Student or educator? Sign up with your school email and get 6 months of Pro free.
Cited by 4,058

The DOI matches its Crossref record.

Isolated Hand Paresis: A Case Series

Open access
Alstadhaug, Karl B., Sjulstad, Ane·2013·Cerebrovascular Diseases Extra

Abstract

Background: Hand knob infarction is a well-known stroke entity. Based on very limited data, embolic stroke mechanism has been considered the most frequent cause; however, prognosis is considered good. We wanted to shed more light on this phenomenon by assessing a cohort of patients referred to a general hospital stroke unit. Methods: Every subject admitted to our stroke unit with an acute isolated hand paresis in the period from 2007 to 2012 was identified prospectively. Patients who had suffered from a stroke in the hand motor cortex or an adjacent area explaining the acute loss of hand function were included in the study. The Trial of Org 10172 in Acute Stroke Treatment criteria were used to classify subtypes of stroke according to etiology. The patients were followed up during autumn 2012. Results: Seventeen subjects were admitted, but in 2 of them symptoms were transitory and magnetic resonance imaging was negative. Two patients were excluded due to persisting sensory deficits. The remaining 13 (11 males and 2 females) patients with an average age of 62.9 (±13.4) years were included, representing 1.5% of all ischemic strokes diagnosed at the stroke unit in the given period. All patients were right-handed, and the dominant hand was affected only in 4 (31%). The average Medical Research Council's scale score was 3.1 (±1.4) on admission, and classified as bad. On follow-up, which occurred on average 29.8 (±19.8) months after the stroke, the score was 4.6 (±0.4) and was classified as fair to good. No patient experienced a new stroke. The outcome was good to excellent in 10 patients (77%). Two patients died (15%), 1 of probable cardiac arrest and 1 of unknown cause. One patient did not participate in the follow-up. The majority of patients had evidence of both small artery (77%) and large artery (85%) disease. On average, there were 1.6 (±0.4) new ischemic lesions per patient. Six patients had a solitary lesion (46%). In 5 of them, small artery occlusion was considered the probable stroke mechanism. In 4 cases, the stroke was of undetermined etiology. Three patients had atrial fibrillation, and in 2 of them cardioembolism was the probable stroke mechanism. Two patients with definite large artery atherosclerosis underwent carotid endarterectomy, and 1 of them had comorbid atrial fibrillation. Conclusion: Strokes causing isolated hand paresis seem to have a heterogeneous etiology. Prognosis regarding hand function is good, but long-term outcome depends on stroke etiology and secondary prophylaxis.

Overview

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Integrity signals

Registry

Crossref · 10.1159/000350708

Cites

Statistics

Verification

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Citations

Cites

No references could be resolved yet.

Research works printed but not linked: 18

  1. 10.1016/0303-8467(95)00037-K · DOI
  2. 10.1093/brain/120.1.141 · DOI · Paper page
  3. 10.1016/j.surneu.2005.07.049 · DOI
  4. 10.3174/ajnr.A0597 · DOI
  5. 10.1212/WNL.58.9.1412 · DOI
  6. 10.1016/S0140-6736(04)16047-9 · DOI
  7. 10.1136/jnnp.2007.127167 · DOI
  8. 10.1212/WNL.57.9.1589 · DOI
  9. 10.1016/j.jstrokecerebrovasdis.2003.09.007 · DOI
  10. 10.1097/NRL.0b013e31814db093 · DOI
  11. 10.1007/s00415-009-5104-8 · DOI
  12. 10.1161/01.STR.19.9.1074 · DOI
  13. 10.1161/01.STR.24.1.35 · DOI · Paper page
  14. 10.1159/000072554 · DOI · Paper page
  15. 10.1161/01.STR.24.8.1178 · DOI · Paper page
  16. 10.1093/brain/awh317 · DOI
  17. 10.1159/000047650 · DOI
  18. 10.1371/journal.pone.0031275 · DOI

Cited by

Metadata

Authors
Alstadhaug, K. B., & Sjulstad, A.
Venue
Cerebrovascular Diseases Extra
Publisher
S. Karger AG
Published
2013-04-17
Volume
3
Issue
1
Pages
65-73
ISSN
1664-5456 (ISSN-L)
OpenAlex
W2067395123
Cited by
4,058
References
0
FWCI
135.55
Type
journal article
Language
en
Open access
gold
Licence

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.