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The DOI matches its Crossref record; 7 of the 7 references it cites were found in a registry or an index.

Aging enhances liver fibrotic response in mice through hampering extracellular matrix remodeling.

Delire, Bénédicte, Lebrun, Valérie, Selvais, Charlotte, Henriet, Patrick, Bertrand, Amélie, Horsmans, Yves, Leclercq, Isabelle A.·2016·Aging
DOI 10.18632/aging.101124PMC PMC5310658
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Crossref · 10.18632/aging.101124

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  • Liver adiposity and fibrosis are known to increase with age and to be influenced by blood heterochronicity.

    “Liver adiposity and fibrosis are known to increase with age and to be influenced by blood heterochronicity [1, 9, 14, 35–38].”

    From Rejuvenation of three germ layers tissues by exchanging old blood plasma with saline-albumin (2020) · cites this paper as [37]

    The Discussion reports more severe fibrosis in older mice after the same profibrotic treatment and describes increased susceptibility to fibrosis with age. The Introduction discusses progression from steatosis to fibrosis in older patients with fatty liver disease, but neither passage establishes that liver adiposity increases with age; the paper also does not address blood heterochronicity. Page numbers were not provided in our copy. (LLM, conf=0.88)

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Research works printed but not linked: 47

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Metadata

Authors
Delire, B., Lebrun, V., Selvais, C., Henriet, P., Bertrand, A., Horsmans, Y., & Leclercq, I. A.
Affiliations
  • Laboratory of Hepato-Gastroenterology, Institut de Recherche Expérimentale et Clinique (IREC), Université catholique de Louvain (UCL), Brussels, Belgium
  • Cell Biology Unit, de Duve Institute, Université catholique de Louvain, Brussels, Belgium
Venue
Aging
Publisher
Impact Journals, LLC
Published
2016-12-09
Volume
9
Issue
1
Pages
98-113
ISSN
1945-4589
DOI
10.18632/aging.101124
PMC
PMC5310658
Cited by
49
References
7
Language
en

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