bHLH Transcription Factor Olig1 Is Required to Repair Demyelinated Lesions in the CNS

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Items 101–200 of 416

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    Sun, Yu, Meijer, Dimphna H., Alberta, John A., Mehta, Shwetal, Kane, Michael F., Tien, An-Chi, Fu, Hui, Petryniak, Magdalena A., Potter, Gregory B., Liu, Zijing et al. · 2011 · Neuron
  4. Nogo receptor blockade overcomes remyelination failure after white matter stroke and stimulates functional recovery in aged mice
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  5. Effects of progesterone on oligodendrocyte progenitors, oligodendrocyte transcription factors, and myelin proteins following spinal cord injury
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  6. β1-Integrin Signaling Mediates Premyelinating Oligodendrocyte Survival But Is Not Required for CNS Myelination and Remyelination
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  7. Oligodendrocyte progenitor cell recruitment and remyelination in multiple sclerosis: the more, the merrier?
    Vanja Tepavčević, Catherine Lubetzki · 2022 · Brain
  8. Paradoxical dysregulation of the neural stem cell pathway sonic hedgehog‐gli1 in autoimmune encephalomyelitis and multiple sclerosis
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  9. IκB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-κB in the central nervous system
    Raasch, Jenni, Zeller, Nicolas, van Loo, Geert, Merkler, Doron, Mildner, Alexander, Erny, Daniel, Knobeloch, Klaus-Peter, Bethea, John R., Waisman, Ari, Knust, Markus et al. · 2011 · Brain
  10. Coherent anti‐stokes Raman scattering imaging of myelin degradation reveals a calcium‐dependent pathway in lyso‐PtdCho‐induced demyelination
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  14. Co‐localization of Nkx6.2 and Nkx2.2 homeodomain proteins in differentiated myelinating oligodendrocytes
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  15. The bHLH transcription factor Olig3 marks the dorsal neuroepithelium of the hindbrain and is essential for the development of brainstem nuclei
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  35. Olig1 Function Is Required to Repress Dlx1/2 and Interneuron Production in Mammalian Brain
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  40. Child abuse associates with an imbalance of oligodendrocyte-lineage cells in ventromedial prefrontal white matter
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  41. Small-molecule-induced epigenetic rejuvenation promotes SREBP condensation and overcomes barriers to CNS myelin regeneration
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  42. Loss of G as6 and A xl signaling results in extensive axonal damage, motor deficits, prolonged neuroinflammation, and less remyelination following cuprizone exposure
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  49. An experimental model of secondary progressive multiple sclerosis that shows regional variation in gliosis, remyelination, axonal and neuronal loss
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  52. Cytoplasmic translocation of Olig2 in adult glial progenitors marks the generation of reactive astrocytes following autoimmune inflammation
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