Nampt/PBEF/Visfatin Regulates Insulin Secretion in β Cells as a Systemic NAD Biosynthetic Enzyme

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Items 101–200 of 912

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3. TARGETED, LCMS-BASED METABOLOMICS FOR QUANTITATIVE MEASUREMENT OF NAD + METABOLITES
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4. NAD + metabolism: A therapeutic target for age-related metabolic disease
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5. Resistin and visfatin: regulators of insulin sensitivity, inflammation and immunity
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6. CD38 as a Regulator of Cellular NAD: A Novel Potential Pharmacological Target for Metabolic Conditions
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7. Visfatin/NAMPT: A Multifaceted Molecule with Diverse Roles in Physiology and Pathophysiology
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9. Unique Toll-Like Receptor 4 Activation by NAMPT/PBEF Induces NFκB Signaling and Inflammatory Lung Injury
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13. NAD+ biosynthesis, aging, and disease
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14. Life Span Extension and Neuronal Cell Protection by Drosophila Nicotinamidase
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16. Nicotinamide Mononucleotide: A Promising Molecule for Therapy of Diverse Diseases by Targeting NAD+ Metabolism
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17. Nicotinamide is an inhibitor of SIRT1 in vitro, but can be a stimulator in cells
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18. The Role of Mammalian Sirtuins in the Regulation of Metabolism, Aging, and Longevity
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19. Adipose Tissue as an Endocrine Organ: An Update on Pro-inflammatory and Anti-inflammatory Microenvironment
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20. Age-related NAD+ decline
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21. Exercise and regulation of adipokine and myokine production
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22. Visfatin, glucose metabolism and vascular disease: a review of evidence
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23. Cytokines in the Progression of Pancreatic β -Cell Dysfunction
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24. Recent Advances in NAMPT Inhibitors: A Novel Immunotherapic Strategy
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25. Visfatin Induces Inflammation and Insulin Resistance via the NF- κ B and STAT3 Signaling Pathways in Hepatocytes
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26. Molecular Characteristics of Serum Visfatin and Differential Detection by Immunoassays
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27. Torpor induction in mammals: recent discoveries fueling new ideas
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28. CD73 Protein as a Source of Extracellular Precursors for Sustained NAD+ Biosynthesis in FK866-treated Tumor Cells
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29. The role of NAD+ metabolism and its modulation of mitochondria in aging and disease
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30. Medicinal Chemistry of Nicotinamide Phosphoribosyltransferase (NAMPT) Inhibitors
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31. Nicotinamide phosphoribosyltransferase (NAMPT/PBEF/visfatin) is constitutively released from human hepatocytes
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33. Biological and Potential Therapeutic Roles of Sirtuin Deacetylases
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34. Possible Adverse Effects of High-Dose Nicotinamide: Mechanisms and Safety Assessment
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35. Circadian Rhythms in Diet-Induced Obesity
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36. Hepatic FoxOs Regulate Lipid Metabolism via Modulation of Expression of the Nicotinamide Phosphoribosyltransferase Gene
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37. Mechanisms Linking Excess Adiposity and Carcinogenesis Promotion
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38. Vascular Effects of Novel Adipocytokines: Focus on Vascular Contractility and Inflammatory Responses
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39. Nicotinamide mononucleotide protects against pro-inflammatory cytokine-mediated impairment of mouse islet function
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40. The Nicotinamide Phosphoribosyltransferase: A Molecular Link between Metabolism, Inflammation, and Cancer
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41. Off the Clock: From Circadian Disruption to Metabolic Disease
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42. Sirtuin and metabolic kidney disease
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44. NAD metabolism: Role in senescence regulation and aging
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45. Vaspin and visfatin/Nampt are interesting interrelated adipokines playing a role in the pathogenesis of type 2 diabetes mellitus
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48. An NAD + -dependent transcriptional program governs self-renewal and radiation resistance in glioblastoma
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49. Visfatin/eNampt induces endothelial dysfunction in vivo: a role for Toll-Like Receptor 4 and NLRP3 inflammasome
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50. Nicotinamide improves glucose metabolism and affects the hepatic NAD-sirtuin pathway in a rodent model of obesity and type 2 diabetes
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54. Mechanisms of enhanced insulin secretion and sensitivity with n-3 unsaturated fatty acids
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70. Visfatin: A Possible Role in Cardiovasculo-Metabolic Disorders
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71. Nicotinamide Phosphoribosyltransferase/Visfatin Does Not Catalyze Nicotinamide Mononucleotide Formation in Blood Plasma
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75. Silybin inhibits NLRP3 inflammasome assembly through the NAD + /SIRT2 pathway in mice with nonalcoholic fatty liver disease
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86. Adipose tissue NAD+-homeostasis, sirtuins and poly(ADP-ribose) polymerases -important players in mitochondrial metabolism and metabolic health
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