In Vitro Characterization of Conditions for Amyloid-β Peptide Oligomerization and Fibrillogenesis

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Amyloid oligomers exacerbate tau pathology in a mouse model of tauopathy

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Deciphering the Molecular Basis of Memory Failure in Alzheimer's Disease

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Electrochemical Methods for the Analysis of Clinically Relevant Biomolecules

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Senolytic therapy alleviates Aβ-associated oligodendrocyte progenitor cell senescence and cognitive deficits in an Alzheimer’s disease model

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A systemic view of Alzheimer disease — insights from amyloid-β metabolism beyond the brain

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NFκB-Activated Astroglial Release of Complement C3 Compromises Neuronal Morphology and Function Associated with Alzheimer’s Disease

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Controlling protein translocation through nanopores with bio-inspired fluid walls

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Astrocyte-Microglia Cross Talk through Complement Activation Modulates Amyloid Pathology in Mouse Models of Alzheimer's Disease

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Astrocyte Senescence as a Component of Alzheimer’s Disease

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Using Graphene Oxide High Near‐Infrared Absorbance for Photothermal Treatment of Alzheimer's Disease

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Solution NMR Studies of the Aβ(1−40) and Aβ(1−42) Peptides Establish that the Met35 Oxidation State Affects the Mechanism of Amyloid Formation

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Small assemblies of unmodified amyloid $beta;-protein are the proximate neurotoxin in Alzheimer?s disease

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Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein

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ThT 101: a primer on the use of thioflavin T to investigate amyloid formation

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Gantenerumab: A Novel Human Anti-Aβ Antibody Demonstrates Sustained Cerebral Amyloid-β Binding and Elicits Cell-Mediated Removal of Human Amyloid-β

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Preparing Synthetic Aβ in Different Aggregation States

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Neuronal depletion of calcium-dependent proteins in the dentate gyrus is tightly linked to Alzheimer's disease-related cognitive deficits

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Are amyloid diseases caused by protein aggregates that mimic bacterial pore-forming toxins?

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The Exosome Secretory Pathway Transports Amyloid Precursor Protein Carboxyl-terminal Fragments from the Cell into the Brain Extracellular Space

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Heat Shock Proteins 70 and 90 Inhibit Early Stages of Amyloid β-(1–42) Aggregation in Vitro

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Matrix Metalloproteinases Expressed by Astrocytes Mediate Extracellular Amyloid-β Peptide Catabolism

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Matrix Metalloproteinase-9 Degrades Amyloid-β Fibrils in Vitro and Compact Plaques in Situ

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Design and Synthesis of Curcumin Analogues for in Vivo Fluorescence Imaging and Inhibiting Copper-Induced Cross-Linking of Amyloid Beta Species in Alzheimer’s Disease

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Role of Metal Ions in the Self-assembly of the Alzheimer’s Amyloid-β Peptide

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Insight Into the Kinetic of Amyloid β (1–42) Peptide Self‐Aggregation: Elucidation of Inhibitors’ Mechanism of Action

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Axonally Synthesized ATF4 Transmits a Neurodegenerative Signal across Brain Regions

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Trehalose differentially inhibits aggregation and neurotoxicity of beta-amyloid 40 and 42

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Altered Gut Microbiome Composition and Tryptic Activity of the 5xFAD Alzheimer’s Mouse Model

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Electrochemical Biosensors for Whole Blood Analysis: Recent Progress, Challenges, and Future Perspectives

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Monoclonal antibodies that target pathological assemblies of Aβ

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Toxic fibrillar oligomers of amyloid-β have cross-β structure

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Presynaptic dystrophic neurites surrounding amyloid plaques are sites of microtubule disruption, BACE1 elevation, and increased Aβ generation in Alzheimer’s disease

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Interaction between Human Prion Protein and Amyloid-β (Aβ) Oligomers

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Amyloid-β Aggregates Cause Alterations of Astrocytic Metabolic Phenotype: Impact on Neuronal Viability

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Soluble β-Amyloid1-40Induces NMDA-Dependent Degradation of Postsynaptic Density-95 at Glutamatergic Synapses

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Amyloid β-protein oligomers and Alzheimer’s disease

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TLR4 mutation reduces microglial activation, increases Aβ deposits and exacerbates cognitive deficits in a mouse model of Alzheimer’s disease

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Activity-Dependent Tau Protein Translocation to Excitatory Synapse Is Disrupted by Exposure to Amyloid-Beta Oligomers

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Site-specific phosphorylation of tau inhibits amyloid-β toxicity in Alzheimer’s mice

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APOE4-specific Changes in Aβ Accumulation in a New Transgenic Mouse Model of Alzheimer Disease

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On the Seeding and Oligomerization of pGlu-Amyloid Peptides (in vitro)

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Spreading of Neurodegenerative Pathology via Neuron-to-Neuron Transmission of -Amyloid

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Docosahexaenoic Acid-Derived Neuroprotectin D1 Induces Neuronal Survival via Secretase- and PPARγ-Mediated Mechanisms in Alzheimer's Disease Models

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Amyloid-β Peptide Inhibits Activation of the Nitric Oxide/cGMP/cAMP-Responsive Element-Binding Protein Pathway during Hippocampal Synaptic Plasticity

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Aβ neurotoxicity depends on interactions between copper ions, prion protein, and N -methyl- d -aspartate receptors

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Disruption of fast axonal transport is a pathogenic mechanism for intraneuronal amyloid beta

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Interactions of Zn(ii) and Cu(ii) ions with Alzheimer's amyloid-beta peptide. Metal ion binding, contribution to fibrillization and toxicity

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Differential effects of oligomeric and fibrillar amyloid-β1–42 on astrocyte-mediated inflammation

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Lipoprotein-Based Nanoparticles Rescue the Memory Loss of Mice with Alzheimer’s Disease by Accelerating the Clearance of Amyloid-Beta

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A Rapid Label-Free Electrochemical Detection and Kinetic Study of Alzheimer's Amyloid Beta Aggregation

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Amyloid-beta modulates microglial responses by binding to the triggering receptor expressed on myeloid cells 2 (TREM2)

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Binding of zinc(II) and copper(II) to the full‐length Alzheimer’s amyloid‐β peptide

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Overexpression of Human Apolipoprotein A-I Preserves Cognitive Function and Attenuates Neuroinflammation and Cerebral Amyloid Angiopathy in a Mouse Model of Alzheimer Disease

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The role of cell-derived oligomers of Aβ in Alzheimer's disease and avenues for therapeutic intervention

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Amyloid beta ion channel: 3D structure and relevance to amyloid channel paradigm

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The Insulin/Akt Signaling Pathway Is Targeted by Intracellular β-Amyloid

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Drusen deposits associated with aging and age-related macular degeneration contain nonfibrillar amyloid oligomers

Luibl, V. (2006) · Journal of Clinical Investigation · doi:10.1172/jci25843 · Cited by 203

Bifunctional Compounds for Controlling Metal-Mediated Aggregation of the Aβ42Peptide

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Neuroprotective natural antibodies to assemblies of amyloidogenic peptides decrease with normal aging and advancing Alzheimer's disease

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Challenges for Alzheimer's Disease Therapy: Insights from Novel Mechanisms Beyond Memory Defects

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β-Amyloid Is Different in Normal Aging and in Alzheimer Disease

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Soluble Aβ oligomers and protofibrils induce NLRP3 inflammasome activation in microglia

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Heterotypic seeding of Tau fibrillization by pre-aggregated Abeta provides potent seeds for prion-like seeding and propagation of Tau-pathology in vivo

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Targeting the proper amyloid-beta neuronal toxins: a path forward for Alzheimer’s disease immunotherapeutics

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The Amyloid-β Peptide of Alzheimer’s Disease Binds Cu I in a Linear Bis-His Coordination Environment: Insight into a Possible Neuroprotective Mechanism for the Amyloid-β Peptide

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Preparation of Amyloid β‐Protein for Structural and Functional Studies

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Amyloid-β 1–42 Slows Clearance of Synaptically Released Glutamate by Mislocalizing Astrocytic GLT-1

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The p75 Neurotrophin Receptor Promotes Amyloid-β(1-42)-Induced Neuritic Dystrophy In Vitro and In Vivo

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Parallel β-Sheet Fibril and Antiparallel β-Sheet Oligomer: New Insights into Amyloid Formation of Hen Egg White Lysozyme under Heat and Acidic Condition from FTIR Spectroscopy

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Jujuboside A promotes Aβ clearance and ameliorates cognitive deficiency in Alzheimer's disease through activating Axl/HSP90/PPARγ pathway

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