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Mechanisms of statin-associated skeletal muscle-associated symptoms
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“Statins lower serum CoQ10 by inhibiting the enzyme 3-hydro-3-methylglutaryl Coenzyme-A (HMG-CoA) reductase, a rate-limiting step of cholesterol biosynthesis, which subsequently inhibits the conversion of mevalonate to CoQ10 [82].”
From The Use of Medications and Dietary Supplements by Masters Athletes—A Review (2022) · cites this paper as [82]
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Transcriptional co-activator PGC-1α drives the formation of slow-twitch muscle fibres
Lin, J., Wu, H., Tarr, P. T., Zhang, C.-Y., Wu, Z., Boss, O., Michael, L. F., Puigserver, P., Isotani, E., Olson, E. N., et al. (2002) · Nature · doi:10.1038/nature00904 · Cited by 2,546
Statins and risk of incident diabetes: A collaborative meta-analysis of randomised statin trials
Sattar, N., Preiss, D., Murray, H. M., Welsh, P., Buckley, B. M., de Craen, A. J., Seshasai, S. R. K., McMurray, J. J., Freeman, D. J., Jukema, J. W., et al. (2010) · The Lancet · doi:10.1016/s0140-6736(09)61965-6 · Cited by 2,436
SLCO1B1 Variants and Statin-Induced Myopathy — A Genomewide Study
(2008) · New England Journal of Medicine · doi:10.1056/nejmoa0801936 · Cited by 2,043
Rosuvastatin in older patients with systolic heart failure
Kjekshus, J., Apetrei, E., Barrios, V., Böhm, M., Cleland, J. G. F., Cornel, J. H., Dunselman, P., Fonseca, C., Goudev, A., Grande, P., et al. (2007) · New England Journal of Medicine · doi:10.1056/nejmoa0706201 · Cited by 1,487
A Century of Cholesterol and Coronaries: From Plaques to Genes to Statins
Goldstein, J. L., & Brown, M. S. (2015) · Cell · doi:10.1016/j.cell.2015.01.036 · Cited by 1,233
Bruckert, E., Hayem, G., Dejager, S., Yau, C., & Bégaud, B. (2005) · Cardiovascular Drugs and Therapy · doi:10.1007/s10557-005-5686-z · Cited by 1,226
Skeletal muscle hypertrophy and atrophy signaling pathways
Glass, D. J. (2005) · The International Journal of Biochemistry & Cell Biology · doi:10.1016/j.biocel.2005.04.018 · Cited by 1,106
Incidence of hospitalized rhabdomyolysis in patients treated with lipid-lowering drugs
Graham, D. J. (2004) · JAMA · doi:10.1001/jama.292.21.2585 · Cited by 875
Signalling pathways that mediate skeletal muscle hypertrophy and atrophy
Glass, D. J. (2003) · Nature Cell Biology · doi:10.1038/ncb0203-87 · Cited by 694
Research works printed but not linked: 104
- Tobert, 2003, Lovastatin and beyond: the history of the HMG-CoA reductase inhibitors, Nat. Rev. Drug Discov., 2, 517, 10.1038/nrd1112 · DOI · Paper page
- Mabuchi, 1983, Reduction of serum cholesterol in heterozygous patients with familial hypercholesterolemia. Additive effects of compactin and cholestyramine, N. Engl. J. Med., 308, 609, 10.1056/NEJM198303173081101 · DOI
- Alberts, 1980, Mevinolin: a highly potent competitive inhibitor of hydroxymethylglutaryl-coenzyme A reductase and a cholesterol-lowering agent, Proc. Natl. Acad. Sci. U. S. A., 77, 3957, 10.1073/pnas.77.7.3957 · DOI · Paper page
- Goldstein, 2006, Protein sensors for membrane sterols, Cell, 124, 35, 10.1016/j.cell.2005.12.022 · DOI · Paper page
- Walley, 2004, Variations and increase in use of statins across Europe: data from administrative databases, BMJ, 328, 385, 10.1136/bmj.328.7436.385 · DOI
- Stroes, 2015, Statin-associated muscle symptoms: impact on statin therapy-European atherosclerosis society consensus panel statement on assessment, aetiology and management, Eur. Heart J., 36, 1012, 10.1093/eurheartj/ehv043 · DOI · Paper page
- Gu, 2014, Prescription cholesterol-lowering medication use in adults aged 40 and over: United States, 2003-2012, NCHS Data Brief, 177, 1
- Wallach-Kildemoes, 2016, Statin prescribing according to gender, age and indication: what about the benefit-risk balance?, J. Eval. Clin. Pract., 22, 235, 10.1111/jep.12462 · DOI
- Farrer, 2018, Beyond statins: emerging evidence for HDL-Increasing therapies and diet in treating cardiovascular disease, Adv. Prev. Med., 2018, 10.1155/2018/6024747 · DOI
- Eriksson, 2011, Comparative efficacy of pitavastatin and simvastatin in high-risk patients: a randomized controlled trial, Adv. Ther., 28, 811, 10.1007/s12325-011-0056-7 · DOI
- Kikutani, 2016, Involvement of monocarboxylate transporter 4 expression in statin-induced cytotoxicity, J. Pharm. Sci., 105, 1544, 10.1016/j.xphs.2016.01.014 · DOI
- Ratz Bravo, 2005, Prevalence of potentially severe drug-drug interactions in ambulatory patients with dyslipidaemia receiving HMG-CoA reductase inhibitor therapy, Drug Saf., 28, 263, 10.2165/00002018-200528030-00007 · DOI
- Roten, 2004, Rhabdomyolysis in association with simvastatin and amiodarone, Ann. Pharmacother., 38, 978, 10.1345/aph.1D498 · DOI
- McKenney, 2003, Pharmacologic characteristics of statins, Clin. Cardiol., 26
- Profumo, 2014, Pleiotropic effects of statins in atherosclerotic disease: focus on the antioxidant activity of atorvastatin, Curr. Top. Med. Chem., 14, 2542, 10.2174/1568026614666141203130324 · DOI
- Antoniades, 2014, Statins: pleiotropic regulators of cardiovascular redox state, Antioxid. Redox Signal., 20, 1195, 10.1089/ars.2014.5836 · DOI
- Thompson, 2016, Statin-associated side effects, J. Am. Coll. Cardiol., 67, 2395, 10.1016/j.jacc.2016.02.071 · DOI · Paper page
- Maghsoodi, 2016, Statin myopathy: over-rated and under-treated?, Curr. Opin. Cardiol., 31, 417, 10.1097/HCO.0000000000000282 · DOI
- Group, 2002, Long-term effectiveness and safety of pravastatin in 9014 patients with coronary heart disease and average cholesterol concentrations: the LIPID trial follow-up, Lancet, 359, 1379, 10.1016/S0140-6736(02)08351-4 · DOI · Paper page
- Sever, 2003, Lancet, 361, 1149, 10.1016/S0140-6736(03)12948-0 · DOI · Paper page
- Tomlinson, 2005, Potential adverse effects of statins on muscle, Phys. Ther., 85, 459, 10.1093/ptj/85.5.459 · DOI
- Meador, 2010, Statin-associated myopathy and its exacerbation with exercise, Muscle Nerve, 42, 469, 10.1002/mus.21817 · DOI
- Sinzinger, 2004, Professional athletes suffering from familial hypercholesterolaemia rarely tolerate statin treatment because of muscular problems, Br. J. Clin. Pharmacol., 57, 525, 10.1111/j.1365-2125.2003.02044.x · DOI
- Mancini, 2011, Diagnosis, prevention, and management of statin adverse effects and intolerance: proceedings of a Canadian Working Group Consensus Conference, Can. J. Cardiol., 27, 635
- Kipfer, 2015, Immune-mediated necrotising myopathy linked to statin use, Lancet, 386, 10.1016/S0140-6736(15)60068-X · DOI
- Kobayashi, 2008, Association between risk of myopathy and cholesterol-lowering effect: a comparison of all statins, Life Sci., 82, 969, 10.1016/j.lfs.2008.02.019 · DOI
- Mullen, 2010, Effect of simvastatin on cholesterol metabolism in C2C12 myotubes and HepG2 cells, and consequences for statin-induced myopathy, Biochem. Pharmacol., 79, 1200, 10.1016/j.bcp.2009.12.007 · DOI
- Morita, 1997, Enhancement of membrane fluidity in cholesterol-poor endothelial cells pre-treated with simvastatin, Endothelium, 5, 107, 10.3109/10623329709079868 · DOI
- Nakahara, 1994, Cytosolic Ca2+ increase and cell damage in L6 rat myoblasts by HMG-CoA reductase inhibitors, Biochem. Biophys. Res. Commun., 202, 1579, 10.1006/bbrc.1994.2112 · DOI
- Sun, 2018, Atorvastatin impaired glucose metabolism in C2C12 cells partly via inhibiting cholesterol-dependent glucose transporter 4 translocation, Biochem. Pharmacol., 150, 108, 10.1016/j.bcp.2018.01.021 · DOI
- Epstein, 1991, Quantitation of prenylcysteines by a selective cleavage reaction, Proc. Natl. Acad. Sci. U. S. A., 88, 9668, 10.1073/pnas.88.21.9668 · DOI
- Wang, 2016, Protein prenylation: unique fats make their mark on biology, Nat. Rev. Mol. Cell Biol., 17, 110, 10.1038/nrm.2015.11 · DOI · Paper page
- Kato, 1992, Isoprenoid addition to Ras protein is the critical modification for its membrane association and transforming activity, Proc. Natl. Acad. Sci. U. S. A., 89, 6403, 10.1073/pnas.89.14.6403 · DOI · Paper page
- Yang, 2012, Cooperative activation of PI3K by Ras and Rho family small GTPases, Mol. Cell, 47, 281, 10.1016/j.molcel.2012.05.007 · DOI
- Sebti, 2005, Protein farnesylation: implications for normal physiology, malignant transformation, and cancer therapy, Cancer Cell, 7, 297, 10.1016/j.ccr.2005.04.005 · DOI
- du Souich, 2017, Myotoxicity of statins: mechanism of action, Pharmacol. Ther., 175, 1, 10.1016/j.pharmthera.2017.02.029 · DOI
- Flint, 1997, HMG CoA reductase inhibitor-induced myotoxicity: pravastatin and lovastatin inhibit the geranylgeranylation of low-molecular-weight proteins in neonatal rat muscle cell culture, Toxicol. Appl. Pharmacol., 145, 99, 10.1006/taap.1997.8174 · DOI
- Shah, 2018, Ras and Rap1: a tale of two GTPases, Semin. Cancer Biol.
- Jaskiewicz, 2018, Geranylgeraniol prevents statin-dependent myotoxicity in C2C12 muscle cells through RAP1 GTPase prenylation and cytoprotective autophagy, Oxid. Med. Cell. Longev., 2018, 10.1155/2018/6463807 · DOI
- Johnson, 2004, Statins induce apoptosis in rat and human myotube cultures by inhibiting protein geranylgeranylation but not ubiquinone, Toxicol. Appl. Pharmacol., 200, 237, 10.1016/j.taap.2004.04.010 · DOI
- Gee, 2015, Inhibition of prenyltransferase activity by statins in both liver and muscle cell lines is not causative of cytotoxicity, Toxicology, 329, 40, 10.1016/j.tox.2015.01.005 · DOI
- JeBailey, 2007, Ceramide- and oxidant-induced insulin resistance involve loss of insulin-dependent Rac-activation and actin remodeling in muscle cells, Diabetes, 56, 394, 10.2337/db06-0823 · DOI
- Kanzaki, 2006, Insulin receptor signals regulating GLUT4 translocation and actin dynamics, Endocr. J., 53, 267, 10.1507/endocrj.KR-65 · DOI
- Sylow, 2014, Akt and Rac1 signaling are jointly required for insulin-stimulated glucose uptake in skeletal muscle and downregulated in insulin resistance, Cell. Signal., 26, 323, 10.1016/j.cellsig.2013.11.007 · DOI
- Sylow, 2013, Rac1 is a novel regulator of contraction-stimulated glucose uptake in skeletal muscle, Diabetes, 62, 1139, 10.2337/db12-0491 · DOI
- Chiu, 2011, Rac1 signalling towards GLUT4/glucose uptake in skeletal muscle, Cell. Signal., 23, 1546, 10.1016/j.cellsig.2011.05.022 · DOI
- Sylow, 2013, Rac1 signaling is required for insulin-stimulated glucose uptake and is dysregulated in insulin-resistant murine and human skeletal muscle, Diabetes, 62, 1865, 10.2337/db12-1148 · DOI
- Li, 2016, Simvastatin inhibits glucose uptake activity and GLUT4 translocation through suppression of the IR/IRS-1/Akt signaling in C2C12 myotubes, Biomed. Pharmacother., 83, 194, 10.1016/j.biopha.2016.06.029 · DOI
- Baker, 2005, Molecular clues into the pathogenesis of statin-mediated muscle toxicity, Muscle Nerve, 31, 572, 10.1002/mus.20291 · DOI
- Sanvee, 2019, Mechanisms of insulin resistance by simvastatin in C2C12 myotubes and in mouse skeletal muscle, Biochem. Pharmacol., 10.1016/j.bcp.2019.02.025 · DOI
- Nunnari, 2012, Mitochondria: in sickness and in health, Cell, 148, 1145, 10.1016/j.cell.2012.02.035 · DOI · Paper page
- Sirvent, 2008, New insights into mechanisms of statin-associated myotoxicity, Curr. Opin. Pharmacol., 8, 333, 10.1016/j.coph.2007.12.010 · DOI
- Bouitbir, 2016, Statins trigger mitochondrial reactive oxygen species-induced apoptosis in glycolytic skeletal muscle, Antioxid. Redox Signal., 24, 84, 10.1089/ars.2014.6190 · DOI
- Kaufmann, 2006, Toxicity of statins on rat skeletal muscle mitochondria, Cell. Mol. Life Sci., 63, 2415, 10.1007/s00018-006-6235-z · DOI · Paper page
- Mullen, 2011, Susceptibility to simvastatin-induced toxicity is partly determined by mitochondrial respiration and phosphorylation state of Akt, Biochim. Biophys. Acta, 1813, 2079, 10.1016/j.bbamcr.2011.07.019 · DOI
- Singh, 2018, PGC-1beta modulates statin-associated myotoxicity in mice, Arch. Toxicol.
- Apostolopoulou, 2015, The role of mitochondria in statin-induced myopathy, Eur. J. Clin. Invest., 45, 745, 10.1111/eci.12461 · DOI
- Bouitbir, 2012, Mitochondria of trained skeletal muscle are protected from deleterious effects of statins, Muscle Nerve, 46, 367, 10.1002/mus.23309 · DOI
- Phillips, 2002, Statin-associated myopathy with normal creatine kinase levels, Ann. Intern. Med., 137, 581, 10.7326/0003-4819-137-7-200210010-00009 · DOI · Paper page
- Gambelli, 2004, Mitochondrial alterations in muscle biopsies of patients on statin therapy, J. Submicrosc. Cytol. Pathol., 36, 85
- Duncan, 2009, Decreased ubiquinone availability and impaired mitochondrial cytochrome oxidase activity associated with statin treatment, Toxicol. Mech. Methods, 19, 44, 10.1080/15376510802305047 · DOI
- Allard, 2018, Statins affect skeletal muscle performance: evidence for disturbances in energy metabolism, J. Clin. Endocrinol. Metab., 103, 75, 10.1210/jc.2017-01561 · DOI
- Galtier, 2012, Effect of a high dose of simvastatin on muscle mitochondrial metabolism and calcium signaling in healthy volunteers, Toxicol. Appl. Pharmacol., 263, 281, 10.1016/j.taap.2012.06.020 · DOI
- Parker, 2012, Effect of statins on creatine kinase levels before and after a marathon run, Am. J. Cardiol., 109, 282, 10.1016/j.amjcard.2011.08.045 · DOI
- Eijsvogels, 2014, Impact of statin use on exercise-induced cardiac troponin elevations, Am. J. Cardiol., 114, 624, 10.1016/j.amjcard.2014.05.047 · DOI
- Ernster, 1995, Biochemical, physiological and medical aspects of ubiquinone function, Biochim. Biophys. Acta, 1271, 195, 10.1016/0925-4439(95)00028-3 · DOI · Paper page
- Muraki, 1985, Coenzyme Q10 reverses mitochondrial dysfunction in atorvastatin-treated mice and increases exercise endurance, J. Appl. Physiol., 113, 479, 10.1152/japplphysiol.01362.2011 · DOI
- Schaefer, 2004, Evaluation of ubiquinone concentration and mitochondrial function relative to cerivastatin-induced skeletal myopathy in rats, Toxicol. Appl. Pharmacol., 194, 10, 10.1016/j.taap.2003.08.013 · DOI
- Laaksonen, 1995, Serum and muscle tissue ubiquinone levels in healthy subjects, J. Lab. Clin. Med., 125, 517
- Marcoff, 2007, The role of coenzyme Q10 in statin-associated myopathy: a systematic review, J. Am. Coll. Cardiol., 49, 2231, 10.1016/j.jacc.2007.02.049 · DOI · Paper page
- Paiva, 2005, High-dose statins and skeletal muscle metabolism in humans: a randomized, controlled trial, Clin. Pharmacol. Ther., 78, 60, 10.1016/j.clpt.2005.03.006 · DOI · Paper page
- Bogsrud, 2013, No effect of combined coenzyme Q10 and selenium supplementation on atorvastatin-induced myopathy, Scand. Cardiovasc. J., 47, 80, 10.3109/14017431.2012.756119 · DOI · Paper page
- Caso, 2007, Effect of coenzyme q10 on myopathic symptoms in patients treated with statins, Am. J. Cardiol., 99, 1409, 10.1016/j.amjcard.2006.12.063 · DOI · Paper page
- Tricarico, 2015, Mevalonate pathway blockade, mitochondrial dysfunction and autophagy: a possible link, Int. J. Mol. Sci., 16, 16067, 10.3390/ijms160716067 · DOI
- Puigserver, 2003, Peroxisome proliferator-activated receptor-gamma coactivator 1 alpha (PGC-1 alpha): transcriptional coactivator and metabolic regulator, Endocr. Rev., 24, 78, 10.1210/er.2002-0012 · DOI · Paper page
- Meador, 2011, Statin-associated changes in skeletal muscle function and stress response after novel or accustomed exercise, Muscle Nerve, 44, 882, 10.1002/mus.22236 · DOI
- Ryter, 2019, Autophagy: a lysosome-dependent process with implications in cellular redox homeostasis and human disease, Antioxid. Redox Signal., 30, 138, 10.1089/ars.2018.7518 · DOI
- Halling, 2016, PGC-1alpha promotes exercise-induced autophagy in mouse skeletal muscle, Physiol. Rep., 4, 10.14814/phy2.12698 · DOI
- Andres, 2014, Mitophagy is required for acute cardioprotection by simvastatin, Antioxid. Redox Signal., 21, 1960, 10.1089/ars.2013.5416 · DOI
- Balaban, 2005, Mitochondria, oxidants, and aging, Cell, 120, 483, 10.1016/j.cell.2005.02.001 · DOI · Paper page
- Anderson, 2006, Type II skeletal myofibers possess unique properties that potentiate mitochondrial H(2)O(2) generation, Am. J. Physiol., Cell Physiol., 290, C844, 10.1152/ajpcell.00402.2005 · DOI · Paper page
- Shadel, 2015, Mitochondrial ROS signaling in organismal homeostasis, Cell, 163, 560, 10.1016/j.cell.2015.10.001 · DOI · Paper page
- Schirris, 2015, Statin-induced myopathy is associated with mitochondrial complex III inhibition, Cell Metab., 22, 399, 10.1016/j.cmet.2015.08.002 · DOI
- Westwood, 2005, Statin-induced muscle necrosis in the rat: distribution, development, and fibre selectivity, Toxicol. Pathol., 33, 246, 10.1080/01926230590908213 · DOI
- Seachrist, 2005, Roles of exercise and pharmacokinetics in cerivastatin-induced skeletal muscle toxicity, Toxicol. Sci., 88, 551, 10.1093/toxsci/kfi305 · DOI
- Ji, 1991, Effect of exercise training on antioxidant and metabolic functions in senescent rat skeletal muscle, Gerontology, 37, 317, 10.1159/000213279 · DOI
- Dirks, 2006, Statin-induced apoptosis and skeletal myopathy, Am. J. Physiol. Cell Physiol., 291, C1208, 10.1152/ajpcell.00226.2006 · DOI
- Sacher, 2005, Delineation of myotoxicity induced by 3-hydroxy-3-methylglutaryl CoA reductase inhibitors in human skeletal muscle cells, J. Pharmacol. Exp. Ther., 314, 1032, 10.1124/jpet.105.086462 · DOI
- Bonifacio, 2015, The AKT/mTOR signaling pathway plays a key role in statin-induced myotoxicity, Biochim. Biophys. Acta, 1853, 1841, 10.1016/j.bbamcr.2015.04.010 · DOI
- Zorov, 2014, Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release, Physiol. Rev., 94, 909, 10.1152/physrev.00026.2013 · DOI · Paper page
- Yu, 2016, Proliferation, survival and metabolism: the role of PI3K/AKT/mTOR signalling in pluripotency and cell fate determination, Development, 143, 3050, 10.1242/dev.137075 · DOI · Paper page
- Bonifacio, 2017, IGF-1 prevents simvastatin-induced myotoxicity in C2C12 myotubes, Arch. Toxicol., 91, 2223, 10.1007/s00204-016-1871-z · DOI
- Verhees, 2011, Glycogen synthase kinase-3beta is required for the induction of skeletal muscle atrophy, Am. J. Physiol. Cell Physiol., 301, C995, 10.1152/ajpcell.00520.2010 · DOI
- Hanai, 2007, The muscle-specific ubiquitin ligase atrogin-1/MAFbx mediates statin-induced muscle toxicity, J. Clin. Invest., 117, 3940
- Mallinson, 2009, Blunted Akt/FOXO signalling and activation of genes controlling atrophy and fuel use in statin myopathy, J. Physiol., 587, 219, 10.1113/jphysiol.2008.164699 · DOI
- Tuckow, 2011, Simvastatin represses protein synthesis in the muscle-derived C(2)C(1)(2) cell line with a concomitant reduction in eukaryotic initiation factor 2B expression, Am. J. Physiol. Endocrinol. Metab., 300, E564, 10.1152/ajpendo.00383.2010 · DOI
- Cederberg, 2015, Increased risk of diabetes with statin treatment is associated with impaired insulin sensitivity and insulin secretion: a 6 year follow-up study of the METSIM cohort, Diabetologia, 58, 1109, 10.1007/s00125-015-3528-5 · DOI · Paper page
- Ridker, 2012, Cardiovascular benefits and diabetes risks of statin therapy in primary prevention: an analysis from the JUPITER trial, Lancet, 380, 565, 10.1016/S0140-6736(12)61190-8 · DOI · Paper page
- Satoh, 2014, Molecular mechanisms for the regulation of insulin-stimulated glucose uptake by small guanosine triphosphatases in skeletal muscle and adipocytes, Int. J. Mol. Sci., 15, 18677, 10.3390/ijms151018677 · DOI
- Hajduch, 2001, Protein kinase B (PKB/Akt)--a key regulator of glucose transport?, FEBS Lett., 492, 199, 10.1016/S0014-5793(01)02242-6 · DOI · Paper page
- Miinea, 2005, AS160, the Akt substrate regulating GLUT4 translocation, has a functional Rab GTPase-activating protein domain, Biochem. J., 391, 87, 10.1042/BJ20050887 · DOI · Paper page
- Smith, 2014, Simvastatin inhibits glucose metabolism and legumain activity in human myotubes, PLoS One, 9, 10.1371/journal.pone.0085721 · DOI
- Yaluri, 2016, Simvastatin induces insulin resistance in L6 skeletal muscle myotubes by suppressing insulin signaling, GLUT4 expression and GSK-3beta phosphorylation, Biochem. Biophys. Res. Commun., 480, 194, 10.1016/j.bbrc.2016.10.026 · DOI
- Sacheck, 2004, IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1, Am. J. Physiol. Endocrinol. Metab., 287, E591, 10.1152/ajpendo.00073.2004 · DOI · Paper page
Cited by
The Use of Medications and Dietary Supplements by Masters Athletes—A Review
Harnett, J., Climstein, M., Walsh, J., & Gifford, J. (2022) · Current Nutrition Reports · doi:10.1007/s13668-022-00398-0 · Cited by 12
Metadata
- Authors
- Bouitbir, J., Sanvee, G. M., Panajatovic, M. V., Singh, F., & Krähenbühl, S.
- Venue
- Pharmacological Research
- Publisher
- Elsevier BV
- Published
- 2020-04
- Volume
- 154
- Pages
- 104201
- ISSN
- 1043-6618
- Cited by
- 136
- References
- 35
- Language
- en
- Licence
- www.elsevier.com/tdm/userlicense/1.0/ (tdm)
- www.elsevier.com/legal/tdmrep-license (tdm)
- Funders
- Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung (31003A-156270)
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