The DOI matches its Crossref record; 14 of the 14 references it cites were found in a registry or an index.
Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus
Overview
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
In the citing paper
“T cells in SLE are characterized by chronic activation and a heavy reliance on oxidative phosphorylation for ATP production, thus increasing mtROS production”
From The role of mitochondria in rheumatic diseases (2022) · cites this paper as [34]
In the citing paper
“Other abnormalities in T cells in SLE include mitochondrial hyperpolarization and ATP depletion, potentially caused by defective activity of the F0F1–ATP synthase34,73”
From The role of mitochondria in rheumatic diseases (2022) · cites this paper as [34]
In the citing paper
“Data from in vivo studies indicate that this increased oxidative stress leads to exhaustion of the antioxidant capacity of the T cells34”
From The role of mitochondria in rheumatic diseases (2022) · cites this paper as [34]
In the citing paper
“Mitochondria in T cells from patients with SLE have various defects of the electron transfer chain, which lead to hyperpolarization of the mitochondria, the production of mtROS and downstream dysregulation of apoptosis34,141”
From The role of mitochondria in rheumatic diseases (2022) · cites this paper as [34]
In the citing paper
“These defects might hinder activation-induced cell death, while promoting necrotic cell death, thus resulting in the persistence of autoreactive lymphocytes and the generation of substrates for autoantibodies34,73”
From The role of mitochondria in rheumatic diseases (2022) · cites this paper as [34]
Citations
Cites
Apoptosis in the Pathogenesis and Treatment of Disease
Thompson, C. B. (1995) · Science · doi:10.1126/science.7878464 · Cited by 6,524
Nagata, S., & Golstein, P. (1995) · Science · doi:10.1126/science.7533326 · Cited by 3,979
NF-κB: a key role in inflammatory diseases
Tak, P. P., & Firestein, G. S. (2001) · Journal of Clinical Investigation · doi:10.1172/jci11830 · Cited by 3,918
Measuring the Functional Components of Social Support
Cohen, S., Mermelstein, R., Kamarck, T., & Hoberman, H. M. (1985) · Social Support: Theory, Research and Applications · doi:10.1007/978-94-009-5115-0_5 · Cited by 2,296
The mechanism of action of cyclosporin A and FK506
Schreiber, S. L., & Crabtree, G. R. (1992) · Immunology Today · doi:10.1016/0167-5699(92)90111-j · Cited by 2,127
Prospects for whole-genome linkage disequilibrium mapping of common disease genes
Kruglyak, L. (1999) · Nature Genetics · doi:10.1038/9642 · Cited by 1,416
Phagocytosis and clearance of apoptotic cells is mediated by MER
Scott, R. S., McMahon, E. J., Pop, S. M., Reap, E. A., Caricchio, R., Cohen, P. L., Earp, H. S., & Matsushima, G. K. (2001) · Nature · doi:10.1038/35075603 · Cited by 1,215
Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation
Demetriou, M., Granovsky, M., Quaggin, S., & Dennis, J. W. (2001) · Nature · doi:10.1038/35055582 · Cited by 901
Garnero, P., Piperno, M., Gineyts, E., Christgau, S., Delmas, P. D., & Vignon, E. (2001) · Annals of the Rheumatic Diseases · doi:10.1136/ard.60.6.619 · Cited by 475
Self-citationMunger, R. G., Folsom, A. R., Kushi, L. H., Kaye, S. A., & Sellers, T. A. (1992) · American Journal of Epidemiology · doi:10.1093/oxfordjournals.aje.a116485 · Cited by 353
Self-citation
Cited by
Sorted by citations
Sex differences in immune responses
Klein, S. L., & Flanagan, K. L. (2016) · Nature Reviews Immunology · doi:10.1038/nri.2016.90 · Cited by 6,370
Boutron, I., Moher, D., Altman, D. G., Schulz, K. F., Ravaud, P., & for the CONSORT Group* (2008) · Annals of Internal Medicine · doi:10.7326/0003-4819-148-4-200802190-00008 · Cited by 2,292
Victora, G. D., & Nussenzweig, M. C. (2012) · Annual Review of Immunology · doi:10.1146/annurev-immunol-020711-075032 · Cited by 1,878
Lood, C., Blanco, L. P., Purmalek, M. M., Carmona-Rivera, C., De Ravin, S. S., Smith, C. K., Malech, H. L., Ledbetter, J. A., Elkon, K. B., & Kaplan, M. J. (2016) · Nature Medicine · doi:10.1038/nm.4027 · Cited by 1,570
HMGB1 and RAGE in Inflammation and Cancer
Sims, G. P., Rowe, D. C., Rietdijk, S. T., Herbst, R., & Coyle, A. J. (2010) · Annual Review of Immunology · doi:10.1146/annurev.immunol.021908.132603 · Cited by 1,436
Metadata
- Authors
- Gergely, P., Grossman, C., Niland, B., Puskas, F., Neupane, H., Allam, F., Banki, K., Phillips, P. E., & Perl, A.
- Venue
- Arthritis & Rheumatism
- Publisher
- Wiley
- Published
- 2002-01
- Volume
- 46
- Issue
- 1
- Pages
- 175-190
- OpenAlex
- W7132852040
- Cited by
- 5
- References
- 14
- Citations by year
- 2025: 1 · 2023: 1 · 2022: 1
- Type
- other
- Language
- en
- Open access
- closed
- Locations
- Pure Amsterdam UMC (submittedVersion)
- Topics
- Health Sciences › Medicine › Rheumatology › Rheumatoid Arthritis Research and Therapies
- Health Sciences › Medicine › Physiology (medical) › Cardiovascular and exercise physiology
- Health Sciences › Medicine › Public Health, Environmental and Occupational Health › Physical Activity and Health
- Keywords
- dynamic strength training, rheumatoid arthritis, functional capacity, resistance training, exercise therapy
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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Cite CoChat for the checks on this page, and the paper for its own findings.
- This page
- CoChat. (2026). Citation check: Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus. https://app.cochat.ai/papers/10.1002/1529-0131(200201)46:1
- The paper
- Gergely, P., Grossman, C., Niland, B., Puskas, F., Neupane, H., Allam, F., Banki, K., Phillips, P. E., & Perl, A. (2002). Mitochondrial hyperpolarization and ATP depletion in patients with systemic lupus erythematosus. Arthritis & Rheumatism. https://doi.org/10.1002/1529-0131(200201)46%3A1
