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The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

A Minicircuitry Comprised of MicroRNA-223 and Transcription Factors NFI-A and C/EBPα Regulates Human Granulopoiesis

Fazi, Francesco, Rosa, Alessandro, Fatica, Alessandro, Gelmetti, Vania, De Marchis, Maria Laura, Nervi, Clara, Bozzoni, Irene·2005·Cell
DOI 10.1016/j.cell.2005.09.023
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  • miR-223 was reported to direct granulocytic development, although this has been questioned using knockout studies in mice.

    “miR-223 was reported to direct granulocytic development, 10 although this has been questioned using knockout (KO) studies in mice.11,12”

    From Macrophage development from HSCs requires PU.1-coordinated microRNA expression (2011) · cites this paper as [10]

  • Several miRs (e.g., miR-223 and the miR-17p-92 cluster) that were down-regulated by PU.1 have been shown to block macrophage differentiation.

    “In agreement with this, several of the miRs (eg, miR-223 and the miR-17p-92 cluster) that were down-regulated by PU.1 have been shown to block macrophage differentiation.10,13”

    From Macrophage development from HSCs requires PU.1-coordinated microRNA expression (2011) · cites this paper as [10]

  • We identified several miRs with previously reported roles in myelopoiesis and/or leukemia as regulated by PU.1.

    “In addition to miRs with thus-far-unknown functions in hematopoietic cells, we identified several miRs with previously reported (partly controversial) roles in myelopoiesis and/or leukemia as regulated by PU.1.10,16,[46][47][48]”

    From Macrophage development from HSCs requires PU.1-coordinated microRNA expression (2011) · cites this paper as [10]

  • miR-223 has been reported to stimulate granulopoiesis, although controversy has been raised about this function.

    “This miR has been reported to stimulate granulopoiesis, 10 although controversy has been raised about this particular function.11,12”

    From Macrophage development from HSCs requires PU.1-coordinated microRNA expression (2011) · cites this paper as [10]

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Research works printed but not linked: 37

  1. Bachurski, 1997, Nuclear factor I family members regulate the transcription of surfactant protein-C, J. Biol. Chem., 272, 32759, 10.1074/jbc.272.52.32759 · DOI
  2. Barnett, 1999, Guideline for the flow cytometric enumeration of CD34+ haematopoietic stem cells. Prepared by the CD34+ haematopoietic stem cell working party. General Haematology Task Force of the British Committee for Standards in Haematology, Clin. Lab. Haematol., 21, 301, 10.1046/j.1365-2257.1999.00253.x · DOI
  3. Bartel, 2004, MicroRNAs: genomics, biogenesis, mechanism, and function, Cell, 116, 281, 10.1016/S0092-8674(04)00045-5 · DOI
  4. Benjamin, 1996, EBV-positive tumor B cell lines derived from patients with American Burkitt's lymphoma, J. Immunol., 156, 1626, 10.4049/jimmunol.156.4.1626 · DOI
  5. Bonci, 2003, ‘Advanced’ generation lentiviruses as efficient vectors for cardiomyocyte gene transduction in vitro and in vivo, Gene Ther., 10, 630, 10.1038/sj.gt.3301936 · DOI
  6. Chakrabarti, 2002, Quantitative assessment of gene targeting in vitro and in vivo by the pancreatic transcription factor, Pdx1. Importance of chromatin structure in directing promoter binding, J. Biol. Chem., 277, 13286, 10.1074/jbc.M111857200 · DOI
  7. Chen, 2004, MicroRNAs modulate hematopoietic lineage differentiation, Science, 303, 83, 10.1126/science.1091903 · DOI · Paper page
  8. Collins, 1990, Retinoic acid-induced granulocytic differentiation of HL-60 cells myeloid leukemia cells is mediated directly through the retinoic acid receptor (RAR-alpha), Mol. Cell. Biol., 10, 2154, 10.1128/MCB.10.5.2154 · DOI
  9. Denti, 2004, A new vector, based on the Pol II promoter of the U1 snRNA gene, for the expression of siRNAs in mammalian cells, Mol. Ther., 10, 191, 10.1016/j.ymthe.2004.04.008 · DOI
  10. Di Croce, 2002, Methyltransferase recruitment and DNA hypermethylation of target promoters by an oncogenic transcription factor, Science, 295, 1079, 10.1126/science.1065173 · DOI
  11. Duprez, 2003, C/EBPbeta: a major PML-RARA-responsive gene in retinoic acid-induced differentiation of APL cells, EMBO J., 22, 5806, 10.1093/emboj/cdg556 · DOI
  12. Fazi, 2005, Retinoic acid targets DNA-methyltransferases and histone deacetylases during APL blast differentiation in vitro and in vivo, Oncogene, 24, 1820, 10.1038/sj.onc.1208286 · DOI
  13. Garlatti, 1993, CCAAT/enhancer-binding protein-related proteins bind to the unusual promoter of the aspartate aminotransferase housekeeping gene, J. Biol. Chem., 268, 6567, 10.1016/S0021-9258(18)53288-9 · DOI
  14. Gombart, 2002, Mutations in the gene encoding the transcription factor CCAAT/enhancer binding protein alpha in myelodysplastic syndromes and acute myeloid leukemias, Blood, 99, 1332, 10.1182/blood.V99.4.1332 · DOI
  15. Gronostajski, 2000, Roles of the NF1/CTF gene family in transcription and development, Gene, 249, 31, 10.1016/S0378-1119(00)00140-2 · DOI
  16. Khanna-Gupta, 2001, C/EBP epsilon mediates myeloid differentiation and is regulated by the CCAAT displacement protein (CDP/cut), Proc. Natl. Acad. Sci. USA, 98, 8000, 10.1073/pnas.141229598 · DOI
  17. Khanna-Gupta, 2003, Chromatin immunoprecipitation (ChIP) studies indicate a role for CCAAT enhancer binding proteins alpha and epsilon (C/EBP alpha and C/EBP epsilon) and CDP/cut in myeloid maturation-induced lactoferrin gene expression, Blood, 101, 3460, 10.1182/blood-2002-09-2767 · DOI
  18. Lanotte, 1991, NB4, a maturation inducible cell line with t(15;17) marker isolated from a human acute promyelocytic leukemia (M3), Blood, 77, 1080, 10.1182/blood.V77.5.1080.1080 · DOI
  19. Lecellier, 2005, A cellular microRNA mediates antiviral defense in human cells, Science, 308, 557, 10.1126/science.1108784 · DOI
  20. Lewis, 2005, Conserved seed pairing, often flanked by adenosines, indicates that thousands of human genes are microRNA targets, Cell, 120, 15, 10.1016/j.cell.2004.12.035 · DOI · Paper page
  21. Lo Coco, 1992, Molecular evaluation of residual disease as a predictor of relapse in acute promyelocytic leukemia, Lancet, 340, 1437, 10.1016/0140-6736(92)92625-P · DOI
  22. Lo Coco, 2002, Progress in differentiation induction as a treatment for acute promyelocytic leukemia and beyond, Cancer Res., 62, 5618
  23. Lodie, 1999, PML/RARalpha induces ATRA-sensitive delocalization of the critical granulocytic differentiation factor C/EBPalpha to a microspeckled nuclear pattern in t(15;17) APL, Blood, 94, 692a
  24. Mandelli, 1997, Molecular remission in PML/RARa-positive acute promyelocytic leukemia by combined all-trans-retinoic acid and idarubicin (AIDA) therapy, Blood, 90, 1014
  25. McKnight, 2001, McBindall—a better name for CCAAT/enhancer binding proteins?, Cell, 107, 259, 10.1016/S0092-8674(01)00543-8 · DOI
  26. Meisterernst, 1988, A quantitative analysis of nuclear factor I/DNA interactions, Nucleic Acids Res., 16, 4419, 10.1093/nar/16.10.4419 · DOI
  27. Melnick, 1999, Deconstructing a disease: RARα, its fusion partners, and their roles in the pathogenesis of acute promyelocytic leukemia, Blood, 93, 3167, 10.1182/blood.V93.10.3167.410k44_3167_3215 · DOI
  28. Meltzer, 2005, Cancer genomics: small RNA with big impacts, Nature, 435, 745, 10.1038/435745a · DOI
  29. Mittelstadt, 1998, Cyclosporin A-sensitive transcription factor Egr-3 regulates Fas ligand expression, Mol. Cell. Biol., 18, 3744, 10.1128/MCB.18.7.3744 · DOI
  30. Pabst, 2001, Dominant-negative mutations of CEBPA, encoding CCAAT/enhancer binding protein-alpha (C/EBPalpha), in acute myeloid leukemia, Nat. Genet., 27, 263, 10.1038/85820 · DOI · Paper page
  31. Park, 1999, CCAAT/enhancer binding protein epsilon is a potential retinoid target gene in acute promyelocytic leukemia treatment, J. Clin. Invest., 103, 1399, 10.1172/JCI2887 · DOI · Paper page
  32. Pasquinelli, 2005, MicroRNAs: a developing story, Curr. Opin. Genet. Dev., 15, 200, 10.1016/j.gde.2005.01.002 · DOI
  33. Santoro, 1988, A family of human CCAAT-box-binding proteins active in transcription and DNA replication: cloning and expression of multiple cDNAs, Nature, 334, 218, 10.1038/334218a0 · DOI
  34. Shao, 1997, A retinoid-resistant acute promyelocytic leukemia subclone expresses a dominant negative PML-RARa mutation, Blood, 89, 4282, 10.1182/blood.V89.12.4282 · DOI
  35. Tenen, 2001, Abnormalities of the CEBP alpha transcription factor: a major target in acute myeloid leukemia, Leukemia, 4, 688, 10.1038/sj.leu.2402088 · DOI
  36. Tenen, 2003, Disruption of differentiation in human cancer: AML shows the way, Nat. Rev. Cancer, 3, 89, 10.1038/nrc989 · DOI · Paper page
  37. Wang, 1999, C/EBPalpha bypasses granulocyte colony-stimulating factor signals to rapidly induce PU.1 gene expression, stimulate granulocytic differentiation, and limit proliferation in 32D cl3 myeloblasts, Blood, 94, 560, 10.1182/blood.V94.2.560 · DOI · Paper page

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Metadata

Authors
Fazi, F., Rosa, A., Fatica, A., Gelmetti, V., De Marchis, M. L., Nervi, C., & Bozzoni, I.
Venue
Cell
Publisher
Elsevier BV
Published
2005-12
Volume
123
Issue
5
Pages
819-831
ISSN
0092-8674
DOI
10.1016/j.cell.2005.09.023
Cited by
894
References
6
Language
en
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