Holloszy, John O.

Improvement in Glucose Tolerance After 1 Wk of Exercise in Patients With Mild NIDDM

Rogers, M. A., Yamamoto, C., King, D. S., Hagberg, J. M., Ehsani, A. A., & Holloszy, J. O. (1988) · Diabetes Care

Physical Activity and HealthDiabetes, Cardiovascular Risks, and Lipoproteins

Cited by 201

Adaptation of skeletal muscle to endurance exercise

HOLLOSZY, J. O. (1975) · Medicine and Science in Sports and Exercise

Cardiovascular and exercise physiologyExercise and Physiological Responses

Cited by 186

The regulation of carbohydrate and fat metabolism during and after exercise

Holloszy, J. O. (1998) · Frontiers in bioscience

Muscle metabolism and nutritionCardiovascular and exercise physiology

Cited by 180

Skeletal muscle “mitochondrial deficiency” does not mediate insulin resistance

Holloszy, J. O. (2009) · The American Journal of Clinical Nutrition

Adipose Tissue and MetabolismMetabolism, Diabetes, and Cancer

Cited by 175

Effect of Long-Term Calorie Restriction With Adequate Protein and Micronutrients on Thyroid Hormones

Fontana, L., Klein, S., Holloszy, J. O., & Premachandra, B. N. (2006) · The Journal of Clinical Endocrinology & Metabolism

Thyroid Disorders and TreatmentsGenetics, Aging, and Longevity in Model Organisms

Cited by 149

The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2

Gan, Z., Burkart-Hartman, E. M., Han, D.-H., Finck, B., Leone, T. C., Smith, E. Y., Ayala, J. E., Holloszy, J., & Kelly, D. P. (2011) · Genes & Development

Peroxisome Proliferator-Activated ReceptorsMetabolism, Diabetes, and Cancer

Cited by 138

The Biology of Aging

Holloszy, J. O. (2000) · Mayo Clinic Proceedings

Genetics, Aging, and Longevity in Model OrganismsAging and Gerontology Research

Cited by 136

Does calorie restriction induce mitochondrial biogenesis? A reevaluation

Hancock, C. R., Han, D., Higashida, K., Kim, S. H., & Holloszy, J. O. (2011) · The FASEB Journal

Mitochondrial Function and PathologyGenetics, Aging, and Longevity in Model Organisms

Cited by 131

DHEA enhances effects of weight training on muscle mass and strength in elderly women and men

Villareal, D. T., & Holloszy, J. O. (2006) · American Journal of Physiology-Endocrinology and Metabolism

Hormonal and reproductive studiesMuscle metabolism and nutrition

Cited by 116

Aerobic power and insulin action improve in response to endurance exercise training in healthy 77–87 yr olds

Evans, E. M., Racette, S. B., Peterson, L. R., Villareal, D. T., Greiwe, J. S., & Holloszy, J. O. (2005) · Journal of Applied Physiology

Cardiovascular and exercise physiologyPhysical Activity and Health

Cited by 112

“Deficiency” of Mitochondria in Muscle Does Not Cause Insulin Resistance

Holloszy, J. O. (2013) · Diabetes

Adipose Tissue and MetabolismMetabolism, Diabetes, and Cancer

Cited by 111

Influence of Weekend Lifestyle Patterns on Body Weight

Racette, S. B., Weiss, E. P., Schechtman, K. B., Steger‐May, K., Villareal, D. T., Obert, K. A., Holloszy, J. O., & Washington University School of Medicine CALERIE Team (2008) · Obesity

Obesity, Physical Activity, DietPhysical Activity and Health

Cited by 106

Prevention of glycogen supercompensation prolongs the increase in muscle GLUT4 after exercise

Garcia-Roves, P. M., Han, D.-H., Song, Z., Jones, T. E., Hucker, K. A., & Holloszy, J. O. (2003) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerMuscle metabolism and nutrition

Cited by 93

Deficiency of the Mitochondrial Electron Transport Chain in Muscle Does Not Cause Insulin Resistance

Han, D.-H., Hancock, C. R., Jung, S. R., Higashida, K., Kim, S. H., & Holloszy, J. O. (2011) · PLoS ONE

Mitochondrial Function and PathologyMetabolism, Diabetes, and Cancer

Cited by 71

Calpain System Regulates Muscle Mass and Glucose Transporter GLUT4 Turnover

Otani, K., Han, D.-H., Ford, E. L., Garcia-Roves, P. M., Ye, H., Horikawa, Y., Bell, G. I., Holloszy, J. O., & Polonsky, K. S. (2004) · Journal of Biological Chemistry

Calpain Protease Function and RegulationMetabolism, Diabetes, and Cancer

Cited by 67

UCP-mediated energy depletion in skeletal muscle increases glucose transport despite lipid accumulation and mitochondrial dysfunction

Han, D.-H., Nolte, L. A., Ju, J.-S., Coleman, T., Holloszy, J. O., & Semenkovich, C. F. (2004) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerAdipose Tissue and Metabolism

Cited by 49

Modest Lifestyle Intervention and Glucose Tolerance in Obese African Americans

Racette, S. B., Weiss, E. P., Obert, K. A., Kohrt, W. M., & Holloszy, J. O. (2001) · Obesity Research

Diabetes, Cardiovascular Risks, and LipoproteinsObesity, Physical Activity, Diet

Cited by 46

IL-6 increases muscle insulin sensitivity only at superphysiological levels

Geiger, P. C., Hancock, C., Wright, D. C., Han, D.-H., & Holloszy, J. O. (2007) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerExercise and Physiological Responses

Cited by 36

Exercise and food restriction in rats

Holloszy, J. O. (1992) · The Journal of Nutrition

Genetics, Aging, and Longevity in Model OrganismsDietary Effects on Health

Cited by 30

Inhibition of calpain results in impaired contraction-stimulated GLUT4 translocation in skeletal muscle

Otani, K., Polonsky, K. S., Holloszy, J. O., & Han, D.-H. (2006) · American Journal of Physiology-Endocrinology and Metabolism

Metabolism, Diabetes, and CancerCalpain Protease Function and Regulation

Cited by 14