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Cited by 610

The DOI matches its Crossref record; 8 of the 8 references it cites were found in a registry or an index.

Autophagy Inhibition Compromises Degradation of Ubiquitin-Proteasome Pathway Substrates

Korolchuk, Viktor I., Mansilla, Alicia, Menzies, Fiona M., Rubinsztein, David C.·2009·Molecular Cell
DOI 10.1016/j.molcel.2009.01.021PMC PMC2669153
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  • Levels as well as chromatin recruitment of USP14 are upregulated in autophagy-deficient cells upon DNA damage, and knockdown of SQSTM1 in autophagy-deficient cells decreases USP14 levels.

    “In addition, levels as well as chromatin recruitment of USP14 are upregulated in autophagy-deficient cells upon DNA damage, and knockdown of SQSTM1 in autophagy-deficient cells decreases USP14 levels [602, 603].”

    From Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) 1 (2021) · cites this paper as [602]

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Research works printed but not linked: 27

  1. Aberle, 1997, β-catenin is a target for the ubiquitin-proteasome pathway, EMBO J., 16, 3797, 10.1093/emboj/16.13.3797 · DOI
  2. Bjorkoy, 2005, p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death, J. Cell Biol., 171, 603, 10.1083/jcb.200507002 · DOI · Paper page
  3. Boyault, 2007, HDAC6 controls major cell response pathways to cytotoxic accumulation of protein aggregates, Genes Dev., 21, 2172, 10.1101/gad.436407 · DOI
  4. Dantuma, 2000, Short-lived green fluorescent proteins for quantifying ubiquitin/proteasome-dependent proteolysis in living cells, Nat. Biotechnol., 18, 538, 10.1038/75406 · DOI · Paper page
  5. Funakoshi, 2002, Budding yeast Dsk2p is a polyubiquitin-binding protein that can interact with the proteasome, Proc. Natl. Acad. Sci. USA, 99, 745, 10.1073/pnas.012585199 · DOI
  6. Gal, 2007, p62 accumulates and enhances aggregate formation in model systems of familial amyotrophic lateral sclerosis, J. Biol. Chem., 282, 11068, 10.1074/jbc.M608787200 · DOI
  7. Iwata, 2005, HDAC6 and microtubules are required for autophagic degradation of aggregated huntingtin, J. Biol. Chem., 280, 40282, 10.1074/jbc.M508786200 · DOI · Paper page
  8. Izumi, 2001, Cell cycle-dependent proteolysis and phosphorylation of human Mcm10, J. Biol. Chem., 276, 48526, 10.1074/jbc.M107190200 · DOI
  9. Kleijnen, 2000, The hPLIC proteins may provide a link between the ubiquitination machinery and the proteasome, Mol. Cell, 6, 409, 10.1016/S1097-2765(00)00040-X · DOI
  10. Kobayashi, 2007, Involvement of valosin-containing protein (VCP)/p97 in the formation and clearance of abnormal protein aggregates, Genes Cells, 12, 889, 10.1111/j.1365-2443.2007.01099.x · DOI
  11. Komatsu, 2006, Loss of autophagy in the central nervous system causes neurodegeneration in mice, Nature, 441, 880, 10.1038/nature04723 · DOI · Paper page
  12. Komatsu, 2007, Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice, Cell, 131, 1149, 10.1016/j.cell.2007.10.035 · DOI · Paper page
  13. Mizushima, 2001, Dissection of autophagosome formation using Apg5-deficient mouse embryonic stem cells, J. Cell Biol., 152, 657, 10.1083/jcb.152.4.657 · DOI · Paper page
  14. Nagaoka, 2004, Increased expression of p62 in expanded polyglutamine-expressing cells and its association with polyglutamine inclusions, J. Neurochem., 91, 57, 10.1111/j.1471-4159.2004.02692.x · DOI
  15. Narain, 1999, A molecular investigation of true dominance in Huntington's disease, J. Med. Genet., 36, 739, 10.1136/jmg.36.10.739 · DOI
  16. Nezis, 2008, Ref(2)P, the Drosophila melanogaster homologue of mammalian p62, is required for the formation of protein aggregates in adult brain, J. Cell Biol., 180, 1065, 10.1083/jcb.200711108 · DOI · Paper page
  17. Raasi, 2003, Rad23 ubiquitin-associated domains (UBA) inhibit 26 S proteasome-catalyzed proteolysis by sequestering lysine 48-linked polyubiquitin chains, J. Biol. Chem., 278, 8951, 10.1074/jbc.M212841200 · DOI
  18. Ravikumar, 2002, Aggregate-prone proteins with polyglutamine and polyalanine expansions are degraded by autophagy, Hum. Mol. Genet., 11, 1107, 10.1093/hmg/11.9.1107 · DOI
  19. Richly, 2005, A series of ubiquitin binding factors connects CDC48/p97 to substrate multiubiquitylation and proteasomal targeting, Cell, 120, 73, 10.1016/j.cell.2004.11.013 · DOI
  20. Sarkar, 2007, Trehalose, a novel mTOR-independent autophagy enhancer, accelerates the clearance of mutant huntingtin and [α]-synuclein, J. Biol. Chem., 282, 5641, 10.1074/jbc.M609532200 · DOI · Paper page
  21. Seibenhener, 2007, Sequestosome 1/p62–more than just a scaffold, FEBS Lett., 581, 175, 10.1016/j.febslet.2006.12.027 · DOI
  22. Tartaglia, 2007, Life on the edge: a link between gene expression levels and aggregation rates of human proteins, Trends Biochem. Sci., 32, 204, 10.1016/j.tibs.2007.03.005 · DOI
  23. Weihl, 2006, Inclusion body myopathy-associated mutations in p97/VCP impair endoplasmic reticulum-associated degradation, Hum. Mol. Genet., 15, 189, 10.1093/hmg/ddi426 · DOI
  24. Wojcik, 2006, Valosin-containing protein (p97) is a regulator of endoplasmic reticulum stress and of the degradation of N-end rule and ubiquitin-fusion degradation pathway substrates in mammalian cells, Mol. Biol. Cell, 17, 4606, 10.1091/mbc.E06-05-0432 · DOI
  25. Wooten, 2006, Signaling, polyubiquitination, trafficking, and inclusions: sequestosome 1/p62's role in neurodegenerative disease, J. Biomed. Biotechnol., 2006, 62079, 10.1155/JBB/2006/62079 · DOI
  26. Xie, 2007, Autophagosome formation: core machinery and adaptations, Nat. Cell Biol., 9, 1102, 10.1038/ncb1007-1102 · DOI
  27. Ye, 2006, Diverse functions with a common regulator: ubiquitin takes command of an AAA ATPase, J. Struct. Biol., 156, 29, 10.1016/j.jsb.2006.01.005 · DOI

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Metadata

Authors
Korolchuk, V. I., Mansilla, A., Menzies, F. M., & Rubinsztein, D. C.
Venue
Molecular Cell
Publisher
Elsevier BV
Published
2009-02
Volume
33
Issue
4
Pages
517-527
ISSN
1097-2765
DOI
10.1016/j.molcel.2009.01.021
PMC
PMC2669153
Cited by
610
References
8
Language
en
Licence

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