The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
Regional Distribution of Amyloid-Bri Deposition and Its Association with Neurofibrillary Degeneration in Familial British Dementia
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
The cited source says what the claim attributes to it.
In the citing paper
“The extensive Aβ42 deposition in the cerebellum and severe CAA in the BRI-Aβ42 mice mimics the pattern of amyloid deposition seen in FBD (Ghiso et al., 2000; Holton et al., 2001; Vidal et al., 1999).”
From Aβ42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice (2005) · cites this paper as Holton et al., 2001
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Neuropathological stageing of Alzheimer-related changes.
Braak, H., & Braak, E. (1991) · Acta Neuropathologica · doi:10.1007/bf00308809 · Cited by 16,123
Production of the Alzheimer Amyloid β Protein by Normal Proteolytic Processing
Shoji, M., Golde, T. E., Ghiso, J., Cheung, T. T., Estus, S., Shaffer, L. M., Cai, X.-D., McKay, D. M., Tintner, R., Frangione, B., et al. (1992) · Science · doi:10.1126/science.1439760 · Cited by 1,447
Self-citationVidal, R., Révész, T., Rostagno, A., Kim, E., Holton, J. L., Bek, T., Bojsen-Møller, M., Braendgaard, H., Plant, G., Ghiso, J., et al. (2000) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.080076097 · Cited by 300
Self-citationBraak, H., & Braak, E. (1985) · Acta Neuropathologica · doi:10.1007/bf00690836 · Cited by 287
Wisniewski, H. M., Sadowski, M., Jakubowska-Sadowswka, K., Tarnawski, M., & Wegiel, J. (1998) · Journal of Neuropathology and Experimental Neurology · doi:10.1097/00005072-199807000-00004 · Cited by 71
Research works printed but not linked: 39
- Worster-Drought, 1933, Familial presenile dementia with spastic paralysis, J Neurol Psychopathol, 14, 27, 10.1136/jnnp.s1-14.53.27 · DOI
- Plant, 1990, Familial cerebral amyloid angiopathy with nonneuritic amyloid plaque formation, Brain, 113, 721, 10.1093/brain/113.3.721 · DOI
- Mead, 2000, Familial British dementia with amyloid angiopathy: early clinical, neuropsychological and imaging findings, Brain, 123, 975, 10.1093/brain/123.5.975 · DOI
- Worster-Drought, 1940, A form of familial presenile dementia with spastic paralysis (including the pathological examination of a case), Brain, 63, 237, 10.1093/brain/63.3.237 · DOI
- Revesz, 1999, Cytoskeletal pathology in familial cerebral amyloid angiopathy (British type) with non-neuritic amyloid plaque formation, Acta Neuropathol (Berl), 97, 170, 10.1007/s004010050970 · DOI
- Vidal, 1999, A stop-codon mutation in the BRI gene associated with familial British dementia, Nature, 399, 776, 10.1038/21637 · DOI · Paper page
- Mirra, 1991, The consortium to establish a registry for Alzheimer's disease (CERAD). 2. Standardization of the neuropathologic assessment of Alzheimer's disease, Neurology, 41, 479, 10.1212/WNL.41.4.479 · DOI · Paper page
- Brion, 1993, Neurofilament monoclonal antibodies RT97 and 8D8 recognize different modified epitopes in paired helical filament-tau in Alzheimer's disease, J Neurochem, 60, 1372, 10.1111/j.1471-4159.1993.tb03298.x · DOI
- Hanger, 1991, Tau in Alzheimer's disease and Down's syndrome is insoluble and abnormally phosphorylated, Biochem J, 275, 99, 10.1042/bj2750099 · DOI
- Laemmli, 1970, Cleavage of structural proteins during the assembly of the head of bacteriophage T4, Nature, 227, 680, 10.1038/227680a0 · DOI · Paper page
- Yamaguchi, 1987, Alzheimer's neurofibrillary tangles are penetrated by astroglial processes and appear eosinophilic in their final stages, Acta Neuropathol (Berl), 72, 214, 10.1007/BF00691092 · DOI
- Bondareff, 1994, Immunohistochemical staging of neurofibrillary degeneration in Alzheimer's disease, J Neuropathol Exp Neurol, 53, 158, 10.1097/00005072-199403000-00007 · DOI
- Bjorklund, 1985, Image analysis of GFA-positive astrocytes from adolescence to senescence, Exp Brain Res, 58, 163, 10.1007/BF00238964 · DOI
- Verga, 1989, Alzheimer patients and Down patients: cerebral preamyloid deposits differ ultrastructurally and histochemically from the amyloid of senile plaques, Neurosci Lett, 105, 294, 10.1016/0304-3940(89)90636-8 · DOI
- Giaccone, 1992, Prion protein preamyloid and amyloid deposits in Gerstmann-Straussler-Scheinker disease, Indiana kindred, Proc Natl Acad Sci USA, 89, 9349, 10.1073/pnas.89.19.9349 · DOI
- Griffiths, 1982, Congophilic angiopathy of the brain: a clinical and pathological report on two siblings, J Neurol Neurosurg Psychiatry, 45, 396, 10.1136/jnnp.45.5.396 · DOI
- Love, 1982, Familial cerebellar ataxia with cerebrovascular amyloid, J Neurol Neurosurg Psychiatry, 45, 271, 10.1136/jnnp.45.3.271 · DOI
- Kalus, 1989, The presubicular region in Alzheimer's disease: topography of amyloid deposits and neurofibrillary changes, Brain Res, 494, 198, 10.1016/0006-8993(89)90164-9 · DOI
- Ogomori, 1989, Beta-protein amyloid is widely distributed in the central nervous system of patients with Alzheimer's disease, Am J Pathol, 134, 243
- Goedert, 1993, The abnormal phosphorylation of tau-protein at ser-202 in Alzheimer disease recapitulates phosphorylation during development, Proc Natl Acad Sci USA, 90, 5066, 10.1073/pnas.90.11.5066 · DOI · Paper page
- Su, 1994, Early phosphorylation of tau in Alzheimer's disease occurs at Ser-202 and is preferentially located within neurites, Neuroreport, 5, 2358, 10.1097/00001756-199411000-00037 · DOI
- Schmidt, 1994, An extensive network of PHF-τ-rich dystrophic neurites permeates neocortex and nearly all neuritic and diffuse amyloid plaques in Alzheimer disease, FEBS Lett, 344, 69, 10.1016/0014-5793(94)00259-2 · DOI
- Yamaguchi, 1991, Secondary deposition of beta amyloid within extracellular neurofibrillary tangles in Alzheimer-type dementia, Am J Pathol, 138, 699
- ZhengFischhofer, 1998, Sequential phosphorylation of Tau by glycogen synthase kinase-3 beta and protein kinase A at Thr212 and Ser214 generates the Alzheimer-specific epitope of antibody AT100 and requires a paired-helical-filament-like conformation, Eur J Biochem, 252, 542, 10.1046/j.1432-1327.1998.2520542.x · DOI
- el Hachimi, 1991, Relationship between non-fibrillary amyloid precursors and cell processes in the cortical neuropil of Alzheimer patients, Neurosci Lett, 129, 119, 10.1016/0304-3940(91)90734-B · DOI
- Pike, 1995, Early association of reactive astrocytes with senile plaques in Alzheimer's disease, Exp Neurol, 132, 172, 10.1016/0014-4886(95)90022-5 · DOI · Paper page
- Selkoe, 1999, Translating cell biology into therapeutic advances in Alzheimer's disease, Nature, 399, A23, 10.1038/399a023 · DOI · Paper page
- Weller, 1998, Cerebral amyloid angiopathy—amyloid beta accumulates in putative interstitial fluid drainage pathways in Alzheimer's disease, Am J Pathol, 153, 725, 10.1016/S0002-9440(10)65616-7 · DOI · Paper page
- Kim, 1999, Furin mediates enhanced production of fibrillogenic ABri peptides in familial British dementia, Nat Neurosci, 2, 984, 10.1038/14783 · DOI · Paper page
- Day, 1993, Region specific expression of furin mRNA in the rat brain, Neurosci Lett, 149, 27, 10.1016/0304-3940(93)90339-M · DOI
- Probst, 1991, Deposition of beta/A4 protein along neuronal plasma membranes in diffuse senile plaques, Acta Neuropathol (Berl), 83, 21, 10.1007/BF00294426 · DOI
- Mann, 1992, Deposition of amyloid beta protein in non-Alzheimer dementias: evidence for a neuronal origin of parenchymal deposits of beta protein in neurodegenerative disease, Acta Neuropathol (Berl), 83, 415, 10.1007/BF00713534 · DOI
- Calhoun, 1999, Neuronal overexpression of mutant amyloid precursor protein results in prominent deposition of cerebrovascular amyloid, Proc Natl Acad Sci USA, 96, 14088, 10.1073/pnas.96.24.14088 · DOI · Paper page
- Ghiso, 1997, Alzheimer's soluble amyloid beta is a normal component of human urine, FEBS Lett, 408, 105, 10.1016/S0014-5793(97)00400-6 · DOI
- Spillantini, 1998, Tau protein pathology in neurodegenerative diseases, Trends Neurosci, 21, 428, 10.1016/S0166-2236(98)01337-X · DOI · Paper page
- Crook, 1998, A variant of Alzheimer's disease with spastic paraparesis and unusual plaques due to deletion of exon 9 of presenilin 1, Nat Med, 4, 452, 10.1038/nm0498-452 · DOI
- Houlden, 2000, Variant Alzheimer's disease with spastic paraparesis and ‘cotton wool’ plaques is caused by PS-1 mutations which lead to exceptionally high Aβ concentrations, Ann Neurol, 68, 806, 10.1002/1531-8249(200011)48:5<806::AID-ANA18>3.0.CO;2-F · DOI
- Gray, 1985, Leukoencephalopathy in diffuse hemorrhagic cerebral amyloid angiopathy, Ann Neurol, 18, 54, 10.1002/ana.410180110 · DOI
- Vinters, 1990, Immunohistochemical study of cerebral amyloid angiopathy. III. Widespread Alzheimer A4 peptide in cerebral microvessel walls colocalizes with gamma trace in patients with leukoencephalopathy, Ann Neurol, 28, 34, 10.1002/ana.410280108 · DOI
Cited by
Sorted by citations
Pathology of Neurodegenerative Diseases
Dugger, B. N., & Dickson, D. W. (2017) · Cold Spring Harbor Perspectives in Biology · doi:10.1101/cshperspect.a028035 · Cited by 1,665
Arendt, T., Stieler, J. T., & Holzer, M. (2016) · Brain Research Bulletin · doi:10.1016/j.brainresbull.2016.08.018 · Cited by 609
Aβ42 Is Essential for Parenchymal and Vascular Amyloid Deposition in Mice
McGowan, E., Pickford, F., Kim, J., Onstead, L., Eriksen, J., Yu, C., Skipper, L., Murphy, M. P., Beard, J., Das, P., et al. (2005) · Neuron · doi:10.1016/j.neuron.2005.06.030 · Cited by 565
Invited review: Neuropathology of tauopathies: principles and practice
Kovacs, G. G. (2015) · Neuropathology and Applied Neurobiology · doi:10.1111/nan.12208 · Cited by 492
Cerebrovascular disease in ageing and Alzheimer’s disease
Love, S., & Miners, J. S. (2016) · Acta Neuropathologica · doi:10.1007/s00401-015-1522-0 · Cited by 299
Biron, K. E., Dickstein, D. L., Gopaul, R., & Jefferies, W. A. (2011) · PLoS ONE · doi:10.1371/journal.pone.0023789 · Cited by 300
Sporadic cerebral amyloid angiopathy: pathology, clinical implications, and possible pathomechanisms
Attems, J. (2005) · Acta Neuropathologica · doi:10.1007/s00401-005-1074-9 · Cited by 242
Attems, J., Jellinger, K. A., & Lintner, F. (2005) · Acta Neuropathologica · doi:10.1007/s00401-005-1064-y · Cited by 188
Tomidokoro, Y., Lashley, T., Rostagno, A., Neubert, T. A., Bojsen-Møller, M., Braendgaard, H., Plant, G., Holton, J., Frangione, B., Révész, T., et al. (2005) · Journal of Biological Chemistry · doi:10.1074/jbc.m504038200 · Cited by 68
Self-citationCastellani, R. J., Smith, M. A., Perry, G., & Friedland, R. P. (2004) · Neurobiology of Aging · doi:10.1016/j.neurobiolaging.2003.12.019 · Cited by 57
Tau as a mediator of neurotoxicity associated to cerebral amyloid angiopathy
You, Y., Perkins, A., Cisternas, P., Muñoz, B., Taylor, X., You, Y., Garringer, H. J., Oblak, A. L., Atwood, B. K., Vidal, R., et al. (2019) · Acta Neuropathologica Communications · doi:10.1186/s40478-019-0680-z · Cited by 33
Self-citationStructure and neurotoxicity of novel amyloids derived from the BRI gene
Gibson, G., El-Agnaf, O. M. A., Anwar, Z., Sidera, C., Isbister, A., & Austen, B. M. (2005) · Biochemical Society Transactions · doi:10.1042/bst0331111 · Cited by 13
Metadata
- Authors
- Holton, J. L., Ghiso, J., Lashley, T., Rostagno, A., Guerin, C. J., Gibb, G., Houlden, H., Ayling, H., Martinian, L., Anderton, B. H., Wood, N. W., Vidal, R., Plant, G., Frangione, B., & Revesz, T.
- Venue
- The American Journal of Pathology
- Publisher
- Elsevier BV
- Published
- 2001-02
- Volume
- 158
- Issue
- 2
- Pages
- 515-526
- ISSN
- 0002-9440
- Cited by
- 114
- References
- 5
- Language
- en
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review