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Tau-mediated dysregulation of RNA: Evidence for a common molecular mechanism of toxicity in frontotemporal dementia and other tauopathies

Koren, Shon A., Galvis-Escobar, Sara, Abisambra, Jose F.·2020·Neurobiology of Disease
DOI 10.1016/j.nbd.2020.104939PMC PMC7397812
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  • The potential reason of reduced neuronal activity in neurons accumulated with pathological tau is the repression of translation by abnormal tau accumulation.

    “The potential reason of reduced neuronal activity in neurons accumulated with pathological tau is the repression of translation91 by abnormal tau accumulation.”

    From GRAMD1B is a regulator of lipid homeostasis, autophagic flux and phosphorylated tau (2025) · cites this paper as [91]

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Metadata

Authors
Koren, S. A., Galvis-Escobar, S., & Abisambra, J. F.
Venue
Neurobiology of Disease
Publisher
Elsevier BV
Published
2020-07
Volume
141
Pages
104939
ISSN
0969-9961
DOI
10.1016/j.nbd.2020.104939
PMC
PMC7397812
Cited by
49
References
13
Language
en
Licence
Funders
National Institutes of Health

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