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Mechanisms of Osteoporosis in Adult and Adolescent Women with Anorexia Nervosa*

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Thyroid hormone regulates TRH biosynthesis in the paraventricular nucleus of the rat hypothalamus

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Effects of Starvation in Rats on Serum Levels of Follicle Stimulating Hormone, Luteinizing Hormone, Thyrotropin, Growth Hormone and Prolactin; Response to LH-Releasing Hormone and Thyrotropin-Releasing Hormone

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Effect of dietary composition on fasting-induced changes in serum thyroid hormones and thyrotropin

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The Production and Metabolism of 3,5,3′-Triiodothyronine and 3,3′,5′-Triiodothyronine in Normal and Fasting Subjects*

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Effect of Starvation on the Production and Metabolism of Thyroxine and Triiodothyronine in Euthyroid Obese Patients

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Triiodothyronine exerts direct cell-specific regulation of thyrotropin-releasing hormone gene expression in the hypothalamic paraventricular nucleus

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Decreased serum triiodothyronine in starving rats is due primarily to diminished thyroidal secretion of thyroxine.

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Release of thyrotropin releasing hormone into hypophysial portal blood is high relative to other neuropeptides and may be related to prolactin secretion

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Nutritional Effects on Thyroid and Catecholamine Metabolism

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Pro-TRH Gene Expression and Precursor Peptides in Rat Brain:.

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Nature of suppressed TSH secretion during undernutrition: Effect of fasting and refeeding on TSH responses to prolonged TRH infusions

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The Effect of Caloric Deprivation upon Thyroid Function-in the Neonatal Rat*

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Studies of growth hormone (GH), Thyrotropin (TSH) and prolactin (PRL) secretion in anorexia nervosa

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Quantitative in-situ hybridization histochemistry in the rat pituitary gland: effect of bromocriptine on prolactin and pro-opiomelanocortin gene expression

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RADIOIMMUNOASSAY OF THYROTROPHIN RELEASING HORMONE

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Importance of Maintaining Species Homology in Thyroid Hormone Radioimmunoassays: Modification of Ηuman’ Radioimmunoassay Kits for Use with Rat Samples

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Comparison between starvation and consumption of a high protein diet: plasma insulin and glucagon and hepatic activities of gluconeogenic enzymes during the first 24 hours.

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Hypothalamic portal blood immunoreactive TRH in the rat: lack of effect of hypothyroidism and thyroid hormone treatment

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Serum thyrotropin (TSH) responses to thyrotropin-releasing hormone (TRH) in patients with anorexia nervosa and bulimia: Influence of changes in body weight and eating disorders

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Thyrotropin Secretion in Starved Rats is Enhanced by Somatostatin Antiserum

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Thyroxine, renal beta-adrenergic receptors, and dipsogenesis in food-deprived rats

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Leptin Signaling in the Central Nervous System and the Periphery

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Leptin Prevents Fasting-Induced Suppression of Prothyrotropin-Releasing Hormone Messenger Ribonucleic Acid in Neurons of the Hypothalamic Paraventricular Nucleus*

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Dangerous dogmas in medicine: the nonthyroidal illness syndrome

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Minireview: From Anorexia to Obesity—The Yin and Yang of Body Weight Control

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Transcriptional regulation of the thyrotropin-releasing hormone gene by leptin and melanocortin signaling

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The TRH neuron: a hypothalamic integrator of energy metabolism

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α-Melanocyte-stimulating hormone is contained in nerve terminals innervating thyrotropin-releasing hormone-synthesizing neurons in the hypothalamic paraventricular nucleus and prevents fasting-induced suppression of prothyrotropin-releasing hormone gene expression

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The central melanocortin system affects the hypothalamo-pituitary thyroid axis and may mediate the effect of leptin

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The Role of the Thyroid Axis in Fish

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Leptin, nutrition, and the thyroid: the why, the wherefore, and the wiring

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Synchronicity of Frequently Sampled Thyrotropin (TSH) and Leptin Concentrations in Healthy Adults and Leptin-Deficient Subjects: Evidence for Possible Partial TSH Regulation by Leptin in Humans

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The Nonthyroidal Illness Syndrome

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Leptin Regulates Prothyrotropin-releasing Hormone Biosynthesis

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Regulation of the hypothalamic Thyrotropin Releasing Hormone (TRH) neuron by neuronal and peripheral inputs

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The Biology of pro-Thyrotropin-Releasing Hormone-Derived Peptides

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Non-thyroidal illness syndrome is a manifestation of hypothalamic-pituitary dysfunction, and in view of current evidence, should be treated with appropriate replacement therapies

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Role of leptin in energy expenditure: the hypothalamic perspective

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Neuropeptide Y has a central inhibitory action on the hypothalamic-pituitary-thyroid axis

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Association of cocaine- and amphetamine-regulated transcript-immunoreactive elements with thyrotropin-releasing hormone-synthesizing neurons in the hypothalamic paraventricular nucleus and its role in the regulation of the hypothalamic-pituitary-thyroid axis during fasting

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Non-invasive measurement of thyroid hormone in feces of a diverse array of avian and mammalian species

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Repeated ICV administration of oxyntomodulin causes a greater reduction in body weight gain than in pair-fed rats

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The Arcuate Nucleus Is the Major Source for Neuropeptide Y-Innervation of Thyrotropin-Releasing Hormone Neurons in the Hypothalamic Paraventricular Nucleus ¹

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Leptin Signaling Targets the Thyrotropin-Releasing Hormone Gene Promoterin Vivo

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Arcuate nucleus ablation prevents fasting-induced suppression of ProTRH mRNA in the hypothalamic paraventricular nucleus

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Agouti-related protein containing nerve terminals innervate thyrotropin-releasing hormone neurons in the hypothalamic paraventricular nucleus

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Changes in adrenal status affect hypothalamic thyrotropin-releasing hormone gene expression in parallel with corticotropin-releasing hormone

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60 YEARS OF NEUROENDOCRINOLOGY: TRH, the first hypophysiotropic releasing hormone isolated: control of the pituitary–thyroid axis

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Role of Thyroid Hormone Deiodination in the Hypothalamus

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NPY and MC4R signaling regulate thyroid hormone levels during fasting through both central and peripheral pathways

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Hypophysiotropic Thyrotropin-Releasing Hormone-Synthesizing Neurons in the Human Hypothalamus Are Innervated by Neuropeptide Y, Agouti-Related Protein, and α-Melanocyte-Stimulating Hormone1

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60 YEARS OF NEUROENDOCRINOLOGY: TRH, the first hypophysiotropic releasing hormone isolated: control of the pituitary–thyroid axis

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Glucocorticoids are Required for Food Deprivation‐Induced Increases in Hypothalamic Neuropeptide Y Expression

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