Macrophage proliferation and apoptosis in atherosclerosis

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Macrophages in the Pathogenesis of Atherosclerosis

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Mediators of endoplasmic reticulum stress-induced apoptosis

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IRE1-mediated unconventional mRNA splicing and S2P-mediated ATF6 cleavage merge to regulate XBP1 in signaling the unfolded protein responsePreprint

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Cell proliferation in human coronary arteries.

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Macrophage plasticity in experimental atherosclerosis

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Reducing endoplasmic reticulum stress through a macrophage lipid chaperone alleviates atherosclerosis

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Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways

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Increased Endoplasmic Reticulum Stress in Atherosclerotic Plaques Associated With Acute Coronary Syndrome

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Reduced Apoptosis and Plaque Necrosis in Advanced Atherosclerotic Lesions of Apoe−/− and Ldlr−/− Mice Lacking CHOP

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The Unfolded Protein Response Is an Important Regulator of Inflammatory Genes in Endothelial Cells

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Atorvastatin Inhibits Hypercholesterolemia-Induced Cellular Proliferation and Bone Matrix Production in the Rabbit Aortic Valve

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Macrophage and smooth muscle cell proliferation in atherosclerotic lesions of WHHL and comparably hypercholesterolemic fat-fed rabbits

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A role for the apoptosis inhibitory factor AIM/Spα/Api6 in atherosclerosis development

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The absence of p53 accelerates atherosclerosis by increasing cell proliferation in vivo

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Phagocytosis in atherosclerosis: Molecular mechanisms and implications for plaque progression and stability

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Histopathology of In-Stent Restenosis in Patients With Peripheral Artery Disease

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Loss of SR-A and CD36 Activity Reduces Atherosclerotic Lesion Complexity Without Abrogating Foam Cell Formation in Hyperlipidemic Mice

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Macrophage Apoptosis Exerts Divergent Effects on Atherogenesis as a Function of Lesion Stage

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IL-10–producing macrophages preferentially clear early apoptotic cells

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Reduced Macrophage Apoptosis Is Associated With Accelerated Atherosclerosis in Low-Density Lipoprotein Receptor-Null Mice

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Molecular imaging and therapy of atherosclerosis with targeted nanoparticles

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Recent Studies of the Human Chromosome 9p21 Locus, Which Is Associated With Atherosclerosis in Human Populations

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Endogenous p53 Protects Vascular Smooth Muscle Cells From Apoptosis and Reduces Atherosclerosis in ApoE Knockout Mice

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Oxidized LDL-Induced Injury and Apoptosis in Atherosclerosis Potential Roles for Oxysterols

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Combinatorial pattern recognition receptor signaling alters the balance of life and death in macrophages

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Macrophage p53 Deficiency Leads to Enhanced Atherosclerosis in APOE*3-Leiden Transgenic Mice

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GM-CSF regulates intimal cell proliferation in nascent atherosclerotic lesions

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Interleukin‐4 and Dexamethasone Counterregulate Extracellular Matrix Remodelling and Phagocytosis in Type‐2 Macrophages

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Active proliferation of different cell types, including lymphocytes, in human atherosclerotic plaques.

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Regression of Atherosclerosis

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Expression of Chr9p21 genes CDKN2B (p15), CDKN2A (p16, p14) and MTAP in human atherosclerotic plaque

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Signal Transducer and Activator of Transcription-1 Is Critical for Apoptosis in Macrophages Subjected to Endoplasmic Reticulum Stress In Vitro and in Advanced Atherosclerotic Lesions In Vivo

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Oxidized LDL Can Induce Macrophage Survival, DNA Synthesis, and Enhanced Proliferative Response to CSF-1 and GM-CSF

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Novel pharmacological strategies for driving inflammatory cell apoptosis and enhancing the resolution of inflammation

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p16INK4a deficiency promotes IL-4–induced polarization and inhibits proinflammatory signaling in macrophages

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Macrophage-Specific p53 Expression Plays a Crucial Role in Atherosclerosis Development and Plaque Remodeling

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Macrophage EP4 Deficiency Increases Apoptosis and Suppresses Early Atherosclerosis

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Granulocyte Colony-Stimulating Factor and Granulocyte Macrophage Colony-Stimulating Factor Exacerbate Atherosclerosis in Apolipoprotein E–Deficient Mice

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p19ARFDeficiency Reduces Macrophage and Vascular Smooth Muscle Cell Apoptosis and Aggravates Atherosclerosis

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Two Intracellular Signaling Pathways for Activation of Protein Kinase C Are Involved in Oxidized Low-Density Lipoprotein–Induced Macrophage Growth

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Control of cell proliferation in atherosclerosis: insights from animal models and human studies

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Hyperlipidemia in Concert With Hyperglycemia Stimulates the Proliferation of Macrophages in Atherosclerotic Lesions

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Statins Suppress Oxidized Low Density Lipoprotein-induced Macrophage Proliferation by Inactivation of the Small G Protein-p38 MAPK Pathway

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Granulocyte Macrophage Colony-Stimulating Factor Regulates Dendritic Cell Content of Atherosclerotic Lesions

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The growth suppressor p27 Kip1 protects against diet‐induced atherosclerosis

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GM-CSF Deficiency Reduces Macrophage PPAR-γ Expression and Aggravates Atherosclerosis in ApoE-Deficient Mice

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Granulocyte-Macrophage Colony–Stimulating Factor Prevents the Progression of Atherosclerosis via Changes in the Cellular and Extracellular Composition of Atherosclerotic Lesions in Watanabe Heritable Hyperlipidemic Rabbits

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M-CSF Accelerates Neointimal Formation in the Early Phase After Vascular Injury in Mice

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Apoptosis and Efferocytosis in Mouse Models of Atherosclerosis

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<i>Cdkn2a</i> Is an Atherosclerosis Modifier Locus That Regulates Monocyte/Macrophage Proliferation

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Induction of Murine Macrophage Growth by Oxidized Low Density Lipoprotein Is Mediated by Granulocyte Macrophage Colony-stimulating Factor

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A glimpse on the phenomenon of macrophage polarization during atherosclerosis

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Extracellular signal-regulated kinase and p38 mitogen-activated protein kinase mediate macrophage proliferation induced by oxidized low-density lipoprotein

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Absence of p21 Waf1/Cip1/Sdi1 Modulates Macrophage Differentiation and Inflammatory Response and Protects Against Atherosclerosis

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C-reactive protein induces M-CSF release and macrophage proliferation

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Macrophage colony stimulating factor prevents the progression of atherosclerosis in Watanabe heritable hyperlipidemic rabbits

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Clinical significance of increased plasma concentration of macrophage colony–stimulating factor in patients with angina pectoris

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Foam cells of the rabbit atherosclerotic plaque arrested in metaphase by colchicine show a macrophage phenotype

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Unsaturated FAs prevent palmitate-induced LOX-1 induction via inhibition of ER stress in macrophages

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Selective inactivation of p27Kip1 in hematopoietic progenitor cells increases neointimal macrophage proliferation and accelerates atherosclerosis

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Activation of AMP-activated Protein Kinase Suppresses Oxidized Low-density Lipoprotein-induced Macrophage Proliferation

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Atheroma stabilizing effects of simvastatin due to depression of macrophages or lipid accumulation in the atheromatous plaques of coronary plaque-prone WHHL rabbits

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M2 macrophages exhibit higher sensitivity to oxLDL-induced lipotoxicity than other monocyte/macrophage subtypes

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Increased p53 gene dosage reduces neointimal thickening induced by mechanical injury but has no effect on native atherosclerosis

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Molecular Imaging of Macrophage Cell Death for the Assessment of Plaque Vulnerability

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Macrophage p53 controls macrophage death in atherosclerotic lesions of apolipoprotein E deficient mice

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Macrophage retinoblastoma deficiency leads to enhanced atherosclerosis development in ApoE‐deficient mice

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