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Idiosyncratic DILI: Analysis of 46,266 Cases Assessed for Causality by RUCAM and Published From 2014 to Early 2019
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Idiosyncratic DILI is complex, multifactorial, not predictable, species-specific, cannot be reproduced; the patient is the sole source of the study.
“Idiosyncratic toxicity is more common. This is a complex, multifactorial disease that may involve factors associated with the poison, the patient, and/or the environment. They are not predictable and are species-specific; thus, they cannot be reproduced. As a consequence, the patient is the sole source of the study.”
From Metabolomic Analysis of Pediatric Patients with Idiosyncratic Drug-Induced Liver Injury According to the Updated RUCAM (2023) · cites this paper as [1]
Use of RUCAM to assess causality has allowed an objective view on DILI characteristics based on 46,266 DILI cases published 2014-2019.
“the use of the Roussel Uclaf Causality Assessment Method (RUCAM) to assess causality, which has allowed an objective view on DILI characteristics based on 46,266 DILI cases published 2014-2019.11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
HILI emerges unpredictably in a limited number of susceptible individuals consuming herbs as medicines, based on an idiosyncratic reaction also known from drugs causing DILI.
“HILI emerges unpredictably in a limited number of susceptible individuals consuming herbs as medicines, based on an idiosyncratic reaction also known from drugs causing DILI.11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
DILI conditions are more stable than HILI case evaluations because DILI is caused by clearly defined conventional chemical drugs.
“Contrasting to fragile HILI case evaluations in many publications, conditions are more stable for DILI by clearly defined conventional chemical drugs”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
Similar omissions of RUCAM quotation have been observed in some publications related to DILI.
“Similar omissions of RUCAM quotation have been observed in some publications related to DILI.11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
HILI features are clearly different from those of DILI.
“ignoring thereby that HILI features are clearly different from those of DILI.5–11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
RUCAM is commonly used in cases of DILI and HILI by TCMs.
“A robust diagnostic algorithm, such as RUCAM, is commonly used in cases of DILI11 and HILI by TCMs,18–62,66”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
The retraction by EMA was the consequence of faulty results based on studies misconducted by not-further identified liver injury experts.
“The retraction by EMA was the consequence of faulty results based on studies misconducted by not-further identified liver injury experts.11,65”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
RUCAM is continuously used without problems, except for some minor questions, addressed and clarified in previous RUCAM publications.
“RUCAM is continuously used without problems,11,15 except for some minor questions, addressed and clarified in previous RUCAM publications.15–17”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
Additional support for RUCAM came from a recent study of 46,266 DILI cases, which had been assessed for causality using RUCAM and were published from 2014 to 2019.
“Additional support for RUCAM came from a recent study of 46,266 DILI cases, which had been assessed for causality using RUCAM and were published from 2014 to 2019.11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
For assessing causality in DILI or HILI cases, no other method exists with such a background of worldwide use and acceptance.
“For assessing causality in DILI or HILI cases, no other method exists with such a background of worldwide use and acceptance.11,15”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
A recent analysis showed that RUCAM performs well provided the RUCAM users do a good job.
“Otherwise, a recent analysis showed that RUCAM performs well provided the RUCAM users do a good job.11”
From Herb-induced Liver Injury in Asia and Current Role of RUCAM for Causality Assessment in 11,160 Published Cases (2020) · cites this paper as [11]
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Citations
Cites
Incidence and Etiology of Drug-Induced Liver Injury in Mainland China
Shen, T., Liu, Y., Shang, J., Xie, Q., Li, J., Yan, M., Xu, J., Niu, J., Liu, J., Watkins, P. B., et al. (2019) · Gastroenterology · doi:10.1053/j.gastro.2019.02.002 · Cited by 615
Agreement Among Different Scales for Causality Assessment in Drug-Induced Liver Injury
Das, S., Behera, S. K., Xavier, A. S., Velupula, S., Dkhar, S. A., & Selvarajan, S. (2018) · Clinical Drug Investigation · doi:10.1007/s40261-017-0601-5 · Cited by 19
Flagged first, then by citations
Research works printed but not linked: 111
- Abbara, 2017, Drug-induced liver injury from antituberculous treatment: a retrospective study from a large TB centre in the UK, BMC Infect. Dis., 17, 231, 10.1186/s12879-017-2330-z · DOI · Paper page
- Aiso, 2019, Analysis of 307 cases with drug-induced liver injury between 2010 and 2018 in Japan, Hepatol. Res., 49, 105, 10.1111/hepr.13288 · DOI · Paper page
- Alempijevic, 2017, Drug-induced liver injury: do we know everything, World J. Hepatol., 9, 491, 10.4254/wjh.v9.i10.491 · DOI
- Alhaddad, 2017, Chronic drug-induced liver injury by Amoxicillin-Clavulanate, J. Hepatol., 3, 2, 10.21767/2471-9706.100018 · DOI
- Amagon, 2017, Methionine and vitamin B-complex ameliorate antitubercular drugs-induced toxicity in exposed patients, Pharma. Res. Per., 5, 10.1002/prp2.360 · DOI
- Andrade, 2017, Prospective European Drug-Induced Liver Injury Registry, PRO-EURO-DILI REGISTRY, 2017. Available at: ProEuroDILI Registry progress report year 1.pdf, referenced by: Teschke R, Danan G. Editorial. Drug-induced liver injury, mortality, and liver transplantation: is it reasonable to use a global introspection causality assessment, AME Med. J., 2, 144, 10.21037/amj.2017.09.05 · DOI
- Andrade, 2016, Drug-induced liver injury clinical consortia: a global research response for a worldwide health challenge, Exp. Opin. Drug Metab. Toxicol., 12, 589, 10.1517/17425255.2016.1141896 · DOI
- Baig, 2015, Acute liver failure after initiation of Rivaroxaban: a case report and review of the literature, North Am. J. Med. Sci., 21, 407, 10.4103/1947-2714.166221 · DOI
- Becker, 2019, Drug and herb-induced liver injury: a critical review of Brasilian cases with proposals for the improvement of causality assessment using RUCAM, 10.1016/j.aohep.2019.03.010 · DOI
- Benesic, 2016, Monocyte-derived hepatocyte-like cells for causality assessment of idiosyncratic drug-induced liver injury, Gut, 65, 1555, 10.1136/gutjnl-2015-309528 · DOI
- Bénichou, 1990, Criteria of drug-induced liver disorders. Report of an international consensus meeting, J. Hepatol., 11, 272, 10.1016/0168-8278(90)90124-A · DOI · Paper page
- Bessone, 2016, The Latin American DILI registry experience: a successful ongoing collaborative strategic initiative, Int. J. Mol. Sci., 17, 313, 10.3390/ijms17030313 · DOI
- Bessone, 2019, When the creation of a consortium provides useful answers: experience of The Latin American DILI Network (LATINDILIN), Clin. Liv. Dis., 13, 51, 10.1002/cld.778 · DOI
- Björnsson, 2014, Epidemiology and risk factors for idiosyncratic drug-induced liver injury, Semin Liver Dis., 34, 115, 10.1055/s-0034-1375953 · DOI
- Björnsson, 2016, Hepatotoxicity by drugs: the most common implicated agents, Int. J. Mol. Sci., 17, 224, 10.3390/ijms17020224 · DOI · Paper page
- Björnsson, 2016, Categorization of drugs implicated in causing liver injury: critical assessment based on published case reports, Hepatology, 63, 590, 10.1002/hep.28323 · DOI · Paper page
- Bohm, 2014, Case report and cohort analysis of drug-induced liver injury associated with Daptomycin, Antimicrob. Agents Chemother., 58, 4902, 10.1128/AAC.03157-14 · DOI
- Cano-Paniagua, 2019, Epidemiology of drug-induced liver injury in a University Hospital from Colombia: updated RUCAM being used for prospective causality assessment, Ann. Hepatol, 18, 501, 10.1016/j.aohep.2018.11.008 · DOI
- Chalasani, 2014, Practice Parameters Committee of the American College of Gastroenterology. ACG Clinical Guideline: the diagnosis and management of idiosyncratic drug-induced liver injury, Am. J. Gastroenterol., 109, 950, 10.1038/ajg.2014.131 · DOI · Paper page
- Chalasani, 2015, Features and outcomes of 899 patients with drug-induced liver injury: the DILIN prospective study, Gastroenterology, 148, 1340, 10.1053/j.gastro.2015.03.006 · DOI · Paper page
- Chang, 2015, Severe hepatic injury associated with different statins in patients with chronic liver disease: a nationwide population-based cohort study, J. Gastroenterol. Hepatol., 30, 155, 10.1111/jgh.12657 · DOI
- Chen, 2014, Statins and the risk of liver injury: a population-based case-control study, Pharmacoepidemiol. Drug Saf., 23, 719, 10.1002/pds.3646 · DOI
- Cheetham, 2014, An automated causality assessment algorithm to detect drug-induced liver injury in electronic medical record data, Pharmacoepidemiol. Drug Saf., 23, 601, 10.1002/pds.3531 · DOI
- Church, 2019, Candidate biomarker for the diagnosis and prognosis of drug-induced liver injury: an international collaborator effort, Hepatology, 69, 760, 10.1002/hep.29802 · DOI · Paper page
- Danan, 1993, Causality assessment of adverse reactions to drugs – I. A novel method based on the conclusions of international consensus meetings: application to drug-induced liver injuries, J. Clin. Epidemiol., 46, 1323, 10.1016/0895-4356(93)90101-6 · DOI · Paper page
- Danan, 2016, RUCAM in drug and herb induced liver injury: the update, Int. J. Mol. Sci., 17, 10.3390/ijms17010014 · DOI · Paper page
- Danan, 2018, Drug-induced liver injury: why is the Roussel Uclaf Causality Assessment Method (RUCAM) still used 25 years after its launch, Drug Saf., 41, 735, 10.1007/s40264-018-0654-2 · DOI · Paper page
- Danan, 2019, Roussel Uclaf Causality Assessment Method for drug-induced liver injury: present and future. In: Special issue: Clinical drug induced liver injury: current diagnostic and mechanistic challenges, guest editors: Rolf Teschke, Gaby Danan, James H. Lewis, Front. Pharmacol, 10, 853, 10.3389/fphar2019.00853 · DOI
- Dragoi, 2018, Proteomics analysis of monocyte-derived hepatocyte-like cells identifies integrin beta 3 as a specific biomarker for drug-induced liver injury by diclofenac, Front. Pharmacol., 9, 699, 10.3389/fphar.2018.00699 · DOI
- Douros, 2014, Drug-induced liver injury: results from the hospital-based Berlin Case-Control Surveillance Study, Br. J. Clin. Pharmacol., 79, 988, 10.1111/bcp.12565 · DOI
- Ferrajolo, 2017, Antibiotic-induced liver injury in paediatric outpatients: a case-control study in primary care databases, Drug Saf., 40, 305, 10.1007/s40264-016-0493-y · DOI
- Fontana, 2014, Pathogenesis of idiosyncratic drug-induced liver injury and clinical perspectives, Gastroenterology, 146, 914, 10.1053/j.gastro.2013.12.032 · DOI
- Forner, 2017, Ramipril-associated cholestasis in the setting of a recurrent drug-induced liver injury, Gastroenterol. Hepatol. Bed. Bench., 1, 143
- Giacomelli, 2018, Clinical and genetic factors associated with increased risk of severe liver toxicity in a monocentric cohort of HIV positive patients receiving nevirapine-based antiretroviral therapy, BMC Inf. Dis., 18, 556, 10.1186/s12879-018-3462-5 · DOI
- Hayashi, 2015, Reliability of causality assessment for drug, herbal and dietary supplement hepatotoxicity in the Drug-Induced Liver Injury Network (DILIN), Liver Int., 35, 1623, 10.1111/liv.12540 · DOI
- Hayashi, 2018, Long-term outcomes after drug-induced liver injury, Curr. Hepatol. Rep., 17, 292, 10.1007/s11901-018-0411-0 · DOI
- Hernández, 2014, Profile of idiosyncratic drug induced liver injury in Latin America: an analysis of published reports, Ann. Hepatol., 13, 231, 10.1016/S1665-2681(19)30886-5 · DOI
- Hornby, 2014, MicroRNAs as potential circulating biomarkers of drug-induced liver injury: key current and future issues for translation to humans, Exp. Rev. Clin. Pharmacol., 7, 349, 10.1586/17512433.2014.904201 · DOI
- Hunt, 2018, Editorial. Expanding our toolkit to better identify drug-induced liver injury in electronic medical records, Liver Int., 38, 585, 10.1111/liv.13710 · DOI
- Khoury, 2015, Drug induced liver injury: review with a focus on genetic factors, tissue diagnosis, and treatment options, J. Clin. Transl. Hepatol., 3, 99, 10.14218/JCTH.2015.00007 · DOI
- Kim, 2017, Revisiting the metabolism and bioactivation of ketoconazole in human and mouse using liquid chromatography-mass spectrometry-based metabolomics, Int. J. Mol. Sci., 18, 621, 10.3390/ijms18030621 · DOI
- Kim, 2016, Update on advances in research on idiosyncratic drug-induced liver injury, Allergy Asthm. Immunol. Res., 8, 3, 10.4168/aair.2015.8.1.3 · DOI
- Kim, 2016, Hepatitis C and not hepatitis B virus is a risk factor for anti-tuberculosis drug induced liver injury, BMC Infect. Dis., 16, 50, 10.1186/s12879-016-1344-2 · DOI
- Iorga, 2017, Drug-induced liver injury: cascade of events leading to cell death, apoptosis or necrosis. In: Special Issue. Molecular Research on Drug Induced Liver Injury, guest editors R. Teschke, and G. Danan, Int. J. Mol. Sci., 18, 1018, 10.3390/ijms18051018 · DOI
- Kullak-Ublick, 2017, Drug-induced liver injury: recent advances in diagnosis and risk assessment, Gut, 66, 1154, 10.1136/gutjnl-2016-313369 · DOI · Paper page
- Kwon, 2019, Nimesulide-induced hepatotoxicity: a systemic review and meta-analysis, PLoS One, 14, 10.1371/journal.pone.0209264 · DOI
- Lammel-Lindemann, 2018, Noncholestatic acute hepatocellular injury following candesartan administration, Br. J. Clin. Pharmacol., 84, 204, 10.1111/bcp.13406 · DOI
- Li, 2018, Suspected drug-induced liver injury associated with iguratimod: a case report and review of the literature, BMC Gastroenterol., 18, 130, 10.1186/s12876-018-0858-z · DOI
- Liao, 2019, A rare case of cefepime-induced cholestatic liver injury, Tzu Chi. Med. J., 31, 124, 10.4103/tcmj.tcmj_151_18 · DOI
- Licata, 2017, A focus on epidemiology of drug-induced liver injury: analysis of a prospective study, Eur. Rev. Med. Pharmacol. Sci., 21, 112
- Lim, 2014, A challenge for diagnosing acute liver injury with concomitant/sequential exposure to multiple drugs: can causality assessment scales be utilized to identify the offending drug, 10.1155/2014/156389 · DOI
- Liu, 2018, The diagnostic role of miR-122 in drug-induced liver injury. A systematic review and meta-analysis, Medicine, 97, 49, 10.1097/MD.0000000000013478 · DOI
- Lu, 2016, Clinical characteristics of drug-induced liver injury and related risk factors, Exp. Ther. Med., 12, 2606, 10.3892/etm.2016.3627 · DOI
- Marrone, 2017, Drug-induced liver injury 2017: the diagnosis is not easy but always to keep in mind, Eur. Rev. Med. Pharmacol. Sci., 21, 122
- Matanovic, 2016, Changes to protocol in the regulation of adverse drug reactions –historical and current European view, Clin Res. Reg. Affairs, 33, 49, 10.3109/10601333.2016.1151610 · DOI
- McEuen, 2017, Associations of drug lipophilicity and extent of metabolism with drug-induced liver injury, Int. J. Mol. Sci., 18, 1335, 10.3390/ijms18071335 · DOI
- Medina-Caliz, 2016, Definition and risk factors for chronicity following acute idiosyncratic drug-induced liver injury, J. Hepatol., 65, 532, 10.1016/j.jhep.2016.05.003 · DOI · Paper page
- Meunier, 2018, Viewpoint: recent advances in hepatotoxicity on non steroidal anti-inflammatory drugs, Ann. Hepatol., 17, 187, 10.5604/01.3001.0010.8633 · DOI
- Morales, 2016, Hepatoxicity: a drug-induced cholestatic pattern, Rev. Col. Gastroenterol., 31, 34
- Moreno, 2015, Recurrent drug-induced liver injury (DILI) with ciprofloxacin and amoxillin/clavunate, Rev. Esp. Enferm. Dig., 107, 767, 10.17235/reed.2015.3810/2015 · DOI
- Naiqiong, 2017, A multicenter and randomized controlled trial of Bicyclol in the treatment of statin-induced liver injury, Med. Sci. Monit., 23, 5760, 10.12659/MSM.904090 · DOI
- Nicoletti, 2017, International Drug-Induced Liver Injury Consortium (iDILIC). Association of liver injury from specific drugs or group of drugs with polymorphisms in HLA and other genes in a Genome-wide Association Study, Gastroenterology, 152, 1078, 10.1053/j.gastro.2016.12.016 · DOI · Paper page
- Nicoletti, 2019, International Drug-Induced Liver Injury Consortium (iDILIC). Drug-induced liver injury due to Flucloxacillin: Relevance of multiple human leukocyte antigen alleles
- Niijima, 2017, Drug-induced liver injury caused by Ipragliflozin administration with causality established by a positive lymphocyte transformation test (LTT) and the Roussel Uclaf Causality Assessment Method (RUCAM): a case report, Ann. Hepatol., 16, 308, 10.5604/16652681.1231594 · DOI
- Ou, 2015, Causes, clinical features and outcomes of drug-induced liver injury in hospitalized patients in a Chinese tertiary care hospital, SpringerPlus, 4, 802, 10.1186/s40064-015-1600-8 · DOI
- Ortega-Alonso, 2016, Case characterization, clinical features and risk factors in drug-induced liver injury, Int. J. Mol. Sci., 17, 714, 10.3390/ijms17050714 · DOI
- Patel, 2018, Drug-induced liver injury associated with the use of Everolimus in a liver transplantant patient
- Raschi, 2017, Drug-induced liver injury: towards early prediction and risk stratification, World J. Hepatol., 9, 30, 10.4254/wjh.v9.i1.30 · DOI
- Rathi, 2017, Drug induced liver injury at a tertiary hospital in India: etiology, clinical features and predictors of mortality, Ann. Hepatol., 16, 442, 10.5604/01.3001.0009.8600 · DOI · Paper page
- Real, 2019, Drug-induced liver injury: highlights of the recent literature, Drug Saf., 42, 365, 10.1007/s40264-018-0743-2 · DOI
- Regev, 2014, Causality assessment for suspected DILI during clinical phases of drug development, Drug Saf., 37, S47, 10.1007/s40264-014-0185-4 · DOI
- Robles-Diaz, 2016, Biomarker in DILI: one step forward, Front. Pharmacol., 7, 267, 10.3389/fphar.2016.00267 · DOI
- Robles-Diaz, 2015, Distinct phenotype of hepatotoxicity associated with illicit use of anabolic androgenic steroids, Aliment Pharmacol. Ther., 41, 116, 10.1111/apt.13023 · DOI · Paper page
- Russmann, 2014, Rivaroxaban postmarketing risk of liver injury, J. Hepatol., 61, 293, 10.1016/j.jhep.2014.03.026 · DOI
- Russo, 2014, Spectrum of statin hepatotoxicity: experience of the drug-induced liver injury network, Hepatology, 60, 679, 10.1002/hep.27157 · DOI
- Sarges, 2016, Drug-induced liver injury: highlights from a review of the 2015 literature, Drug Saf., 39, 10.1007/s40264-016-0427-8 · DOI
- Schueller, 2018, The role of miRNAs in the pathophysiology of liver diseases and toxicity, Int. J. Mol. Sci., 19, 261, 10.3390/ijms19010261 · DOI
- Senior, 2014, Evolution of the Food and Drug Administration approach to liver safety assessment for new drugs: current status and challenges, Drug Saf., 37, 17, 10.1007/s40264-014-0182-7 · DOI
- Seyfarth, 2014, Genetic susceptibility to hepatoxicity due to bosentan treatment in pulmonary hypertension, Ann. Hepatol., 13, 803, 10.1016/S1665-2681(19)30983-4 · DOI
- Shahbaz, 2017, Highlights of drug- and herb-induced liver injury in the literature from 2016: how best to translate new information into clinical practice, Exp. Opin. Drug Metab. Toxicol., 13, 935, 10.1080/17425255.2017.1362391 · DOI · Paper page
- Sobhonslidsuk, 2016, The incidence, presentation, outcomes, risk of mortality and economic data of drug-induced liver injury from a national database in Thailand: a population-base study, BMC Gastroenterol., 16, 135, 10.1186/s12876-016-0550-0 · DOI
- Tan, 2018, Systematic review and meta-analysis of algorithms used to identify drug-induced liver injury (DILI) in health record databases, Liver Int., 38, 742, 10.1111/liv.13646 · DOI
- Taneja, 2017, Acute liver failure due to Etodolac, a selective cyclogenase-2 (COX-2) inhibitor Non-steroidal anti-inflammatory drug established by RUCAM-based causality assessment, Ann. Hepatol., 16, 818, 10.5604/01.3001.0010.2829 · DOI
- Tang, 2015, Acute hepatocellular drug-induced liver injury from Bupropion and Doxycycline, ACG Case Rep. J., 3, 66, 10.14309/crj.2015.103 · DOI
- Tao, 2018, Genetic polymorphisms of UGT 1A1 and susceptibility to anti-tuberculosis drug-induced liver: a RUCAM-based case-control study, Int. J. Immunopathol. Pharmacol., 32, 1, 10.1177/2058738418816288 · DOI
- Terziroli Beretta-Piccoli, 2017, Atovaquone/Proguanil-induced autoimmune-like hepatitis, Hepatol. Commun., 1, 293, 10.1002/hep4.1039 · DOI
- Teschke, 2015, Editorial. Drug-induced liver injury: expanding our knowledge by enlarging population analysis with prospective and scoring causality assessment, Gastroenterology, 148, 1271, 10.1053/j.gastro.2015.04.027 · DOI
- Teschke, 2016, Diagnosis and management of drug-induced liver injury (DILI) in patients with pre-existing liver disease, Drug Saf., 39, 729, 10.1007/s40264-016-0423-z · DOI
- Teschke, 2016, Traditional Chinese Medicine (TCM) and herbal hepatotoxicity: RUCAM and the role of novel diagnostic biomarkers such as microRNAs, Medicines, 3, 18, 10.3390/medicines3030018 · DOI · Paper page
- Teschke, 2017, Editorial. Prospective Indian study of DILI with confirmed causality using the Roussel Uclaf Causality Assessment Method (RUCAM): a report of excellence, Ann. Hepatol., 16, 324, 10.5604/16652681.1235471 · DOI
- Teschke, 2017, Drug induced liver injury: can biomarkers assist RUCAM in causality assessment? Special issue: molecular research on drug induced liver injury, Int. J. Mol. Sci., 18, 803, 10.3390/ijms18040803 · DOI
- Teschke, 2017, Exp. Opin. Drug Metab. Toxicol., 13, 425, 10.1080/17425255.2017.1252749 · DOI
- Teschke, 2018, Commentary. Drug- and herb-induced liver injury in 2016 with highly appreciated critical comments: related or not, that is the question, Clin. Diagn. Pathol., 2, 1, 10.15761/CDP.1000122 · DOI
- Teschke, 2018, Causality assessment methods in drug-induced liver injury. In: Drug-induced Liver Toxicity (Chapter 27), 555
- Teschke, 2018, Review: drug induced liver injury with analysis of alternative causes as confounding variables, Br. J. Clin. Pharmacol., 84, 1467, 10.1111/bcp.13593 · DOI
- Teschke, 2018, Review. Top-ranking drugs out of 3312 drug-induced liver injury cases evaluated by the Roussel Uclaf Causality Assessment Method, Exp. Opin. Drug Metab. Toxicol., 14, 1169, 10.1080/17425255.2018.1539077 · DOI · Paper page
- Teschke, 2018, Paracetamol (acetaminophen), alcohol, and liver injury: biomarkers, clinical issues, and experimental aspects, SL Pharmacol. Toxicol., 1, 113
- Teschke, 2019, Drug induced liver injury
- Tillmann, 2018, Novel approaches to causality adjudication in drug-induced liver disease, Curr. Hepatol. Rep, 17, 276, 10.1007/s11901-018-0416-8 · DOI
- Veluswamy, 2014, Adverse drug reaction: pomalidomide-induced liver injury, Lancet, 383, 2125, 10.1016/S0140-6736(14)61030-8 · DOI
- Visentin, 2018, Drug-induced bile duct injury, Biochim. Biophys. Acta., 1864, 1498, 10.1016/j.bbadis.2017.08.033 · DOI
- Wang, 2017, Drug induced autoimmune hepatitis (DIAIH): pathological and clinical study, Biomed Res., 28, 6028
- Wang, 2016, Chronic hepatitis B infection and risk of antituberculosis drug-induced liver injury: systematic review and meta-analysis, J. Chin. Med. Assoc., 79, 368, 10.1016/j.jcma.2015.12.006 · DOI
- Wang, 2018, Risk factors for acute liver failure among inpatients with anti-tuberculosis drug-induced liver injury, J. Int. Med. Res., 26, 300060518811512, 10.1177/0300060518811512 · DOI
- Watkins, 2015, How to diagnose and exclude drug-induced liver injury, Dig. Dis., 33, 472, 10.1159/000374091 · DOI
- Weiler, 2015, Drug-induced liver injury: the dawn of biomarkers, F1000Prime Rep., 7, 34, 10.12703/P7-34 · DOI
- Yamashita, 2017, Idiosyncratic drug-induced liver injury: a short review, Hepatol. Commun., 1, 494, 10.1002/hep4.1064 · DOI
- Yang, 2016, Clinical characteristics of drug-induced liver injury and primary biliary cirrhosis, World J. Gastroenterol., 22, 7579, 10.3748/wjg.v22.i33.7579 · DOI
- Yu, 2017, CSH guidelines for the diagnosis and treatment of drug-induced liver injury, Hepatol. Int., 11, 221, 10.1007/s12072-017-9793-2 · DOI
- Zhu, 2016, Comparison between Chinese herbal medicine and Western medicine-induced liver injury of 1985 patients, J. Gastroenterol. Hepatol., 31, 1476, 10.1111/jgh.13323 · DOI · Paper page
- Zhu, 2015, Causes, features, and outcomes of drug-induced liver injury in 69 children from China, Gut. Liver, 9, 525, 10.5009/gnl14184 · DOI
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Sorted by citations
Teschke, R., Zhu, Y., & Jing, J. (2020) · Journal of Clinical and Translational Hepatology · doi:10.14218/jcth.2020.00009 · Cited by 41
Self-citationAndújar-Vera, F., Alés-Palmer, M. L., Muñoz-de-Rueda, P., Iglesias-Baena, I., & Ocete-Hita, E. (2023) · International Journal of Molecular Sciences · doi:10.3390/ijms241713562 · Cited by 10
Shamseddeen, H., Vilar-Gomez, E., Chalasani, N., Myers, R. P., Subramanian, G. M., Shlevin, H. H., Allgood, A. E., & Orman, E. S. (2020) · Drug Safety · doi:10.1007/s40264-019-00896-1 · Cited by 6
Metadata
- Authors
- Teschke, R.
- Venue
- Frontiers in Pharmacology
- Publisher
- Frontiers Media SA
- Published
- 2019-07-23
- Volume
- 10
- ISSN
- 1663-9812
- PMC
- PMC6664244
- Cited by
- 62
- References
- 2
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
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