The DOI matches its Crossref record; 26 of the 26 references it cites were found in a registry or an index.
Part I. Male Pseudohermaphroditism in the Laboratory Norway Rat
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The tfm mutation in the rat is a consequence of a point mutation in the androgen receptor gene corresponding to the steroid-binding domain, resulting in largely nonfunctional androgen receptors.
“In the rat (Stanley, Gumbreck, Allison, & Easley, 1973), the tfm is a consequence of a point mutation in the androgen receptor gene corresponding to the steroid-binding domain (Yarbrough et al., 1990) resulting in largely nonfunctional androgen receptors.”
From Play fighting in androgen-insensitivetfm rats: Evidence that androgen receptors are necessary for the development of adult playful attack and defense (2006) · cites this paper as Stanley, Gumbreck, Allison, & Easley, 1973
Because the androgen receptor gene is on the X chromosome, tfm-affected XY males are insensitive to the direct effect of androgens and develop peripherally as phenotypic females.
“Because the androgen receptor gene is resident on the X chromosome tfm-affected XY males are insensitive to the direct effect of androgens and develop peripherally as phenotypic females, with a perforate vagina, a nipple line (Stanley et al., 1973), and a smaller body size than WT males (McGinnis, Marcelli, & Lamb, 2002;Vanderschueren et al., 1994).”
From Play fighting in androgen-insensitivetfm rats: Evidence that androgen receptors are necessary for the development of adult playful attack and defense (2006) · cites this paper as Stanley et al., 1973
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Metadata
- Authors
- STANLEY, A. J., GUMBRECK, L. G., ALLISON, J. E., & EASLEY, R. B.
- Affiliations
- University of Oklahoma Health Sciences Center (US)
- Duke University Hospital (US)
- ORCID
- EASLEY, RONALD B.
- Venue
- Proceedings of the 1972 Laurentian Hormone Conference
- Publisher
- Elsevier
- Published
- 1973
- Volume
- 29
- Pages
- 43-64
- OpenAlex
- W20444889
- Cited by
- 80
- References
- 26
- Citations by year
- 2020: 1 · 2015: 1
- FWCI
- 2.85
- Type
- book chapter
- Language
- en
- Open access
- closed
- Locations
- Elsevier eBooks (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Sexual Differentiation and Disorders
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Genetics › Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities
- Health Sciences › Medicine › Urology › Urological Disorders and Treatments
- Keywords
- 46,XY disorders of sex development, Rattus norvegicus, genetic transmission, female carriers, androgen insensitivity syndrome, spermatogenic arrest, gonadotropin stimulation
- MeSH terms
- Adrenal Glands, Animals, Female, Gonadotropins, Pituitary, Male, Molecular Biology, Norway, Organ Size, Parabiosis, Pregnancy, Disorders of Sex Development, Testis, Testosterone, 17-Ketosteroids, Rats
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
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