The DOI matches its Crossref record; 11 of the 11 references it cites were found in a registry or an index.
The Role of Prolactin in Mammary Carcinoma
Open accessAbstract
The contribution of prolactin (PRL) to the pathogenesis and progression of human breast cancer at the cellular, transgenic, and epidemiological levels is increasingly appreciated. Acting at the endocrine and autocrine/paracrine levels, PRL functions to stimulate the growth and motility of human breast cancer cells. The actions of this ligand are mediated by at least six recognized PRL receptor isoforms found on, or secreted by, human breast epithelium. The PRL/PRL receptor complex associates with and activates several signaling networks that are shared with other members of the cytokine receptor superfamily. Coupled with the recently identified intranuclear function of PRL, these networks are integrated into the in vitro and in vivo actions induced by ligand. These findings indicate that antagonists of PRL/PRL receptor interaction or PRL receptor-associated signal transduction may be of considerable utility in the treatment of human breast cancer.
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Blume-Jensen, P., & Hunter, T. (2001) · Nature · doi:10.1038/35077225 · Cited by 3,738
Wen, Z., Zhong, Z., & Darnell, J. E. (1995) · Cell · doi:10.1016/0092-8674(95)90311-9 · Cited by 1,974
Bowman, T., Garcia, R., Turkson, J., & Jove, R. (2000) · Oncogene · doi:10.1038/sj.onc.1203527 · Cited by 1,743
The role of STATs in transcriptional control and their impact on cellular function
Bromberg, J., & Darnell, J. E. (2000) · Oncogene · doi:10.1038/sj.onc.1203476 · Cited by 1,201
Specific Inhibition of Stat3 Signal Transduction by PIAS3
Chung, C. D., Liao, J., Liu, B., Rao, X., Jay, P., Berta, P., & Shuai, K. (1997) · Science · doi:10.1126/science.278.5344.1803 · Cited by 980
Specific protection against breast cancers by cyclin D1 ablation
Yu, Q., Geng, Y., & Sicinski, P. (2001) · Nature · doi:10.1038/35082500 · Cited by 934
Serine phosphorylation of STATs
Decker, T., & Kovarik, P. (2000) · Oncogene · doi:10.1038/sj.onc.1203481 · Cited by 887
Kirchhausen, T. (2000) · Annual Review of Biochemistry · doi:10.1146/annurev.biochem.69.1.699 · Cited by 560
SOCS: physiological suppressors of cytokine signaling
Krebs, D. L., & Hilton, D. J. (2000) · Journal of Cell Science · doi:10.1242/jcs.113.16.2813 · Cited by 463
Mammary gland involution is delayed by activated Akt in transgenic mice
Schwertfeger, K. L., Richert, M. M., & Anderson, S. M. (2001) · Molecular Endocrinology · doi:10.1210/mend.15.6.0663 · Cited by 169
Cited by
The influence of bio-behavioural factors on tumour biology: pathways and mechanisms
Antoni, M. H., Lutgendorf, S. K., Cole, S. W., Dhabhar, F. S., Sephton, S. E., McDonald, P. G., Stefanek, M., & Sood, A. K. (2006) · Nature Reviews Cancer · doi:10.1038/nrc1820 · Cited by 981
The Nurses׳ Health Study: lifestyle and health among women
Colditz, G. A., & Hankinson, S. E. (2005) · Nature reviews. Cancer · doi:10.1038/nrc1608 · Cited by 590
Self-citationOestrogen exposure and breast cancer risk
Travis, R. C., & Key, T. J. (2003) · Breast Cancer Research · doi:10.1186/bcr628 · Cited by 346
Focus on prolactin as a metabolic hormone
Ben-Jonathan, N., Hugo, E. R., Brandebourg, T. D., & LaPensee, C. R. (2006) · Trends in Endocrinology & Metabolism · doi:10.1016/j.tem.2006.02.005 · Cited by 339
Risk for cancer in a cohort of patients hospitalized for schizophrenia in Denmark, 1969–1993
Dalton, S. O., Mellemkjær, L., Thomassen, L., Mortensen, P. B., & Johansen, C. (2005) · Schizophrenia Research · doi:10.1016/j.schres.2004.11.009 · Cited by 142
Metadata
- Authors
- Clevenger, C. V., Furth, P. A., Hankinson, S. E., & Schuler, L. A.
- Venue
- Endocrine Reviews
- Publisher
- The Endocrine Society
- Published
- 2003-02-01
- Volume
- 24
- Issue
- 1
- Pages
- 1-27
- ISSN
- 0163-769X (ISSN-L)
- 1945-7189
- OpenAlex
- W2023523424
- Cited by
- 545
- References
- 11
- FWCI
- 20.83
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
