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Molecular responses of the Ts65Dn and Ts1Cje mouse models of Down syndrome to MK-801

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Rapamycin restores BDNF-LTP and the persistence of long-term memory in a model of Down's syndrome

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New technique using galactose‐specific lectin for isolation of fetal cells from maternal blood

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Widespread impairment of cell proliferation in the neonate Ts65Dn mouse, a model for Down syndrome

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Distinct Defects in Spine Formation or Pruning in Two Gene Duplication Mouse Models of Autism

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Brain ventriculomegaly in Down syndrome mice is caused by Pcp4 dose-dependent cilia dysfunction.

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The in vivo Down syndrome genomic library in mouse

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Aging and intellectual disability: Insights from mouse models of down syndrome

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DYRK1A regulates Hap1–Dcaf7/WDR68 binding with implication for delayed growth in Down syndrome

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Alterations of in vivo CA1 network activity in Dp(16)1Yey Down syndrome model mice

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Interferon hyperactivity impairs cardiogenesis in Down syndrome via downregulation of canonical Wnt signaling

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Dysregulated systemic metabolism in a Down syndrome mouse model

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Mouse-based genetic modeling and analysis of Down syndrome

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Human and mouse model cognitive phenotypes in Down syndrome

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Direct modulation of G protein-gated inwardly rectifying potassium (GIRK) channels

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The Critical Region in Trisomy 21

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Autonomous trisomic rescue of Down syndrome cells

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Neprilysin Is Suppressed by Dual-Specificity Tyrosine-Phosphorylation Regulated Kinase 1A (DYRK1A) in Down-Syndrome-Derived Fibroblasts

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Cholinergic neurodegeneration in Alzheimer disease mouse models

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Perfect Conserved Linkage Across the Entire Mouse Chromosome 10 Region Homologous to Human Chromosome 21Preprint

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Ts1Cje mouse model for Down syndrome research

Ling, K. H. (2015) · Universiti Putra Malaysia Institutional Repository (Universiti Putra Malaysia)

A mouse model for Down syndrome exhibits learning and behaviour deficits [see comments]

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Ts1Cje Down syndrome model mice exhibit environmental stimuli-triggered locomotor hyperactivity and sociability concurrent with increased flux through central dopamine and serotonin metabolism

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