Cited by 234

The DOI matches its Crossref record; 14 of the 14 references it cites were found in a registry or an index.

The presence of sodium dodecyl sulphate-stable Aβ dimers is strongly associated with Alzheimer-type dementia

Mc Donald, Jessica M., Savva, George M., Brayne, Carol, Welzel, Alfred T., Forster, Gill, Shankar, Ganesh M., Selkoe, Dennis J., Ince, Paul G., Walsh, Dominic M.·2010·Brain

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Crossref · 10.1093/brain/awq065

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  • Concentrations of soluble Aβ species, not the presence of plaques, seem to strongly correlate with cognitive deficits in mouse models and AD pathology in patients.

    “As recent works suggest, the concentrations of soluble Aβ species, not the presence of plaques, seem to strongly correlate with the cognitive deficits in mouse models (54) and AD pathology in patients (55,56).”

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Research works printed but not linked: 25

  1. Ball, 1997, Consensus recommendations for the postmortem diagnosis of Alzheimers-disease, Neurobiol Aging, 18, S12
  2. Brayne, 2006, Medical Research Council Cognitive Function and Ageing Study (CFAS). Cohort profile: the Medical Research Council Cognitive Function and Ageing Study (CFAS), Int J Epidemiol, 35, 1140, 10.1093/ije/dyl199 · DOI
  3. Copeland, 1990, Dementia and depression in elderly persons: AGECAT compared with DSM III and pervasive illness, Int J Geriatr Psychiatry, 5, 47, 10.1002/gps.930050108 · DOI
  4. Dickson, 1991, Identification of normal and pathological aging in prospectively studied nondemeneted elderly individuals, Neurobiol Aging, 13, 179, 10.1016/0197-4580(92)90027-U · DOI · Paper page
  5. Funato, 1999, Presence of sodium dodecyl sulfate-stable amyloid β-protein in the hippocampus CA1 not exhibiting neurofibrillary tangle formation, Am J Pathol, 155, 23, 10.1016/S0002-9440(10)65094-8 · DOI
  6. Funato, 1998, Quantitation of amyloid beta-protein (Aβ) in the cortex during aging and in Alzheimer’s-disease, Am J Pathol, 152, 1633
  7. Huppert, 1995, CAMCOG-a concise neuropsychological test to assist dementia diagnosis: sociodemographic determinants in an elderly population sample, Br J Clin Psychol, 34, 529, 10.1111/j.2044-8260.1995.tb01487.x · DOI
  8. Ida, 1996, Analysis of heterogeneous A4 peptides in human cerebrospinal fluid and blood by a newly developed sensitive Western blot assay, J Biol Chem, 271, 22908, 10.1074/jbc.271.37.22908 · DOI
  9. Johnson-Wood, 1997, Amyloid precursor protein processing and Aß42 deposition in a transgenic mouse model of Alzheimer disease, Proc Natl Acad Sci, 94, 1550, 10.1073/pnas.94.4.1550 · DOI
  10. Klein, 2001, Targeting small Abeta oligomers: the solution to an Alzheimer's disease conundrum?, Trends Neurosci, 24, 219, 10.1016/S0166-2236(00)01749-5 · DOI · Paper page
  11. Kuo, 1996, Water-soluble Aβ (N-40, N-42) oligomers in normal and Alzheimer disease brains, J Biol Chem, 271, 4077, 10.1074/jbc.271.8.4077 · DOI · Paper page
  12. Lue, 1999, Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease, Am J Pathol, 155, 853, 10.1016/S0002-9440(10)65184-X · DOI · Paper page
  13. McLean, 1999, Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease, Ann Neurol, 46, 860, 10.1002/1531-8249(199912)46:6<860::AID-ANA8>3.0.CO;2-M · DOI
  14. Mirra, 1991, The Consortium to Establish a Registry for Alzheimer's disease (CERAD), Part II. Standardization of the neuropathologic assessment of Alzheimer's disease. Neurology, 41, 479
  15. Morishima-Kawashima, 1998, The presence of amyloid ß-protein in the detergent-insoluble membrane compartment of human neuroblastoma cells, Biochemistry, 37, 15247, 10.1021/bi981843u · DOI
  16. Piert, 1996, Diminished glucose transport and phosphorylation in Alzheimer's disease determined by dynamic FDG-PET, J Nucl Med, 37, 201
  17. Roher, 1993, β-amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer's disease, Proc Natl Acad Sci, 90, 10836, 10.1073/pnas.90.22.10836 · DOI · Paper page
  18. Schagger, 1987, Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa, Anal Biochem, 166, 368, 10.1016/0003-2697(87)90587-2 · DOI
  19. Selkoe, 1991, The molecular pathology of Alzheimer's disease, Neuron, 6, 487, 10.1016/0896-6273(91)90052-2 · DOI · Paper page
  20. Steinerman, 2008, Distinct pools of beta-amyloid in alzheimer disease-affected brain, Arch Neurol, 65, 906, 10.1001/archneur.65.7.906 · DOI · Paper page
  21. Stenh, 2005, Amyloid-beta oligomers are inefficiently measured by enzyme-linked immunosorbent assay, Ann Neurol, 58, 147, 10.1002/ana.20524 · DOI · Paper page
  22. Tamaoka, 1994, Biochemical evidence for the long-tail form (Abeta 1-42/43) of amyloid beta protein as a seed molecule in cerebral deposits of Alzheimer's disease, Biochem Biophys Res Commun, 205, 834, 10.1006/bbrc.1994.2740 · DOI
  23. Terry, 1991, Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment, Ann Neurol, 30, 572, 10.1002/ana.410300410 · DOI · Paper page
  24. Walsh, 2003, The many faces of Aß: Structures and activity, Curr Med Chem Immununol EndocrMetab Agent, 3, 277, 10.2174/1568013033483311 · DOI
  25. Walsh, 2000, Detection of intracellular oligomers of amyloid ß-protein in cells derived from human brain, Biochemistry, 39, 10831, 10.1021/bi001048s · DOI · Paper page

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Metadata

Authors
Mc Donald, J. M., Savva, G. M., Brayne, C., Welzel, A. T., Forster, G., Shankar, G. M., Selkoe, D. J., Ince, P. G., & Walsh, D. M.
Venue
Brain
Published
2010-04-19
Volume
133
Issue
5
Pages
1328-1341
ISSN
  • 1460-2156
  • 0006-8950
Cited by
234
References
14
Language
en

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