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Neonatal Exposure to Perfluorooctane Sulfonate (PFOS) and Perfluorooctanoic Acid (PFOA) Causes Neurobehavioural Defects in Adult Mice

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Perfluorooctanesulfonate and Related Fluorinated Hydrocarbons in Marine Mammals, Fishes, and Birds from Coasts of the Baltic and the Mediterranean Seas

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Neonatal Exposure to PFOS and PFOA in Mice Results in Changes in Proteins which are Important for Neuronal Growth and Synaptogenesis in the Developing Brain

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The Applicability of Biomonitoring Data for Perfluorooctanesulfonate to the Environmental Public Health Continuum

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Knockdown of AMPA Receptor GluR2 Expression Causes Delayed Neurodegeneration and Increases Damage by Sublethal Ischemia in Hippocampal CA1 and CA3 Neurons

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Acute Enhancement of Synaptic Transmission and Chronic Inhibition of Synaptogenesis Induced by Perfluorooctane Sulfonate through Mediation of Voltage-dependent Calcium Channel

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Long-term exposure to endogenous levels of tributyltin decreases GluR2 expression and increases neuronal vulnerability to glutamate

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Prenatal exposure to perfluorooctanesulfonate in rat resulted in long‐lasting changes of expression of synapsins and synaptophysin

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