Mitochondrial dysfunction - the beginning of the end in Alzheimer's disease? Separate and synergistic modes of tau and amyloid-β toxicity

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Alzheimer's Disease and Parkinson's Disease

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ABAD Directly Links Aß to Mitochondrial Toxicity in Alzheimer's Disease

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S-Nitrosylation of Drp1 Mediates β-Amyloid-Related Mitochondrial Fission and Neuronal Injury

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Accumulation of amyloid precursor protein in the mitochondrial import channels of human Alzheimer's disease brain is associated with mitochondrial dysfunction

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Mitochondrial Aβ: a potential focal point for neuronal metabolic dysfunction in Alzheimer's disease

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Mitochondrial Oxidative Stress Causes Hyperphosphorylation of Tau

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Dynamin-Like Protein 1 Reduction Underlies Mitochondrial Morphology and Distribution Abnormalities in Fibroblasts from Sporadic Alzheimer's Disease Patients

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Copper-Dependent Inhibition of Human Cytochrome<i>c</i>Oxidase by a Dimeric Conformer of Amyloid-β_1-42

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Altered neuronal gene expression in brain regions differentially affected by Alzheimer's disease: a reference data set

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Amyloid β-induced Changes in Nitric Oxide Production and Mitochondrial Activity Lead to Apoptosis

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Cytochrome c oxidase is decreased in Alzheimer’s disease platelets

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Induction of Tau Pathology by Intracerebral Infusion of Amyloid-β-Containing Brain Extract and by Amyloid-β Deposition in APP × Tau Transgenic Mice

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Increasing Cu bioavailability inhibits Aβ oligomers and tau phosphorylation

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Mitochondrial Dysfunction: Common Final Pathway in Brain Aging and Alzheimer’s Disease—Therapeutic Aspects

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Mitochondrial permeability transition pore in Alzheimer's disease: Cyclophilin D and amyloid beta

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Phosphorylation of tau antagonizes apoptosis by stabilizing β-catenin, a mechanism involved in Alzheimer's neurodegeneration

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Annonacin, a Natural Mitochondrial Complex I Inhibitor, Causes Tau Pathology in Cultured Neurons

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Amyloid-beta Leads to Impaired Cellular Respiration, Energy Production and Mitochondrial Electron Chain Complex Activities in Human Neuroblastoma Cells

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PS2APP Transgenic Mice, Coexpressing hPS2mut and hAPPswe, Show Age-Related Cognitive Deficits Associated with Discrete Brain Amyloid Deposition and Inflammation

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Mitochondrial Dysfunction in Aging and Alzheimer's Disease: Strategies to Protect Neurons

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Neurotoxic Mechanisms Caused by the Alzheimer's Disease-linked Swedish Amyloid Precursor Protein Mutation

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Is Tau Aggregation Toxic or Protective: A Sensible Question in the Absence of Sensitive Methods?

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Recent advances in Alzheimer's disease: Mechanisms, clinical trials and new drug development strategies.

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Human mitochondrial transcriptional factor A breaks the mitochondria-mediated vicious cycle in Alzheimer’s disease

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