The DOI matches its Crossref record; 15 of the 15 references it cites were found in a registry or an index.
Chromatin Remodeling in Cardiovascular Development and Physiology
Open accessAbstract
Chromatin regulation provides an important means for controlling cardiac gene expression under different physiological and pathological conditions. Processes that direct the development of normal embryonic hearts and pathology of stressed adult hearts may share general mechanisms that govern cardiac gene expression by chromatin-regulating factors. These common mechanisms may provide a framework for us to investigate the interactions among diverse chromatin remodelers/modifiers and various transcription factors in the fine regulation of gene expression, essential for all aspects of cardiovascular biology. Aberrant cardiac gene expression, triggered by a variety of pathological insults, can cause heart diseases in both animals and humans. The severity of cardiomyopathy and heart failure correlates strongly with abnormal cardiac gene expression. Therefore, controlling cardiac gene expression presents a promising approach to the treatment of human cardiomyopathy. This review focuses on the roles of ATP-dependent chromatin-remodeling factors and chromatin-modifying enzymes in the control of gene expression during cardiovascular development and disease.
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1 retracted
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Mediation of Poly(ADP-Ribose) Polymerase-1-Dependent Cell Death by Apoptosis-Inducing Factor
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Mammalian sirtuins—emerging roles in physiology, aging, and calorie restriction
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JmjC-domain-containing proteins and histone demethylation
Klose, R. J., Kallin, E. M., & Zhang, Y. (2006) · Nature Reviews Genetics · doi:10.1038/nrg1945 · Cited by 1,290
Mammalian Sir2 homolog SIRT3 regulates global mitochondrial lysine acetylation
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Chromatin remodelling during development
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Increased Nuclear NAD Biosynthesis and SIRT1 Activation Prevent Axonal Degeneration
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Vascular Smooth Muscle Cells and Arterial Stiffening: Relevance in Development, Aging, and Disease
Lacolley, P., Regnault, V., Segers, P., & Laurent, S. (2017) · Physiological Reviews · doi:10.1152/physrev.00003.2017 · Cited by 734
The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system
Kietzmann, T., Petry, A., Shvetsova, A., Gerhold, J. M., & Görlach, A. (2017) · British Journal of Pharmacology · doi:10.1111/bph.13792 · Cited by 253
Genetic Networks Governing Heart Development
Waardenberg, A. J., Ramialison, M., Bouveret, R., & Harvey, R. P. (2014) · Cold Spring Harbor Perspectives in Medicine · doi:10.1101/cshperspect.a013839 · Cited by 59
Blum, K., Bowirrat, A., Baron, D., Elman, I., Makale, M. T., Cadet, J. L., Thanos, P. K., Hanna, C., Ahmed, R., Gondre-Lewis, M. C., et al. (2024) · Gene & Protein in Disease · doi:10.36922/gpd.1966 · Cited by 14
(2024) · Journal of Addiction Psychiatry · doi:10.17756/jap.2024-045 · Cited by 3
Metadata
- Authors
- Han, P., Hang, C. T., Yang, J., & Chang, C.-P.
- Venue
- Circulation Research
- Published
- 2011-02-04
- Volume
- 108
- Issue
- 3
- Pages
- 378-396
- ISSN
- 0009-7330 (ISSN-L)
- 1524-4571
- OpenAlex
- W2077489944
- Cited by
- 165
- References
- 15
- FWCI
- 6.70
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Han, P., Hang, C. T., Yang, J., & Chang, C.-P. (2011). Chromatin Remodeling in Cardiovascular Development and Physiology. Circulation Research. https://doi.org/10.1161/circresaha.110.224287
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
