CoChat
Sign Up
Cited by 41

The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.

Food Allergies and Intolerances: A Clinical Approach to the Diagnosis and Management of Adverse Reactions to Food

Onyimba, Frances, Crowe, Sheila E., Johnson, Sarah, Leung, John·2021·Clinical Gastroenterology and Hepatology
DOI 10.1016/j.cgh.2021.01.025
Open this paper in CoChat — free account

Abstract

Food allergy is an aberrant immunological response to food antigen, which can result in potentially life-threatening reactions. It is often challenging to differentiate food allergy from other adverse reactions to food because their presentations can be indistinguishable. The purpose of this article is to give an overview of the classification, evaluation, and management of adverse food reactions, key differentiating features of food allergy, roles and limitations of various food allergy testing, and promising areas of emerging research. Case studies are used to highlight some of the clinical pearls in diagnosing and managing food-related diseases.

Overview

Integrity signals

Registry

Crossref · 10.1016/j.cgh.2021.01.025

Cites

5 of 5 references verified0 citation errors · 0 not checkable

Statistics

Recomputing p-values needs the paper’s full text.

Verification

Claims the paper makes

This paper’s full text isn’t openly available, so its claims and self-check weren’t run. The registry and citation checks still apply.

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Citations

Cites

Research works printed but not linked: 64

  1. Young, 1994, A population study of food intolerance, Lancet, 343, 1127, 10.1016/S0140-6736(94)90234-8 · DOI
  2. Pereira, 2005, Prevalence of sensitization to food allergens, reported adverse reaction to foods, food avoidance, and food hypersensitivity among teenagers, J Allergy Clin Immunol, 116, 884, 10.1016/j.jaci.2005.05.047 · DOI
  3. Nwaru, 2014, Prevalence of common food allergies in Europe: a systematic review and meta-analysis, Allergy, 69, 992, 10.1111/all.12423 · DOI
  4. Osborne, 2011, Prevalence of challenge-proven IgE-mediated food allergy using population-based sampling and predetermined challenge criteria in infants, J Allergy Clin Immunol, 127, 668, 10.1016/j.jaci.2011.01.039 · DOI
  5. Rona, 2007, The prevalence of food allergy: a meta-analysis, J Allergy Clin Immunol, 120, 638, 10.1016/j.jaci.2007.05.026 · DOI
  6. Acker, 2017, Prevalence of food allergies and intolerances documented in electronic health records, J Allergy Clin Immunol, 140, 1587, 10.1016/j.jaci.2017.04.006 · DOI
  7. Sampson, 2014, Food allergy: a practice parameter update-2014, J Allergy Clin Immunol, 134, 1016, 10.1016/j.jaci.2014.05.013 · DOI · Paper page
  8. Bischoff, 2005, Gastrointestinal food allergy: new insights into pathophysiology and clinical perspectives, Gastroenterology, 128, 1089, 10.1053/j.gastro.2004.08.015 · DOI
  9. Gupta, 2019, Prevalence and severity of food allergies among US adults, JAMA Netw Open, 2, 10.1001/jamanetworkopen.2018.5630 · DOI
  10. Sampson, 1999, Food allergy. Part 1: immunopathogenesis and clinical disorders, J Allergy Clin Immunol, 103, 717, 10.1016/S0091-6749(99)70411-2 · DOI
  11. Kamdar, 2015, Prevalence and characteristics of adult-onset food allergy, J Allergy Clin Immunol Pract, 3, 114, 10.1016/j.jaip.2014.07.007 · DOI
  12. Boman, 2000, Innate immunity and the normal microflora, Immunol Rev, 173, 5, 10.1034/j.1600-065X.2000.917301.x · DOI
  13. Zasloff, 2002, Antimicrobial peptides in health and disease, N Engl J Med, 347, 1199, 10.1056/NEJMe020106 · DOI
  14. Kuchroo, 1995, B7-1 and B7-2 costimulatory molecules activate differentially the Th1/Th2 developmental pathways: application to autoimmune disease therapy, Cell, 80, 707, 10.1016/0092-8674(95)90349-6 · DOI
  15. Steinbrink, 1997, Pillars article: induction of tolerance by IL-10-treated dendritic cells, J Immunol, 159, 4772, 10.4049/jimmunol.159.10.4772 · DOI
  16. Bellou, 2003, Toll receptors modulate allergic responses: interaction with dendritic cells, T cells and mast cells, Curr Opin Allergy Clin Immunol, 3, 487, 10.1097/00130832-200312000-00011 · DOI
  17. Hussain, 2018, Basophil-derived IL-4 promotes epicutaneous antigen sensitization concomitant with the development of food allergy, J Allergy Clin Immunol, 141, 223, 10.1016/j.jaci.2017.02.035 · DOI
  18. Noval Rivas, 2016, IL-4 production by group 2 innate lymphoid cells promotes food allergy by blocking regulatory T-cell function, J Allergy Clin Immunol, 138, 801, 10.1016/j.jaci.2016.02.030 · DOI
  19. Crespo, 2003, Food allergy in adulthood, Allergy, 58, 98, 10.1034/j.1398-9995.2003.02170.x · DOI
  20. Turnbull, 2015, Review article: the diagnosis and management of food allergy and food intolerances, Aliment Pharmacol Ther, 41, 3, 10.1111/apt.12984 · DOI
  21. Commins, 2009, Delayed anaphylaxis, angioedema, or urticaria after consumption of red meat in patients with IgE antibodies specific for galactose-alpha-1,3-galactose, J Allergy Clin Immunol, 123, 426, 10.1016/j.jaci.2008.10.052 · DOI
  22. Commins, 2011, The relevance of tick bites to the production of IgE antibodies to the mammalian oligosaccharide galactose-α-1,3-galactose, J Allergy Clin Immunol, 127, 1286, 10.1016/j.jaci.2011.02.019 · DOI · Paper page
  23. Green, 2007, Clinical characteristics of peanut-allergic children: recent changes, Pediatrics, 120, 1304, 10.1542/peds.2007-0350 · DOI
  24. Eigenmann, 1998, Prevalence of IgE-mediated food allergy among children with atopic dermatitis, Pediatrics, 101, E8, 10.1542/peds.101.3.e8 · DOI
  25. Talley, 1990, Eosinophilic gastroenteritis: a clinicopathological study of patients with disease of the mucosa, muscle layer, and subserosal tissues, Gut, 31, 54, 10.1136/gut.31.1.54 · DOI
  26. Kotlyar, 2014, Non-pulmonary allergic diseases and inflammatory bowel disease: a qualitative review, World J Gastroenterol, 20, 11023, 10.3748/wjg.v20.i32.11023 · DOI
  27. Ciacci, 2004, Allergy prevalence in adult celiac disease, J Allergy Clin Immunol, 113, 1199, 10.1016/j.jaci.2004.03.012 · DOI
  28. Boyce, 2010, Guidelines for the diagnosis and management of food allergy in the United States: report of the NIAID-sponsored expert panel, J Allergy Clin Immunol, 126, S1, 10.1016/j.jaci.2010.10.008 · DOI
  29. Warners, 2018, Abnormal responses to local esophageal food allergen injections in adult patients with eosinophilic esophagitis, Gastroenterology, 154, 57, 10.1053/j.gastro.2017.08.062 · DOI
  30. Soares-Weiser, 2014, The diagnosis of food allergy: a systematic review and meta-analysis, Allergy, 69, 76, 10.1111/all.12333 · DOI
  31. Kelly, 1995, Eosinophilic esophagitis attributed to gastroesophageal reflux: improvement with an amino acid-based formula, Gastroenterology, 109, 1503, 10.1016/0016-5085(95)90637-1 · DOI
  32. Rank, 2020, Technical review on the management of eosinophilic esophagitis: a report from the AGA Institute and the joint task force on allergy-immunology practice parameters, Ann Allergy Asthma Immunol, 124, 424, 10.1016/j.anai.2020.03.021 · DOI
  33. Restani, 2009, Meat allergy, Curr Opin Allergy Clin Immunol, 9, 265, 10.1097/ACI.0b013e32832aef3d · DOI
  34. Fischer, 2017, Prevalence of type I sensitization to alpha-gal in forest service employees and hunters, Allergy, 72, 1540, 10.1111/all.13156 · DOI
  35. Villalta, 2016, High prevalence of sIgE to galactose-α-1,3-galactose in rural pre-Alps area: a cross-sectional study, Clin Exp Allergy, 46, 377, 10.1111/cea.12655 · DOI
  36. Shadick, 1999, The natural history of exercise-induced anaphylaxis: survey results from a 10-year follow-up study, J Allergy Clin Immunol, 104, 123, 10.1016/S0091-6749(99)70123-5 · DOI
  37. Robson-Ansley, 2010, Pathophysiology, diagnosis and management of exercise-induced anaphylaxis, Curr Opin Allergy Clin Immunol, 10, 312, 10.1097/ACI.0b013e32833b9bb0 · DOI
  38. Ma, 2003, A survey on the management of pollen-food allergy syndrome in allergy practices, J Allergy Clin Immunol, 112, 784, 10.1016/S0091-6749(03)02008-6 · DOI
  39. Akin, 2017, Mast cell activation syndromes, J Allergy Clin Immunol, 140, 349, 10.1016/j.jaci.2017.06.007 · DOI
  40. Castells, 2019, Mast cell activation syndrome and mastocytosis: initial treatment options and long-term management, J Allergy Clin Immunol Pract, 7, 1097, 10.1016/j.jaip.2019.02.002 · DOI · Paper page
  41. Horan, 1990, Cromolyn sodium in the management of systemic mastocytosis, J Allergy Clin Immunol, 85, 852, 10.1016/0091-6749(90)90067-E · DOI
  42. Vickery, 2018, AR101 oral immunotherapy for peanut allergy, N Engl J Med, 379, 1991, 10.1056/NEJMoa1812856 · DOI
  43. Sicherer, 2018, Food allergy: a review and update on epidemiology, pathogenesis, diagnosis, prevention, and management, J Allergy Clin Immunol, 141, 41, 10.1016/j.jaci.2017.11.003 · DOI
  44. Chey, 2016, Food: the main course to wellness and illness in patients with irritable bowel syndrome, Am J Gastroenterol, 111, 366, 10.1038/ajg.2016.12 · DOI
  45. Levitt, 1998, Evaluation of an extremely flatulent patient: case report and proposed diagnostic and therapeutic approach, Am J Gastroenterol, 93, 2276
  46. Chey, 2002, Utility of the Rome I and Rome II criteria for irritable bowel syndrome in U.S. women, Am J Gastroenterol, 97, 2803, 10.1111/j.1572-0241.2002.07026.x · DOI
  47. Boettcher, 2013, Dietary proteins and functional gastrointestinal disorders, Am J Gastroenterol, 108, 728, 10.1038/ajg.2013.97 · DOI
  48. Drossman, 2006, The functional gastrointestinal disorders and the Rome III process, Gastroenterology, 130, 1377, 10.1053/j.gastro.2006.03.008 · DOI · Paper page
  49. Drossman, 2016, Functional gastrointestinal disorders: history, pathophysiology, clinical features and Rome IV, Gastroenterology, 10.1053/j.gastro.2016.02.032 · DOI
  50. Barrett, 2010, Dietary poorly absorbed, short-chain carbohydrates increase delivery of water and fermentable substrates to the proximal colon, Aliment Pharmacol Ther, 31, 874, 10.1111/j.1365-2036.2010.04237.x · DOI
  51. Shepherd, 2013, Short-chain carbohydrates and functional gastrointestinal disorders, Am J Gastroenterol, 108, 707, 10.1038/ajg.2013.96 · DOI
  52. Ong, 2010, Manipulation of dietary short chain carbohydrates alters the pattern of gas production and genesis of symptoms in irritable bowel syndrome, J Gastroenterol Hepatol, 25, 1366, 10.1111/j.1440-1746.2010.06370.x · DOI · Paper page
  53. Halmos, 2014, A diet low in FODMAPs reduces symptoms of irritable bowel syndrome, Gastroenterology, 146, 67, 10.1053/j.gastro.2013.09.046 · DOI · Paper page
  54. Altobelli, 2017, Low-FODMAP diet improves irritable bowel syndrome symptoms: a meta-analysis, Nutrients, 9, 940, 10.3390/nu9090940 · DOI
  55. Catassi, 2017, The overlapping area of non-celiac gluten sensitivity (NCGS) and wheat-sensitive irritable bowel syndrome (IBS): An Update, Nutrients, 9, 1268, 10.3390/nu9111268 · DOI
  56. Dicke, 1953, Coeliac disease. II. The presence in wheat of a factor having a deleterious effect in cases of coeliac disease, Acta Paediatr, 42, 34, 10.1111/j.1651-2227.1953.tb05563.x · DOI
  57. Campanella, 2008, Clinical response to gluten withdrawal is not an indicator of coeliac disease, Scand J Gastroenterol, 43, 1311, 10.1080/00365520802200036 · DOI
  58. Ludvigsson, 2013, The Oslo definitions for coeliac disease and related terms, Gut, 62, 43, 10.1136/gutjnl-2011-301346 · DOI · Paper page
  59. Hadjivassiliou, 2016, Neurological dysfunction in coeliac disease and non-coeliac gluten sensitivity, Am J Gastroenterol, 111, 561, 10.1038/ajg.2015.434 · DOI
  60. Molina-Infante, 2017, Suspected nonceliac gluten sensitivity confirmed in few patients after gluten challenge in double-blind, placebo-controlled trials, Clin Gastroenterol Hepatol, 15, 339, 10.1016/j.cgh.2016.08.007 · DOI
  61. Skodje, 2018, Fructan, rather than gluten, induces symptoms in patients with self-reported non-celiac gluten sensitivity, Gastroenterology, 154, 529, 10.1053/j.gastro.2017.10.040 · DOI
  62. Biesiekierski, 2013, No effects of gluten in patients with self-reported non-celiac gluten sensitivity after dietary reduction of fermentable, poorly absorbed, short-chain carbohydrates, Gastroenterology, 145, 320, 10.1053/j.gastro.2013.04.051 · DOI
  63. Biesiekierski, 2011, Gluten causes gastrointestinal symptoms in subjects without celiac disease: a double-blind randomized placebo-controlled trial, Am J Gastroenterol, 106, 508, 10.1038/ajg.2010.487 · DOI
  64. Vazquez-Roque, 2013, A controlled trial of gluten-free diet in patients with irritable bowel syndrome-diarrhea: effects on bowel frequency and intestinal function, Gastroenterology, 144, 903, 10.1053/j.gastro.2013.01.049 · DOI

Cited by

Sorted by citations

See all 41

Metadata

Authors
Onyimba, F., Crowe, S. E., Johnson, S., & Leung, J.
ORCID
Leung, John
Venue
Clinical Gastroenterology and Hepatology
Publisher
Elsevier BV
Published
2021-11
Volume
19
Issue
11
Pages
2230-2240.e1
ISSN
1542-3565
DOI
10.1016/j.cgh.2021.01.025
Cited by
41
References
5
Language
en
Licence

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review