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Damage to DNA by reactive oxygen and nitrogen species: role in inflammatory disease and progression to cancer
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Review Article| January 01 1996 Damage to DNA by reactive oxygen and nitrogen species: role in inflammatory disease and progression to cancer Helen WISEMAN; Helen WISEMAN *Department of Nutrition and Dietetics, King's College London, Campden Hill Road, London W8 7AH, U.K. Search for other works by this author on: This Site PubMed Google Scholar Barry HALLIWELL Barry HALLIWELL †Pharmacology Group, King's College London, Manresa Road, London SW3 6LX, U.K. Search for other works by this author on: This Site PubMed Google Scholar Biochem J (1996) 313 (1): 17–29. https://doi.org/10.1042/bj3130017 Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Facebook Twitter LinkedIn Email Cite Icon Cite Get Permissions Citation Helen WISEMAN, Barry HALLIWELL; Damage to DNA by reactive oxygen and nitrogen species: role in inflammatory disease and progression to cancer. Biochem J 1 January 1996; 313 (1): 17–29. doi: https://doi.org/10.1042/bj3130017 Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsBiochemical Journal Search Advanced Search This content is only available as a PDF. The Biochemical Society, London © 19961996 Article PDF first page preview Close Modal You do not currently have access to this content.
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“A number of endogenous factors such as free radicals [34] generated during metabolic processes as well as exogenous factors such as UV or ionizing radiations [35] are known to interfere with genome integrity.”
From Molecular Mechanisms of Ultraviolet Radiation‐Induced DNA Damage and Repair (2010) · cites this paper as [34]
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“At low to moderate levels, ROS act as signaling transducers to activate cancer cell proliferation, migration, invasion, angiogenesis, and drug resistance.2, 6, 7”
From Reactive Oxygen Species in Cancer: Current Findings and Future Directions (2021) · cites this paper as [6]
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Metadata
- Authors
- WISEMAN, H., & HALLIWELL, B.
- Affiliations
- King's College London (GB)
- Corresponding author
- WISEMAN, Helen
- ORCID
- HALLIWELL, Barry
- Venue
- Biochemical Journal
- Publisher
- Portland Press Ltd.
- Published
- 1996-01-01
- Volume
- 313
- Issue
- 1
- Pages
- 17-29
- ISSN
- 0264-6021 (ISSN-L)
- 1470-8728
- PMC
- PMC1216878
- OpenAlex
- W2164613158
- Cited by
- 2,345
- References
- 204
- Citations by year
- 2026: 18 · 2025: 37 · 2024: 42 · 2023: 36 · 2022: 56 · 2021: 70 · 2020: 77 · 2019: 95 · 2018: 111 · 2017: 132 · 2016: 107 · 2015: 129 · 2014: 129 · 2013: 124 · 2012: 120
- FWCI
- 4.58
- Type
- journal article
- Language
- en
- Open access
- bronze
- doi.org
- Locations
- Biochemical Journal (publishedVersion)
- PubMed Central (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Redox biology and oxidative stress
- Health Sciences › Medicine › Biochemistry (medical) › Antioxidant Activity and Oxidative Stress
- Life Sciences › Immunology and Microbiology › Immunology › Neutrophil, Myeloperoxidase and Oxidative Mechanisms
- Keywords
- DNA damage, reactive oxygen species, reactive nitrogen species, inflammatory diseases, tumor progression, oxidative stress, inflammation and carcinogenesis
- MeSH terms
- Animals, DNA, DNA Damage, Free Radicals, Humans, Inflammation, Neoplasms, Nitrogen Oxides, Reactive Oxygen Species, Disease Progression
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