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Cited by 308

The DOI matches its Crossref record; 4 of the 4 references it cites were found in a registry or an index.

Effect of Exercise on Protein Turnover in Man

Rennie, M. J., Edwards, R. H. T., Krywawych, S., Davies, C. T. M., Halliday, D., Waterlow, J. C., Millward, D. J.·1981·Clinical Science
DOI 10.1042/cs0610627
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Abstract

1. We have investigated the effects of moderate long-term exercise on protein turnover in fed man by measuring the extent of whole-body nitrogen production, the labelling of urinary ammonia from ingested [15N]glycine and plasma, muscle and urine free amino acid concentrations. 2. Judged both from nitrogen production, and from the extent of 13CO2 production from ingested l-[l-13C]leucine, exercise causes a substantial rise in amino acid catabolism. 3. Amino acids catabolized during exercise appear to become available through a fall in whole-body protein synthesis and a rise in whole-body protein breakdown. After exercise, protein balance becomes positive through a rise in the rate of whole-body synthesis in excess of breakdown. 4. Studies of free 3-methylhistidine in muscle, plasma and urine samples suggest that exercise decreases the fractional rate of myofibrillar protein breakdown, in contrast with the apparent rise in whole-body breakdown.

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Crossref · 10.1042/cs0610627

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Metadata

Authors
Rennie, M. J., Edwards, R. H. T., Krywawych, S., Davies, C. T. M., Halliday, D., Waterlow, J. C., & Millward, D. J.
Venue
Clinical Science
Publisher
Portland Press Ltd.
Published
1981-11-01
Volume
61
Issue
5
Pages
627-639
ISSN
  • 0143-5221 (ISSN-L)
  • 1470-8736
DOI
10.1042/cs0610627
OpenAlex
W152338347
Cited by
308
References
4
FWCI
3.73
Type
journal article
Language
en
Open access
closed

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.