The DOI matches its Crossref record; 240 of the 240 references it cites were found in a registry or an index.
The Response-to-Retention Hypothesis of Early Atherogenesis
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“The retention of lipoproteins in the arterial extracellular matrix is a critical step in atherogenesis in which lipoproteins with higher affinity for arterial proteoglycans are more atherogenic (55, 56).”
From Role of the N- and C-Terminal Domains in Binding of Apolipoprotein E Isoforms to Heparan Sulfate and Dermatan Sulfate: A Surface Plasmon Resonance Study (2008) · cites this paper as [55]
In the citing paper
“The ECM has a central role in initiation of atherosclerosis, primarily through the interaction between the negatively charged side chains of proteoglycans (particularly chondroitin sulphate of biglycan and versican and heparin sulphate of perlecan 110 ) with positively charged apolipoproteins (especially apolipoprotein B) , which leads to the retention of plasma-derived lipoproteins 101,111 - as”
From Vascular smooth muscle cells in atherosclerosis (2019) · cites this paper as [113]
In the citing paper
“the interaction of apoB-containing lipoproteins (the non-HDL lipoproteins) with intimal extracellular proteoglycans (referred to as retention)1,43,44 inhibits the movement of the lipoproteins, thereby resulting in much higher subendothelial concentrations of these lipoproteins in the intima than in any other tissue45.”
From Inflammation and its resolution in atherosclerosis: mediators and therapeutic opportunities (2019) · cites this paper as [1]
In the citing paper
“Atherosclerosis is a disease characterized by low-grade, chronic inflammation of the arterial wall and is triggered by the subendothelial retention of plasma-derived, apolipoprotein B (apoB)-containing lipoproteins in the affected inner layer of the arterial wall, the intima1–3.”
From Inflammation and its resolution in atherosclerosis: mediators and therapeutic opportunities (2019) · cites this paper as [1]
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Metadata
- Authors
- Williams, K. J., & Tabas, I.
- Affiliations
- Thomas Jefferson University (US)
- Venue
- Arteriosclerosis, Thrombosis, and Vascular Biology
- Published
- 1995-05
- Volume
- 15
- Issue
- 5
- Pages
- 551-561
- ISSN
- 1079-5642 (ISSN-L)
- 1524-4636
- PMC
- PMC2924812
- OpenAlex
- W1968171440
- Cited by
- 1,413
- References
- 240
- Citations by year
- 2026: 27 · 2025: 34 · 2024: 37 · 2023: 40 · 2022: 42 · 2021: 39 · 2020: 55 · 2019: 47 · 2018: 42 · 2017: 41 · 2016: 59 · 2015: 73 · 2014: 60 · 2013: 56 · 2012: 60
- FWCI
- 12.35
- Type
- journal article
- Language
- en
- Open access
- green
- www.ncbi.nlm.nih.gov
- Locations
- Arteriosclerosis Thrombosis and Vascular Biology (publishedVersion)
- PubMed (publishedVersion)
- PubMed Central (acceptedVersion)
- Funders
- American Heart Association
- National Institutes of Health (5p50hl021006-17, 5r01hl039703-08)
- Genentech
- National Heart, Lung, and Blood Institute (HL 38956, HL-39703, HL 21006, R01HL038956)
- Topics
- Life Sciences › Immunology and Microbiology › Immunology › Atherosclerosis and Cardiovascular Diseases
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Biochemistry › Cholesterol and Lipid Metabolism
- Health Sciences › Medicine › Physiology (medical) › Nitric Oxide and Endothelin Effects
- Keywords
- atherosclerosis initiation, plaque retention, endothelial dysfunction
- MeSH terms
- Animals, Arteries, Arteriosclerosis, Humans, Lipoproteins, LDL, Models, Biological, Proteoglycans
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
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