Hormonal mechanisms of recovery from insulin-induced hypoglycemia in man.
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Other counter-regulatory hormones like cortisol or thyroxin play only a minor role for the liver.
“Other counter-regulatory hormones like cortisol or thyroxin play only a minor role for the liver [10], [12], [13].”
From Quantifying the Contribution of the Liver to Glucose Homeostasis: A Detailed Kinetic Model of Human Hepatic Glucose Metabolism (2012) · cites this paper as [12]
Epinephrine compensates largely for deficient glucagon secretion.
“Glucagon plays a primary counterregulatory role; epinephrine compensates largely for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
Glucose recovery is not due solely to dissipation of insulin.
“Glucose recovery is not due solely to dissipation of insulin.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
The principles of hypoglycemic glucose counterregulation and those of nonhypoglycemic glucose counterregulation in both the postprandial and postabsorptive states are fundamentally the same.
“Thus, the principles of hypoglycemic glucose counterregulation (1-7) and those of nonhypoglycemic glucose counterregulation in both the postprandial (8, 9) and the postabsorptive states are fundamentally the same.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
Glucagon plays a primary counterregulatory role.
“Glucagon plays a primary counterregulatory role; epinephrine compensates largely for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
In both hypoglycemic glucose counterregulation and another model of nonhypoglycemic glucose counterregulation, adrenomedullary epinephrine, but not sympathetic neural norepinephrine, compensates for deficient glucagon secretion.
“Second, in both hypoglycemic glucose counterregulation (1-7) and in another model of nonhypoglycemic glucose counterregulation (9), adrenomedullary epinephrine, but not sympathetic neural norepinephrine, compensates for deficient glucagon secretion.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
Postabsorptive plasma glucose concentrations are not discernibly reduced in epinephrine-deficient (adrenalectomized) persons.
“and postabsorptive plasma glucose concentrations are not discernibly reduced in epinephrine-deficient (adrenalectomized) persons (3, 9).”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
Glucose recovery fails to occur only in the absence of both glucagon and epinephrine.
“Glucose recovery fails to occur only in the absence of both glucagon and epinephrine.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
Insulin-induced hypoglycemia fails to produce an increase in plasma norepinephrine in persons who have undergone bilateral adrenalectomy, indicating that the small increment seen in normal subjects is the result of norepinephrine release from the adrenal medullae rather than from sympathetic nerves.
“Insulin-induced hypoglycemia fails to produce an in'crease in plasma norepinephrine in persons who have undergone bilateral adrenalectomy (3), indicating that the small increment seen in normal subjects is the result of norepinephrine release from the adrenal medullae rather than from sympathetic nerves.”
From Epinephrine supports the postabsorptive plasma glucose concentration and prevents hypoglycemia when glucagon secretion is deficient in man (1984) · cites this paper as [3]
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- Authors
- Gerich, J., Davis, J., Lorenzi, M., Rizza, R., Bohannon, N., Karam, J., Lewis, S., Kaplan, R., Schultz, T., & Cryer, P.
- ORCID
- Venue
- American Journal of Physiology-Endocrinology and Metabolism
- Publisher
- American Physiological Society
- Published
- 1979-04-01
- Volume
- 236
- Issue
- 4
- Pages
- E380
- ISSN
- 0193-1849 (ISSN-L)
- 1522-1555
- OpenAlex
- W2409751958
- Cited by
- 183
- References
- 17
- Citations by year
- 2025: 2 · 2024: 1 · 2022: 1 · 2021: 4 · 2020: 3 · 2019: 2 · 2018: 1 · 2017: 1 · 2016: 3 · 2015: 4 · 2014: 2 · 2012: 4
- FWCI
- 7.89
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- American Journal of Physiology-Endocrinology and Metabolism (publishedVersion)
- PubMed (publishedVersion)
- Topics
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Diabetes Management and Research
- Health Sciences › Medicine › Surgery › Pancreatic function and diabetes
- Health Sciences › Medicine › Endocrinology, Diabetes and Metabolism › Growth Hormone and Insulin-like Growth Factors
- Keywords
- insulin-induced hypoglycemia, hormonal mechanisms, counterregulatory hormones, glucose homeostasis, insulin
- MeSH terms
- Adult, Blood Glucose, Epinephrine, Female, Glucagon, Humans, Hydrocortisone, Hypoglycemia, Insulin, Male, Norepinephrine, Somatostatin, Growth Hormone
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
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