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The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Disabled-2 (Dab2) Is Required for Transforming Growth Factor β-induced Epithelial to Mesenchymal Transition (EMT)

Prunier, Céline, Howe, Philip H.·2005·Journal of Biological Chemistry
DOI 10.1074/jbc.m500974200
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Crossref · 10.1074/jbc.m500974200

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  • Several mechanisms have been suggested for Dab2 involvement in the TGF-beta pathway, but their actual operation in intact animals and physiological settings has yet to be demonstrated.

    “Thus, several mechanisms have been suggested for the involvement of Dab2 in TGF-beta pathway, such as receptor recycling, adapting function for Smad activation, or non-canonical TGF-beta signaling through the Ras-MAPK pathway, although the actual operation of these mechanisms in intact animals and physiological settings have yet to be demonstrated.”

    From Endocytosis and Physiology: Insights from Disabled-2 Deficient Mice (2016) · cites this paper as Prunier and Howe, 2005

    The introductory passage describes Dab2 as an adaptor linking TGF-β receptors to Smads, and the Discussion considers a possible Smad-independent MAPK pathway. The Discussion also says that pathway remains under investigation, while the reported TGF-β experiments use cultured cells. Our copy does not specifically propose TGF-β receptor recycling or a Ras–MAPK mechanism, or explicitly state that all the listed mechanisms await demonstration in intact animals. (LLM, conf=0.84)

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Research works printed but not linked: 50

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Metadata

Authors
Prunier, C., & Howe, P. H.
Venue
Journal of Biological Chemistry
Publisher
Elsevier BV
Published
2005-04
Volume
280
Issue
17
Pages
17540-17548
ISSN
0021-9258
DOI
10.1074/jbc.m500974200
Cited by
120
References
2
Language
en
Licence

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