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Cited by 252

The DOI matches its Crossref record.

Apolipoprotein E Affects the Rate of Alzheimer Disease Expression: β-Amyloid Burden Is a Secondary Consequence Dependent on APOE Genotype and Duration of Disease

Roses, Allen D.·1994·Journal of Neuropathology & Experimental Neurology
DOI 10.1097/00005072-199409000-00002
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Abstract

Allen D. Roses, M.D.; Apolipoprotein E Affects the Rate of Alzheimer Disease Expression: β-Amyloid Burden Is a Secondary Consequence Dependent on APOE Genotype

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Crossref · 10.1097/00005072-199409000-00002

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  • Each ApoE4 allele reduces the age of Alzheimer's disease onset by approximately 3-5 years compared to ApoE3 homozygotes.

    “Each ApoE4 allele reduces the age of Alzheimer’s disease (AD) onset by approximately 3–5 years compared to ApoE3 homozygotes, which comprise ~80% of the human population (Roses, 1994; Sando et al., 2008).”

    From NHE6 depletion corrects ApoE4-mediated synaptic impairments and reduces amyloid plaque load (2021) · cites this paper as Roses, 1994

  • ApoE3 homozygotes comprise approximately 80% of the human population.

    “Each ApoE4 allele reduces the age of Alzheimer’s disease (AD) onset by approximately 3–5 years compared to ApoE3 homozygotes, which comprise ~80% of the human population (Roses, 1994; Sando et al., 2008).”

    From NHE6 depletion corrects ApoE4-mediated synaptic impairments and reduces amyloid plaque load (2021) · cites this paper as Roses, 1994

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Metadata

Authors
Roses, A. D.
Venue
Journal of Neuropathology & Experimental Neurology
Publisher
Oxford University Press (OUP)
Published
1994-09
Volume
53
Issue
5
Pages
429-437
ISSN
  • 0022-3069
  • 1554-6578
DOI
10.1097/00005072-199409000-00002
Cited by
252
References
0
Language
en

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