likely to respond to naltrexone, raising the possibi…”
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“, fluoxetine, buspirone) might be comorbidity of a mood or anxiety disorder, although treatment of the underlying psychiatric problems may thus have an important beneficial secondary effect in reducing alcohol consumption (Swift, 1999; Mann, 2004).”
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“Of these, bromocriptine appears to be ineffective, while tiapride, a D2-receptor antagonist not currently available in the United States, appeared to lower relapse rates and use of health care services[ 24 ].”
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“Studies have shown that a reduced neurotransmission in GABAA and an enhanced neurotransmission in glutamatergic pathways results in an imbalance between inhibitory and excitatory neurotransmitters [3], which leads to the nervous system hyperactivity [4].”
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Li, Y., Shen, J., Fang, M., Huang, X. B., Yan, H., Jin, Y., Li, J., & Li, X. (2018) · Biomedicine & Pharmacotherapy · doi:10.1016/j.biopha.2018.09.123 · Cited by 9
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“We assessed participants by using the following scales 15 days after disappearance of withdrawal symptoms, on the basis of the observations that detoxification requires an average of 7–10 days for alcohol addiction, and that anxiety and mood disorders occur more frequently during the initial 2 weeks after ceasing the…”
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Opioid Antagonists and Alcoholism Treatment
Swift, R. M. (2000) · CNS Spectrums · doi:10.1017/s1092852900012815 · Cited by 8
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Disulfiram induced peripheral neuropathy: between the devil and the deep blue sea
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“Drug interactions are reported between disulfiram and other drugs using the cytochrome P450 enzyme system.(2) Dose adjustment of these medications may be required to avoid adverse effects.”
MethodA comparative study using Disulfiram and Naltrexone in alcohol-dependent adolescents
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“It does not seem to be sufficiently recognized that DSF generally has no specific effect unless it is monitored and supervised by family members or professionals (Barth & Malcolm, 2010; De Sousa, 2010; Fuller & Gordis, 2004; Krampe & Ehrenreich, 2010; Suh et al., 2006; Swift, 1999).”
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“These components focus on four core areas (1) identification and modification of alcohol expectancies, (2) increasing drinking refusal-self efficacy skills, (3) developing more effective coping strategies, and (4) teaching problem-solving skills.”
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“Research evidence confirms that the personal and societal impact of AUD can be mitigated through treatments, which include psychological, social, and pharmaceutical interventions aimed at reducing alcohol consumption and its associated consequences [2].”
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Fernández, C. G., González-Castro, A., Sánchez, M. L., & Borregán, J. C. R. (2009) · Medicina Clínica · doi:10.1016/j.medcli.2008.05.005 · Cited by 3
Tratamiento farmacológico de la dependencia alcohólica
Ponce, G., Jiménez‐Arriero, M. Á., & Rubio, G. (2003) · Trastornos Adictivos · doi:10.1016/s1575-0973(03)70103-2 · Cited by 3
Child and Adolescent Alcohol Use and Abuse
Hilarski, C. (2004) · Journal of Evidence-Based Social Work · doi:10.1300/j394v01n01_05 · Cited by 3
“Interventions with mixed empirical outcomes are relapse prevention (Carroll, 1996), behavioral self-control, antabuse (Garbutt, West, Carey, Lohr, & Crews, 1999; Swift, 1999), aversion therapy (Swift, 1999), and Naltrexone (Swift, 1999).”
BackgroundBrust, J. C. M. (2004) · Elsevier eBooks · doi:10.1016/b978-0-7506-7313-6.50016-7 · Cited by 3
Ait‐Daoud, N., & Johnson, B. A. (2002) · Addictive Disorders & Their Treatment · doi:10.1097/00132576-200209000-00001 · Cited by 3
“First, up to one half of alcoholics relapse shortly after detoxification, psychosocial, or behavioral treatment.(1,2) Second, advances in the neurosciences have implicated several target neurotransmitter systems, such as those within the mesocorticolimbic pathway that mediate some of alcohol’s rewarding effects associ…”
BackgroundComorbidity of Anxiety Disorders in Substance Abuse
Mueller, T., & Swift, R. M. (2001) · Informa Healthcare eBooks · doi:10.1201/9780203908075.pt2 · Cited by 3
Self-citationTemel, Y., Hescham, S., Melse, M., & Visser-Vandewalle, V. E. R. M. (2015) · Wiley eBooks · doi:10.1002/9781118801086.ch16 · Cited by 3
Lohse, M. J., Lorenzen, A., & Müller-Oerlinghausen, B. (2004) · Springer eBooks · doi:10.1007/978-3-642-18512-0_41 · Cited by 3
Advances in research on medications for the treatment of alcohol use disorders: A review
Chen, Y., Zong, L., Zhao, Q., & Liu, C. (2024) · Medicine · doi:10.1097/md.0000000000040045 · Cited by 2
“[12] Clinical research and experimental results indicate that although disulfiram can induce discomfort following alcohol intake, its effect on inhibiting relapse behavior in patients is inconsistent.”
ResultAdvances in the Treatment of Alcohol Dependence
Rosenthal, R. N., Brady, K. T., Levounis, P., & Willenbring, M. L. (2007) · The Journal of Clinical Psychiatry · doi:10.4088/jcp.v68n0721 · Cited by 2
“22 Therefore, Dr. Rosenthal emphasized that patients must be aware of the ingredi-ents of products that they come in contact with to avoid accidental exposure.”
BackgroundInfluentialMartinotti, G., Festa, G. M., Ceci, F., Muzio, I. D., Callea, A., Capparelli, A., Bonifaci, L., Chillemi, E., Pepe, M., Pettorruso, M., et al. (2022) · Alcohol and Alcoholism · doi:10.1093/alcalc/agac051 · Cited by 2
Sacroileítis piógena complicada con absceso de psoas y neumonía
Miranda, I. M. A., Ruiz, J. M. Q., de Mesa, C. L., & Fernández, M. L. G.-A. (2005) · Anales de Medicina Interna · doi:10.4321/s0212-71992005001200018 · Cited by 2
Borini, P., Guimarães, R. C., & Borini, S. B. (2003) · Revista do Hospital das Clínicas · doi:10.1590/s0041-87812003000300004 · Cited by 2
Are Alcohol Anti-relapsing and Alcohol Withdrawal Drugs Useful in Cannabinoid Users?
Kleczkowska, P., Smaga, I., Filip, M., & Bujalska‐Zadrożny, M. (2016) · Neurotoxicity Research · doi:10.1007/s12640-016-9655-z · Cited by 2
“As reported, patients remained abstinent from alcohol for almost 1 year after acamprosate treatment in comparison to subjects receiving placebo (Swift 1999).”
ResultThe pharmacological understandings of alcohol use and misuse
Rose, A. K., & Jones, A. (2021) · Elsevier eBooks · doi:10.1016/b978-0-12-816720-5.00007-4 · Cited by 2
Occupational Health and Public Health : Lessons from the Past - Challenges for the Future
Nelson, M. C. (2006) · Cited by 2
Desintoxicación ambulatoria de alcohol: una opción segura
Nogueira, J. M., Freijeiro, C. G., Ben, B. G., & Domínguez, N. D. (2005) · Adicciones · doi:10.20882/adicciones.384 · Cited by 2
Jones, A. W. (2004) · Humana Press eBooks · doi:10.1007/978-1-59259-654-6_12 · Cited by 2
Lohse, M. J., Lorenzen, A., & Müller-Oerlinghausen, B. (2006) · Springer eBooks · doi:10.1007/3-540-30505-x_43 · Cited by 2
Metadata
- Authors
- Swift, R. M.
- Affiliations
- Brown University (US)
- Corresponding author
- Swift, Robert M.
- ORCID
- Swift, Robert M.
- Venue
- New England Journal of Medicine
- Publisher
- Massachusetts Medical Society
- Published
- 1999-05-13
- Volume
- 340
- Issue
- 19
- Pages
- 1482-1490
- ISSN
- 0028-4793 (ISSN-L)
- 1533-4406
- OpenAlex
- W1984819772
- Cited by
- 255
- References
- 97
- Citations by year
- 2025: 3 · 2024: 7 · 2022: 7 · 2021: 2 · 2020: 5 · 2019: 4 · 2018: 11 · 2016: 5 · 2015: 9 · 2014: 7 · 2013: 10 · 2012: 5
- FWCI
- 3.88
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- New England Journal of Medicine (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Epidemiology › Substance Abuse Treatment and Outcomes
- Health Sciences › Medicine › Pathology and Forensic Medicine › Alcohol Consumption and Health Effects
- Health Sciences › Medicine › Clinical Neurology › Alcoholism and Thiamine Deficiency
- Keywords
- alcoholism, pharmacotherapy, alcohol tolerance, impaired control, DSM-IV diagnostic criteria, health care costs
- MeSH terms
- Alcohol Deterrents, Ethanol, Alcoholism, Antidepressive Agents, Mental Disorders, Humans, Narcotic Antagonists, Receptors, Neurotransmitter, Dopamine Antagonists
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