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Cited by 519

The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

TNF-Induced Activation of the Nox1 NADPH Oxidase and Its Role in the Induction of Necrotic Cell Death

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Kim, You-Sun, Morgan, Michael J., Choksi, Swati, Liu, Zheng-gang·2007·Molecular Cell
DOI 10.1016/j.molcel.2007.04.021
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Citations

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Show all 6 references

Research works printed but not linked: 29

  1. Chamseddine, 2003, Gp91phox contributes to NADPH oxidase activity in aortic fibroblasts but not smooth muscle cells, Am. J. Physiol. Heart Circ. Physiol., 285, H2284, 10.1152/ajpheart.00459.2003 · DOI
  2. Chen, 2002, TNF-R1 signaling: a beautiful pathway, Science, 296, 1634, 10.1126/science.1071924 · DOI · Paper page
  3. Cheng, 2004, NOXO1, regulation of lipid binding, localization, and activation of Nox1 by the Phox homology (PX) domain, J. Biol. Chem., 279, 4737, 10.1074/jbc.M305968200 · DOI
  4. Cheng, 2006, Nox1-dependent reactive oxygen generation is regulated by Rac1, J. Biol. Chem., 281, 17718, 10.1074/jbc.M512751200 · DOI · Paper page
  5. Dang, 2006, A specific p47phox -serine phosphorylated by convergent MAPKs mediates neutrophil NADPH oxidase priming at inflammatory sites, J. Clin. Invest., 116, 2033, 10.1172/JCI27544 · DOI · Paper page
  6. Dewas, 2003, TNF-alpha induces phosphorylation of p47(phox) in human neutrophils: partial phosphorylation of p47phox is a common event of priming of human neutrophils by TNF-alpha and granulocyte-macrophage colony-stimulating factor, J. Immunol., 171, 4392, 10.4049/jimmunol.171.8.4392 · DOI
  7. Fiers, 1999, More than one way to die: apoptosis, necrosis and reactive oxygen damage, Oncogene, 18, 7719, 10.1038/sj.onc.1203249 · DOI · Paper page
  8. Frey, 2002, PKCzeta regulates TNF-alpha-induced activation of NADPH oxidase in endothelial cells, Circ. Res., 90, 1012, 10.1161/01.RES.0000017631.28815.8E · DOI
  9. Heyworth, 2003, Chronic granulomatous disease, Curr. Opin. Immunol., 15, 578, 10.1016/S0952-7915(03)00109-2 · DOI · Paper page
  10. Hilenski, 2004, Distinct subcellular localizations of Nox1 and Nox4 in vascular smooth muscle cells, Arterioscler. Thromb. Vasc. Biol., 24, 677, 10.1161/01.ATV.0000112024.13727.2c · DOI · Paper page
  11. Holler, 2000, Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule, Nat. Immunol., 1, 489, 10.1038/82732 · DOI · Paper page
  12. Kamata, 2005, Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases, Cell, 120, 649, 10.1016/j.cell.2004.12.041 · DOI · Paper page
  13. Karin, 2002, NF-kappaB at the crossroads of life and death, Nat. Immunol., 3, 221, 10.1038/ni0302-221 · DOI · Paper page
  14. Legler, 2003, Recruitment of TNF receptor 1 to lipid rafts is essential for TNFalpha-mediated NF-kappaB activation, Immunity, 18, 655, 10.1016/S1074-7613(03)00092-X · DOI · Paper page
  15. Lin, 1999, Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis, Genes Dev., 13, 2514, 10.1101/gad.13.19.2514 · DOI · Paper page
  16. Lin, 2004, Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation, J. Biol. Chem., 279, 10822, 10.1074/jbc.M313141200 · DOI · Paper page
  17. Liu, 2000, Activation of apoptosis signal-regulating kinase 1 (ASK1) by tumor necrosis factor receptor-associated factor 2 requires prior dissociation of the ASK1 inhibitor thioredoxin, Mol. Cell. Biol., 20, 2198, 10.1128/MCB.20.6.2198-2208.2000 · DOI · Paper page
  18. Miyano, 2006, Direct involvement of the small GTPase Rac in activation of the superoxide-producing NADPH oxidase Nox1, J. Biol. Chem., 281, 21857, 10.1074/jbc.M513665200 · DOI
  19. Nguyen, 2003, Null mutation of gp91phox reduces muscle membrane lysis during muscle inflammation in mice, J. Physiol., 553, 833, 10.1113/jphysiol.2003.051912 · DOI
  20. Sakon, 2003, NF-kappaB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death, EMBO J., 22, 3898, 10.1093/emboj/cdg379 · DOI · Paper page
  21. Saran, 2003, To what end does nature produce superoxide? NADPH oxidase as an autocrine modifier of membrane phospholipids generating paracrine lipid messengers, Free Radic. Res., 37, 1045, 10.1080/10715760310001594631 · DOI
  22. Suh, 1999, Cell transformation by the superoxide-generating oxidase Mox1, Nature, 401, 79, 10.1038/43459 · DOI · Paper page
  23. Tang, 2001, Inhibition of JNK activation through NF-kappaB target genes, Nature, 414, 313, 10.1038/35104568 · DOI · Paper page
  24. Teixeira, 1999, Production of singlet oxygen by eosinophils activated in vitro by C5a and leukotriene B4, FEBS Lett., 453, 265, 10.1016/S0014-5793(99)00728-0 · DOI
  25. Tobiume, 2001, ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis, EMBO Rep., 2, 222, 10.1093/embo-reports/kve046 · DOI · Paper page
  26. Ueyama, 2006, Involvement of Rac1 in activation of multicomponent Nox1- and Nox3-based NADPH oxidases, Mol. Cell. Biol., 26, 2160, 10.1128/MCB.26.6.2160-2174.2006 · DOI
  27. Ventura, 2006, Chemical genetic analysis of the time course of signal transduction by JNK, Mol. Cell, 21, 701, 10.1016/j.molcel.2006.01.018 · DOI · Paper page
  28. Wajant, 2003, Tumor necrosis factor signaling, Cell Death Differ., 10, 45, 10.1038/sj.cdd.4401189 · DOI · Paper page
  29. Yu, 1999, Biosynthesis of flavocytochrome b558. gp91(phox) is synthesized as a 65-kDa precursor (p65) in the endoplasmic reticulum, J. Biol. Chem., 274, 4364, 10.1074/jbc.274.7.4364 · DOI

Cited by

Metadata

Authors
Kim, Y.-S., Morgan, M. J., Choksi, S., & Liu, Z.-G.
Venue
Molecular Cell
Publisher
Elsevier BV
Published
2007-06
Volume
26
Issue
5
Pages
675-687
ISSN
1097-2765 (ISSN-L)
DOI
10.1016/j.molcel.2007.04.021
OpenAlex
W2104421483
Cited by
519
References
6
FWCI
13.53
Type
journal article
Language
en
Open access
bronze
Licence

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