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Cited by 92

The DOI matches its Crossref record; 19 of the 19 references it cites were found in a registry or an index.

Roles of SIRT1 in the Acute and Restorative Phases following Induction of Inflammation

Zhang, Zhiyong, Lowry, Stephen F., Guarente, Leonard, Haimovich, Beatrice·2010·Journal of Biological Chemistry
DOI 10.1074/jbc.m110.174482
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Crossref · 10.1074/jbc.m110.174482

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  • Pharmacologic activation of SIRT1 may prevent inflammatory responses in mice.

    “pharmacologic activation of SIRT1 may prevent inflammatory responses in mice”

    From The Hallmarks of Aging (2013) · cites this paper as Zhang et al., 2010

    The Results section reports that resveratrol pretreatment prevented several endotoxin-induced changes in mice, including ATP decline and increased autophagy and HIF-1α expression; these protective effects depended on SIRT1. It does not show that resveratrol prevented the inflammatory response itself or establish that its effect came from direct pharmacologic activation of SIRT1. (LLM, conf=0.84)

    Experimental — accuracy not yet measured

  • Reduction of SIRT1 levels correlates with the development and progression of many inflammatory diseases.

    “reduction of SIRT1 levels correlates with the development and progression of many inflammatory diseases”

    From The Hallmarks of Aging (2013) · cites this paper as Zhang et al., 2010

    The Discussion reports that endotoxin-induced systemic inflammation caused a robust decrease in SIRT1 expression in mouse liver. It only speculates that similar metabolic perturbations may contribute to many diseases; it does not establish a correlation between reduced SIRT1 and the development or progression of many inflammatory diseases. (LLM, conf=0.88)

    Experimental — accuracy not yet measured

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Metadata

Authors
Zhang, Z., Lowry, S. F., Guarente, L., & Haimovich, B.
Venue
Journal of Biological Chemistry
Publisher
Elsevier BV
Published
2010-12
Volume
285
Issue
53
Pages
41391-41401
ISSN
0021-9258
DOI
10.1074/jbc.m110.174482
Cited by
92
References
19
Language
en
Licence

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