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The DOI matches its Crossref record; 41 of the 41 references it cites were found in a registry or an index.

TREM2‐dependent effects on microglia in Alzheimer's disease

Zhou, Yingyue, Ulland, Tyler K., Colonna, Marco·2018·Frontiers in Aging Neuroscience
DOI 10.3389/fnagi.2018.00202PMC PMC6046445
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Research works printed but not linked: 32

  1. Bemiller, 2017, TREM2 deficiency exacerbates tau pathology through dysregulated kinase signaling in a mouse model of tauopathy, Mol. Neurodegener., 12, 74, 10.1186/s13024-017-0216-6 · DOI · Paper page
  2. Bertram, 2010, The genetics of Alzheimer disease: back to the future, Neuron, 68, 270, 10.1016/j.neuron.2010.10.013 · DOI · Paper page
  3. Caccamo, 2017, p62 improves AD-like pathology by increasing autophagy, Mol. Psychiatry, 22, 865, 10.1038/mp.2016.139 · DOI
  4. Cady, 2014, TREM2 variant p.R47H as a risk factor for sporadic amyotrophic lateral sclerosis, JAMA Neurol., 71, 449, 10.1001/jamaneurol.2013.6237 · DOI · Paper page
  5. Colonna, 2016, TREM2 variants: new keys to decipher Alzheimer disease pathogenesis, Nat. Rev. Neurosci., 17, 201, 10.1038/nrn.2016.7 · DOI · Paper page
  6. Condello, 2015, Microglia constitute a barrier that prevents neurotoxic protofibrillar Aβ42 hotspots around plaques, Nat. Commun., 6, 6176, 10.1038/ncomms7176 · DOI · Paper page
  7. Frank, 2008, TREM2 is upregulated in amyloid plaque-associated microglia in aged APP23 transgenic mice, Glia, 56, 1438, 10.1002/glia.20710 · DOI · Paper page
  8. Ginhoux, 2010, Fate mapping analysis reveals that adult microglia derive from primitive macrophages, Science, 330, 841, 10.1126/science.1194637 · DOI · Paper page
  9. Hollingworth, 2011, Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer’s disease, Nat. Genet., 43, 429, 10.1038/ng.803 · DOI · Paper page
  10. Jay, 2017, Disease progression-dependent effects of TREM2 deficiency in a mouse model of Alzheimer’s disease, J. Neurosci., 37, 637, 10.1523/JNEUROSCI.2110-16.2016 · DOI · Paper page
  11. Jonsson, 2013, Variant of TREM2 associated with the risk of Alzheimer’s disease, N. Engl. J. Med., 368, 107, 10.1056/NEJMoa1211103 · DOI · Paper page
  12. Kabeya, 2000, LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing, EMBO J., 19, 5720, 10.1093/emboj/19.21.5720 · DOI · Paper page
  13. Kleinberger, 2017, The FTD-like syndrome causing TREM2 T66M mutation impairs microglia function, brain perfusion, and glucose metabolism, EMBO J., 36, 1837, 10.15252/embj.201796516 · DOI
  14. Lessard, 2018, High affinity interactions and signal transduction between Aβ oligomers and TREM2, BioRxiv, 10.1101/269787 · DOI
  15. Mizushima, 2010, Methods in mammalian autophagy research, Cell, 140, 313, 10.1016/j.cell.2010.01.028 · DOI · Paper page
  16. N’Diaye, 2009, TREM-2 (triggering receptor expressed on myeloid cells 2) is a phagocytic receptor for bacteria, J. Cell Biol., 184, 215, 10.1083/jcb.200808080 · DOI
  17. Otero, 2012, TREM2 and β-catenin regulate bone homeostasis by controlling the rate of osteoclastogenesis, J. Immunol., 188, 2612, 10.4049/jimmunol.1102836 · DOI
  18. Parajuli, 2013, Oligomeric amyloid β induces IL-1β processing via production of ROS: implication in Alzheimer’s disease, Cell Death Dis., 4, e975, 10.1038/cddis.2013.503 · DOI · Paper page
  19. Parkhurst, 2013, Microglia promote learning-dependent synapse formation through BDNF, Cell, 155, 1596, 10.1016/j.cell.2013.11.030 · DOI · Paper page
  20. Peng, 2010, TREM2- and DAP12-dependent activation of PI3K requires DAP10 and is inhibited by SHIP1, Sci. Signal., 3, ra38, 10.1126/scisignal.2000500 · DOI · Paper page
  21. Pickford, 2008, The autophagy-related protein beclin 1 shows reduced expression in early Alzheimer disease and regulates amyloid β accumulation in mice, J. Clin. Invest., 118, 2190, 10.1172/JCI33585 · DOI · Paper page
  22. Stevens, 2007, The classical complement cascade mediates CNS synapse elimination, Cell, 131, 1164, 10.1016/j.cell.2007.10.036 · DOI · Paper page
  23. Strittmatter, 1993, Apolipoprotein E: high-avidity binding to β-amyloid and increased frequency of type 4 allele in late-onset familial Alzheimer disease, Proc. Natl. Acad. Sci. U S A, 90, 1977, 10.1073/pnas.90.5.1977 · DOI · Paper page
  24. Takasugi, 2003, The role of presenilin cofactors in the γ-secretase complex, Nature, 422, 438, 10.1038/nature01506 · DOI
  25. Thelen, 2014, Investigation of the role of rare TREM2 variants in frontotemporal dementia subtypes, Neurobiol. Aging, 35, 2657.e13, 10.1016/j.neurobiolaging.2014.06.018 · DOI
  26. Turnbull, 2006, Cutting edge: TREM-2 attenuates macrophage activation, J. Immunol., 177, 3520, 10.4049/jimmunol.177.6.3520 · DOI · Paper page
  27. Venegas, 2017, Microglia-derived ASC specks cross-seed amyloid-β in Alzheimer’s disease, Nature, 552, 355, 10.1038/nature25158 · DOI · Paper page
  28. Wang, 2015, TREM2 lipid sensing sustains the microglial response in an Alzheimer’s disease model, Cell, 160, 1061, 10.1016/j.cell.2015.01.049 · DOI · Paper page
  29. Wang, 2012, IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia, Nat. Immunol., 13, 753, 10.1038/ni.2360 · DOI · Paper page
  30. Yuan, 2016, TREM2 haplodeficiency in mice and humans impairs the microglia barrier function leading to decreased amyloid compaction and severe axonal dystrophy, Neuron, 90, 724, 10.1016/j.neuron.2016.05.003 · DOI · Paper page
  31. Zhao, 2018, TREM2 is a receptor for β-amyloid that mediates microglial function, Neuron, 97, 1023.e7, 10.1016/j.neuron.2018.01.031 · DOI · Paper page
  32. Zhong, 2017, Soluble TREM2 induces inflammatory responses and enhances microglial survival, J. Exp. Med., 214, 597, 10.1084/jem.20160844 · DOI

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Metadata

Authors
Zhou, Y., Ulland, T. K., & Colonna, M.
Venue
Frontiers in Aging Neuroscience
Publisher
Frontiers Media SA
Published
2018-07-09
Volume
10
ISSN
1663-4365
DOI
10.3389/fnagi.2018.00202
PMC
PMC6046445
Cited by
82
References
41
Licence
creativecommons.org/licenses/by/4.0/ (unspecified)
Funders
  • National Institutes of Health (RF1AG05148501)
  • Cure Alzheimer’s Fund

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