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Heat Shock Proteins 70 and 90 Inhibit Early Stages of Amyloid β-(1–42) Aggregation in Vitro

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Elevation of Glutathione as a Therapeutic Strategy in Alzheimer Disease

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Elucidation of Primary Structure Elements Controlling Early Amyloid β-Protein Oligomerization

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Overview on the Effects of N-Acetylcysteine in Neurodegenerative Diseases

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Amyloid beta peptide and NMDA induce ROS from NADPH oxidase and AA release from cytosolic phospholipase A 2 in cortical neurons

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Amyloid-β Protein Oligomer at Low Nanomolar Concentrations Activates Microglia and Induces Microglial Neurotoxicity

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Amyloid-β Aggregates Cause Alterations of Astrocytic Metabolic Phenotype: Impact on Neuronal Viability

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Aβ-degrading enzymes: potential for treatment of Alzheimer disease

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A 1-42-RAGE Interaction Disrupts Tight Junctions of the Blood-Brain Barrier Via Ca2+-Calcineurin Signaling

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Intracellular Aβ and cognitive deficits precede β-amyloid deposition in transgenic arcAβ mice

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APOE4-specific Changes in Aβ Accumulation in a New Transgenic Mouse Model of Alzheimer Disease

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On the Seeding and Oligomerization of pGlu-Amyloid Peptides (<i>in vitro</i>)

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Amyloid β-Protein Monomer Folding: Free-Energy Surfaces Reveal Alloform-Specific Differences

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β-Amyloid1–42Induces Neuronal Death through the p75 Neurotrophin Receptor

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Elucidation of Amyloid β-Protein Oligomerization Mechanisms: Discrete Molecular Dynamics Study

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High-resolution Atomic Force Microscopy of Soluble Aβ42 Oligomers

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Human neprilysin is capable of degrading amyloid β peptide not only in the monomeric form but also the pathological oligomeric form

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Disruption of fast axonal transport is a pathogenic mechanism for intraneuronal amyloid beta

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Amyloid precursor protein (APP) and the biology of proteolytic processing: relevance to Alzheimer’s disease

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Metal Ions Differentially Influence the Aggregation and Deposition of Alzheimer's β-Amyloid on a Solid Template

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Differential effects of oligomeric and fibrillar amyloid-β1–42 on astrocyte-mediated inflammation

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Rutin inhibits β-amyloid aggregation and cytotoxicity, attenuates oxidative stress, and decreases the production of nitric oxide and proinflammatory cytokines

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Mitochondrial Ca2+ Overload Underlies Aβ Oligomers Neurotoxicity Providing an Unexpected Mechanism of Neuroprotection by NSAIDs

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Zinc-Amyloid β Interactions on a Millisecond Time-Scale Stabilize Non-fibrillar Alzheimer-Related Species

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Physicochemical interactions of amyloid β-peptide with lipid bilayers

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Nanofluidic Biosensing for β-Amyloid Detection Using Surface Enhanced Raman Spectroscopy

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Soluble oligomers of amyloid-β peptide induce neuronal apoptosis by activating a cPLA2-dependent sphingomyelinase-ceramide pathway

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Lipid-based nanoparticles with high binding affinity for amyloid-β1–42 peptide

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Ion Channel Formation by Amyloid-β42 Oligomers but Not Amyloid-β40 in Cellular Membranes

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Subtyping of circulating exosome-bound amyloid β reflects brain plaque deposition

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Bifunctional Compounds for Controlling Metal-Mediated Aggregation of the Aβ₄₂Peptide

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Detection of Misfolded Aβ Oligomers for Sensitive Biochemical Diagnosis of Alzheimer’s Disease

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Rapid Photochemical Cross-LinkingA New Tool for Studies of Metastable, Amyloidogenic Protein Assemblies

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Nucleation-Dependent Polymerization Is an Essential Component of Amyloid-Mediated Neuronal Cell Death

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CSF Biomarkers for Alzheimer's Disease Diagnosis

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Molecular Dynamics Simulation of Amyloid β Dimer Formation

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Alzheimer's Associated β-Amyloid Protein Inhibits Influenza A Virus and Modulates Viral Interactions with Phagocytes

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