Triglycerides as vascular risk factors: new epidemiologic insights.

Kannel, William B; Vasan, Ramachandran S

doi:10.1097/hco.0b013e32832c1284

Cited by 149Lipoproteins and Cardiovascular HealthDiabetes, Cardiovascular Risks, and LipoproteinsLipid metabolism and disorders

Triglycerides as vascular risk factors: new epidemiologic insights.

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Kannel, William B, Vasan, Ramachandran S·2009·Current Opinion in Cardiology

DOI 10.1097/hco.0b013e32832c1284PMC PMC3012388

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Abstract

PURPOSE OF REVIEW: Targeting triglycerides as a vascular risk factor is justified because of the role of triglyceride-rich lipoproteins in atherogenesis. This review examines recent evidence connecting triglycerides with cardiovascular disease (CVD) in the context of advances in insights concerning the pathophysiology, population burden and prognostic impact of fasting versus nonfasting values. RECENT FINDINGS: Cross-sectional surveys indicate that mean triglyceride levels in the United States have increased in recent decades. Although elevated fasting triglycerides are consistently associated with increased CVD risk, adjustment for other risk factors (especially high-density lipoprotein cholesterol (HDL-C)) substantially attenuates this relationship. A recent meta-analysis of 27 prospective studies of western populations reported a triglyceride impact on CVD in both sexes, for both fasting and nonfasting values. Nonfasting triglycerides maintained an independent graded relationship with CVD in fully adjusted analyses, with elevated 4 h postprandial triglyceride imposing a 4.5-fold increment relative to lower levels. SUMMARY: Evidence supports a potential role for both fasting and nonfasting triglycerides as vascular risk factors, owing in part to the accompanying burden of atherogenic remnant particles, small dense low-density lipoprotein, reduced HDL-C and a high frequency of accompanying insulin resistance. Triglyceride-associated CVD risk occurs even in patients with low low-density lipoprotein cholesterol (LDL-C), and lowering both lipids provides more benefit than reducing LDL-C alone.

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Although triglyceride itself is not a component of arterial plaque, cholesterol within triglyceride-rich particles may contribute to plaque development.
“Although triglyceride itself is not a component of arterial plaque, it is thought that cholesterol within triglyceride-rich particles may contribute to plaque development (60, 61).”
From Evaluation and Treatment of Hypertriglyceridemia: An Endocrine Society Clinical Practice Guideline (2012) · cites this paper as [61]
In “New Insights into the role of TG in Atherogenesis” (page unavailable), the paper says triglyceride-rich lipoproteins can enter the arterial wall and become foam cells, supporting their possible contribution to plaque development. It does not state that triglyceride itself is absent from plaque or specifically identify cholesterol within those particles as the contributing component. (LLM, conf=0.82)
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Summary of the second report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel II)
Grundy, S. M., Bilheimer, D. W., Chait, A., Clark, L. T., Denke, M. A., Havel, R. J., Hazzard, W. R., Hulley, S. B., Hunninghake, D. B., Kreisberg, R. A., et al. (1993) · JAMA · doi:10.1001/jama.269.23.3015 · Cited by 27,944 · see citing works
Executive Summary of the Third Report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III)
Expert Panel on Detection, E. (2001) · JAMA: The Journal of the American Medical Association · doi:10.1001/jama.285.19.2486 · Cited by 26,011 · see citing works
Nonfasting triglycerides and risk of myocardial infarction, ischemic heart disease, and death in men and women
Nordestgaard, B. G., Benn, M., Schnohr, P., & Tybjærg-Hansen, A. (2007) · JAMA · doi:10.1001/jama.298.3.299 · Cited by 2,058 · see citing works
Plasma triglyceride level is a risk factor for cardiovascular disease independent of high-density lipoprotein cholesterol level: a meta-analysis of population-based prospective studies
Hokanson, J. E., & Austin, M. A. (1996) · Journal of Cardiovascular Risk · doi:10.1097/00043798-199604000-00014 · Cited by 1,706 · see citing works
Atherogenesis: a postprandial phenomenon.
Zilversmit, D. B. (1979) · Circulation · doi:10.1161/01.cir.60.3.473 · Cited by 1,701 · see citing works
Fasting compared with nonfasting triglycerides and risk of cardiovascular events in women
Bansal, S., Buring, J. E., Rifai, N., Mora, S., Sacks, F. M., & Ridker, P. M. (2007) · JAMA · doi:10.1001/jama.298.3.309 · Cited by 1,619 · see citing works
Atherogenic lipoprotein phenotype. A proposed genetic marker for coronary heart disease risk
Austin, M. A., King, M. C., Vranizan, K. M., & Krauss, R. M. (1990) · Circulation · doi:10.1161/01.cir.82.2.495 · Cited by 1,411 · see citing works
Triglycerides and the Risk of Coronary Heart Disease: 10 158 Incident Cases Among 262 525 Participants in 29 Western Prospective Studies
Sarwar, N., Danesh, J., Eiriksdottir, G., Sigurdsson, G., Wareham, N., Bingham, S., Boekholdt, S. M., Khaw, K.-T., & Gudnason, V. (2007) · Circulation · doi:10.1161/circulationaha.106.637793 · Cited by 1,408 · see citing works
Subendothelial Lipoprotein Retention as the Initiating Process in Atherosclerosis: Update and Therapeutic Implications
Tabas, I., Williams, K. J., & Borén, J. (2007) · Circulation · doi:10.1161/circulationaha.106.676890 · Cited by 1,399 · see citing works
Six new loci associated with blood low-density lipoprotein cholesterol, high-density lipoprotein cholesterol or triglycerides in humans
Kathiresan, S., Melander, O., Guiducci, C., Surti, A., Burtt, N. P., Rieder, M. J., Cooper, G. M., Roos, C., Voight, B. F., Havulinna, A. S., et al. (2008) · Nature Genetics · doi:10.1038/ng.75 · Cited by 1,386 · see citing works
Common variants at 30 loci contribute to polygenic dyslipidemia
Kathiresan, S., Willer, C. J., Peloso, G. M., Demissie, S., Musunuru, K., Schadt, E. E., Kaplan, L., Bennett, D., Li, Y., Tanaka, T., et al. (2009) · Nature Genetics · doi:10.1038/ng.291 · Cited by 1,364 · see citing works
Relation of triglyceride metabolism and coronary artery disease. Studies in the postprandial state
Patsch, J. R., Miesenböck, G., Hopferwieser, T., Mühlberger, V, Knapp, E., Dunn, J. K., Gotto, A. M., & Patsch, W. (1992) · Arteriosclerosis and Thrombosis: A Journal of Vascular Biology · doi:10.1161/01.atv.12.11.1336 · Cited by 1,122 · see citing works
Metabolic origins and clinical significance of LDL heterogeneity
Berneis, K. K., & Krauss, R. M. (2002) · Journal of Lipid Research · doi:10.1194/jlr.r200004-jlr200 · Cited by 896 · see citing works
Non–HDL Cholesterol, Apolipoproteins A-I and B <sub>100</sub> , Standard Lipid Measures, Lipid Ratios, and CRP as Risk Factors for Cardiovascular Disease in Women
Ridker, P. M., Rifai, N., Cook, N. R., Bradwin, G., & Buring, J. E. (2005) · JAMA · doi:10.1001/jama.294.3.326 · Cited by 744 · see citing works
Lipoprotein Management in Patients With Cardiometabolic Risk
Brunzell, J. D., Davidson, M. H., Furberg, C. D., Goldberg, R. B., Howard, B. V., Stein, J. H., & Witztum, J. L. (2008) · Diabetes Care · doi:10.2337/dc08-9018 · Cited by 710 · see citing works
Plasma triglyceride and coronary heart disease
Austin, M. A. (1991) · Arteriosclerosis and Thrombosis: A Journal of Vascular Biology · doi:10.1161/01.atv.11.1.2 · Cited by 697 · see citing works
Overproduction of large VLDL particles is driven by increased liver fat content in man
Adiels, M., Taskinen, M.-R., Packard, C., Caslake, M. J., Soro-Paavonen, A., Westerbacka, J., Vehkavaara, S., Häkkinen, A., Olofsson, S.-O., Yki-Järvinen, H., et al. (2006) · Diabetologia · doi:10.1007/s00125-005-0125-z · Cited by 690 · see citing works
Hyperlipidemia in Coronary Heart Disease I. LIPID LEVELS IN 500 SURVIVORS OF MYOCARDIAL INFARCTION
Goldstein, J. L., Hazzard, W. R., Schrott, H. G., Bierman, E. L., & Motulsky, A. G. (1973) · Journal of Clinical Investigation · doi:10.1172/jci107331 · Cited by 621 · see citing works
Impact of triglyceride levels beyond low-density lipoprotein cholesterol after acute coronary syndrome in the PROVE IT-TIMI 22 trial
Miller, M., Cannon, C. P., Murphy, S. A., Qin, J., Ray, K. K., & Braunwald, E. (2008) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2007.10.038 · Cited by 610 · see citing works
Fasting and Nonfasting Lipid Levels: Influence of Normal Food Intake on Lipids, Lipoproteins, Apolipoproteins, and Cardiovascular Risk Prediction
Langsted, A., Freiberg, J. J., & Nordestgaard, B. G. (2008) · Circulation · doi:10.1161/circulationaha.108.804146 · Cited by 588 · see citing works
Nonfasting triglycerides and risk of ischemic stroke in the general population
Freiberg, J. J. (2008) · JAMA · doi:10.1001/jama.2008.621 · Cited by 542 · see citing works
Low-Density Lipoprotein and High-Density Lipoprotein Particle Subclasses Predict Coronary Events and Are Favorably Changed by Gemfibrozil Therapy in the Veterans Affairs High-Density Lipoprotein Intervention Trial
Otvos, J. D., Collins, D. E., Freedman, D. S., SHALAUROVA, I. Y., Schaefer, E. J., McNamara, J. R., Bloomfield, H. E., & Robins, S. J. (2006) · Circulation · doi:10.1161/circulationaha.105.565135 · Cited by 542 · see citing works
Trends in Serum Lipids and Lipoproteins of Adults, 1960-2002
Carroll, M. D. (2005) · JAMA · doi:10.1001/jama.294.14.1773 · Cited by 529 · see citing works
Lipoprotein Management in Patients With Cardiometabolic Risk
Brunzell, J. D., Davidson, M. H., Furberg, C. D., Goldberg, R. B., Howard, B. V., Stein, J. H., & Witztum, J. L. (2008) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2008.02.034 · Cited by 523 · see citing works
Serum Triglycerides in Coronary Artery Disease
Albrink, M. J. (1959) · Archives of Internal Medicine · doi:10.1001/archinte.1959.00270010010002 · Cited by 466 · see citing works
Fasting compared with nonfasting lipids and apolipoproteins for predicting incident cardiovascular events
Mora, S., Rifai, N., Buring, J. E., & Ridker, P. M. (2008) · Circulation · doi:10.1161/circulationaha.108.777334 · Cited by 445 · see citing works
Serum triglycerides as a risk factor for cardiovascular diseases in the Asia-Pacific region.
Asia Pacific Cohort Studies Collaboration (2004) · Circulation · doi:10.1161/01.cir.0000145615.33955.83 · Cited by 358 · see citing works
Triglyceride-rich lipoproteins isolated by selected-affinity anti-apolipoprotein B immunosorption from human atherosclerotic plaque
Rapp, J. H., Lespine, A., Hamilton, R. L., Colyvas, N., Chaumeton, A. H., Tweedie-Hardman, J., Kotite, L., Kunitake, S. T., Havel, R. J., & Kane, J. P. (1994) · Arteriosclerosis and Thrombosis: A Journal of Vascular Biology · doi:10.1161/01.atv.14.11.1767 · Cited by 340 · see citing works
Selective Retention of VLDL, IDL, and LDL in the Arterial Intima of Genetically Hyperlipidemic Rabbits In Vivo: Molecular Size as a Determinant of Fractional Loss From the Intima–Inner Media
Nordestgaard, B. G., Wootton, R., & Lewis, B. (1995) · Arteriosclerosis, Thrombosis, and Vascular Biology · doi:10.1161/01.atv.15.4.534 · Cited by 334 · see citing works
Mechanisms of HDL lowering in insulin resistant, hypertriglyceridemic states: the combined effect of HDL triglyceride enrichment and elevated hepatic lipase activity
Rashid, S., Watanabe, T., Sakaue, T., & Lewis, G. F. (2003) · Clinical Biochemistry · doi:10.1016/s0009-9120(03)00078-x · Cited by 277 · see citing works
Increased apo A-I and apo A-II fractional catabolic rate in patients with low high density lipoprotein-cholesterol levels with or without hypertriglyceridemia.
Brinton, E. A., Eisenberg, S., & Breslow, J. L. (1991) · Journal of Clinical Investigation · doi:10.1172/jci115028 · Cited by 255 · see citing works
Plasma triglyceride and high density lipoprotein cholesterol as predictors of ischaemic heart disease in British men: The Caerphilly and Speedwell Collaborative Heart Disease Studies
Bainton, D., Miller, N., Bolton, C., YARNELL, J. W. G., Sweetnam, P. M., Baker, I. A., Lewis, B., & Elwood, P. C. (1992) · Heart · doi:10.1136/hrt.68.7.60 · Cited by 210 · see citing works
Quantitative studies of transfer in vivo of low density, Sf 12-60, and Sf 60-400 lipoproteins between plasma and arterial intima in humans.
Shaikh, M., Wootton, R., Nordestgaard, B. G., Baskerville, P., Lumley, J. S., La Ville, A. E., Quiney, J., & Lewis, B. (1991) · Arteriosclerosis and Thrombosis: A Journal of Vascular Biology · doi:10.1161/01.atv.11.3.569 · Cited by 206 · see citing works
Prevalence and extent of dyslipidemia and recommended lipid levels in US adults with and without cardiovascular comorbidities: the National Health and Nutrition Examination Survey 2003–2004
Ghandehari, H., Kamal-Bahl, S., & Wong, N. D. (2008) · American Heart Journal · doi:10.1016/j.ahj.2008.03.005 · Cited by 180 · see citing works
Usefulness of the Triglyceride–High-Density Lipoprotein Versus the Cholesterol–High-Density Lipoprotein Ratio for Predicting Insulin Resistance and Cardiometabolic Risk (from the Framingham Offspring Cohort)
Kannel, W. B., Vasan, R. S., Keyes, M. J., SULLIVAN, L. M., & Robins, S. J. (2008) · The American Journal of Cardiology · doi:10.1016/j.amjcard.2007.09.109 · Cited by 164 · see citing works
Self-citation
Molecular processes that handle — and mishandle — dietary lipids
Williams, K. J. (2008) · Journal of Clinical Investigation · doi:10.1172/jci35206 · Cited by 161 · see citing works
Changes in triglyceride levels and risk for coronary heart disease in young men.
Tirosh, A., Rudich, A., Shochat, T., Tekes-Manova, D., Israeli, E., Henkin, Y., Kochba, I., & Shai, I. (2007) · Annals of Internal Medicine · doi:10.7326/0003-4819-147-6-200709180-00007 · Cited by 151 · see citing works
Multivariate analyses of serum apolipoproteins and risk factors in relation to acute myocardial infarction.
Fager, G., Wiklund, O., Olofsson, S. ‐., Wilhelmsen, L., & Bondjers, G. (1981) · Arteriosclerosis An Official Journal of the American Heart Association Inc · doi:10.1161/01.atv.1.4.273 · Cited by 139 · see citing works
Atherosclerosis and Alimentary Hyperlipemia
Moreton, J. R. (1947) · Science · doi:10.1126/science.106.2748.190 · Cited by 124 · see citing works
Changes in Triglyceride Levels and Risk for Coronary Heart Disease in Young Men
(2007) · Annals of Internal Medicine · doi:10.7326/0003-4819-147-6-200709180-00003 · Cited by 118 · see citing works
Fasting and postprandial determinants for the occurrence of small dense LDL species in non-insulin-dependent diabetic patients with and without hypertriglyceridaemia: the involvement of insulin, insulin precursor species and insulin resistance
Tan, K. C. B., Cooper, M. B., Ling, K. L. E., Griffin, B. A., Freeman, D. J., Packard, C. J., Shepherd, J., Hales, C. N., & Betteridge, D. J. (1995) · Atherosclerosis · doi:10.1016/0021-9150(94)05454-q · Cited by 110 · see citing works
Remnant lipoproteinemia is a risk factor for endothelial vasomotor dysfunction and coronary artery disease in metabolic syndrome.
Nakamura, T., Takano, H., Umetani, K., Kawabata, K.-I., Obata, J.-E., Kitta, Y., Kodama, Y., Mende, A., Ichigi, Y., Fujioka, D., et al. (2005) · Atherosclerosis · doi:10.1016/j.atherosclerosis.2005.01.012 · Cited by 90 · see citing works
The complex role of triglycerides in cardiovascular disease
Abdel-Maksoud, M. F., & Hokanson, J. E. (2002) · Seminars in Vascular Medicine · doi:10.1055/s-2002-35403 · Cited by 76 · see citing works
Postprandial Lipemia Associates with Liver Fat Content
Matikainen, N., Mänttäri, S., Westerbacka, J., Vehkavaara, S., Lundbom, N. M. I., Yki‐Järvinen, H., & Taskinen, M. (2007) · The Journal of Clinical Endocrinology & Metabolism · doi:10.1210/jc.2007-0187 · Cited by 76 · see citing works
Plasma Apolipoprotein C-III Transport in Centrally Obese Men: Associations with Very Low-Density Lipoprotein Apolipoprotein B and High-Density Lipoprotein Apolipoprotein A-I Metabolism
Chan, D. C., Nguyen, M. N., Watts, G. F., & Barrett, P. H. R. (2007) · The Journal of Clinical Endocrinology & Metabolism · doi:10.1210/jc.2006-2676 · Cited by 62 · see citing works
High-Density Lipoprotein (HDL) Transport in the Metabolic Syndrome: Application of a New Model for HDL Particle Kinetics
Ji, J., Watts, G. F., Johnson, A. G., Chan, D. C., Ooi, E. M. M., Rye, K., Serone, A., & Barrett, P. H. R. (2006) · The Journal of Clinical Endocrinology & Metabolism · doi:10.1210/jc.2005-1895 · Cited by 52 · see citing works
Cardioprotection With High-Density Lipoproteins
Das, D. K. (2003) · Circulation Research · doi:10.1161/01.res.0000058881.44913.7b · Cited by 21 · see citing works
Triglyceride-rich lipoprotein remnant levels and metabolism
Brinton, E. A., Nanjee, M. N., & Hopkins, P. N. (2004) · Journal of the American College of Cardiology · doi:10.1016/j.jacc.2004.04.001 · Cited by 9 · see citing works

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Evaluation and Treatment of Hypertriglyceridemia: An Endocrine Society Clinical Practice Guideline
Berglund, L., Brunzell, J. D., Goldberg, A. C., Goldberg, I. J., Sacks, F., Murad, M. H., & Stalenhoef, A. F. H. (2012) · The Journal of Clinical Endocrinology & Metabolism · doi:10.1210/jc.2011-3213 · Cited by 887 · see citing works
“Although triglyceride itself is not a component of arterial plaque, it is thought that cholesterol within triglyceride-rich particles may contribute to plaque development (60, 61).”
Antisense oligonucleotide inhibition of apolipoprotein C-III reduces plasma triglycerides in rodents, nonhuman primates, and humans
Graham, M. J., Lee, R. G., Bell, T. A., Fu, W., Mullick, A. E., Alexander, V. J., Singleton, W., Viney, N., Geary, R., Su, J., et al. (2013) · Circulation Research · doi:10.1161/circresaha.111.300367 · Cited by 377 · see citing works
Assessment and clinical relevance of non-fasting and postprandial triglycerides: an expert panel statement
D. Kolovou, G., P. Mikhailidis, D., Kovar, J., Lairon, D., G. Nordestgaard, B., Chye Ooi, T., Perez-Martinez, P., Bilianou, H., Anagnostopoulou, K., & Panotopoulos, G. (2011) · Current Vascular Pharmacology · doi:10.2174/157016111795495549 · Cited by 306 · see citing works
Apolipoprotein C-III: from pathophysiology to pharmacology
Norata, G. D., Tsimikas, S., Pirillo, A., & Catapano, A. L. (2015) · Trends in Pharmacological Sciences · doi:10.1016/j.tips.2015.07.001 · Cited by 182 · see citing works
Postprandial lipemia as a cardiometabolic risk factor
Pirillo, A., Norata, G. D., & Catapano, A. L. (2014) · Current Medical Research and Opinion · doi:10.1185/03007995.2014.909394 · Cited by 108 · see citing works
Induction of ATF3 Gene Network by Triglyceride-Rich Lipoprotein Lipolysis Products Increases Vascular Apoptosis and Inflammation
Aung, H. H., Lame, M. W., Gohil, K., An, C.-I., Wilson, D. W., & Rutledge, J. C. (2013) · Arteriosclerosis, Thrombosis, and Vascular Biology · doi:10.1161/atvbaha.113.301375 · Cited by 86 · see citing works
Apolipoprotein A-V gene therapy for disease prevention/treatment: a critical analysis
Trudy, M., Vineeta, S., & Robert, O. (2016) · The Journal of Biomedical Research · doi:10.7555/jbr.30.20150059 · Cited by 18 · see citing works

Authors: Kannel, W. B., & Vasan, R. S.
Affiliations: Boston University (US)
Corresponding author: Kannel, William B
ORCID: Vasan, Ramachandran S
Venue: Current Opinion in Cardiology
Publisher: Ovid Technologies (Wolters Kluwer Health)
Published: 2009-07
Volume: 24
Issue: 4
Pages: 345-350
ISSN: 0268-4705 (ISSN-L)
DOI: 10.1097/hco.0b013e32832c1284
PMC: PMC3012388
OpenAlex: W2084500770
Cited by: 149
References: 48
Citations by year: 2026: 1 · 2025: 3 · 2024: 4 · 2023: 1 · 2022: 2 · 2021: 4 · 2020: 9 · 2019: 2 · 2018: 5 · 2017: 7 · 2016: 8 · 2015: 12 · 2014: 13 · 2013: 16 · 2012: 16
FWCI: 3.30
Type: journal article
Language: en
Open access: green
www.ncbi.nlm.nih.gov
Locations: Current Opinion in Cardiology (publishedVersion)
PubMed (publishedVersion)
PubMed Central (acceptedVersion)
Funders: National Institutes of Health (n01hc25195-41-0-1)
National Heart, Lung, and Blood Institute (2K24 HL 04334, N01‐HC25195, K24HL004334, N01-HC-025195)
Topics: Health Sciences › Medicine › Surgery › Lipoproteins and Cardiovascular Health
Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes, Cardiovascular Risks, and Lipoproteins
Health Sciences › Medicine › Cardiology and Cardiovascular Medicine › Lipid metabolism and disorders
Keywords: triglycerides, cardiovascular risk, triglyceride-rich lipoproteins, atherogenesis, fasting triglycerides, postprandial triglycerides, small dense LDL, HDL cholesterol, insulin resistance, lipid-lowering therapy
MeSH terms: Cardiovascular Diseases, Coronary Artery Disease, Humans, Insulin Resistance, Risk Factors, Triglycerides, United States, Hypertriglyceridemia

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Triglycerides as vascular risk factors: new epidemiologic insights.

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