Cited by 729Regulation of Appetite and ObesityAdipokines, Inflammation, and Metabolic DiseasesPancreatic function and diabetes

Effects of parabiosis of obese with diabetes and normal mice.

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Coleman, D. L.·1973·Diabetologia

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Crossref · 10.1007/bf01221857

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  • ob and db are recessive mouse mutations that cause massive obesity as a result of profound hyperphagia and reduced energy expenditure.

    “ob and db are recessive mouse mutations that cause massive obesity as a result of profound hyperphagia and reduced energy expenditure.”

    From Leptin at 14 y of age: an ongoing story (2009) · cites this paper as [19]

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Authors
Coleman, D. L.
Affiliations
Jackson Laboratory (US)
Corresponding author
Coleman, D. L.
Venue
Diabetologia
Published
1973-08
Volume
9
Issue
4
Pages
294-298
ISSN
  • 0012-186X (ISSN-L)
  • 1432-0428
OpenAlex
W2041553669
Cited by
729
References
7
Citations by year
2026: 6 · 2025: 4 · 2024: 6 · 2023: 11 · 2022: 10 · 2021: 32 · 2020: 22 · 2019: 20 · 2018: 9 · 2017: 22 · 2016: 13 · 2015: 24 · 2014: 34 · 2013: 15 · 2012: 34
FWCI
2.79
Type
journal article
Language
en
Open access
Locations
  • Diabetologia (publishedVersion)
  • PubMed (publishedVersion)
  • The Mouseion at the JAXlibrary (Jackson Laboratory) (submittedVersion)
Topics
  • Life Sciences › Neuroscience › Endocrine and Autonomic Systems › Regulation of Appetite and Obesity
  • Health Sciences › Medicine › Epidemiology › Adipokines, Inflammation, and Metabolic Diseases
  • Health Sciences › Medicine › Surgery › Pancreatic function and diabetes
Keywords
parabiosis, ob/ob mice, db/db mice, obesity, diabetes mellitus, satiety factor, satiety center, food intake regulation, hypoglycemia, weight loss
MeSH terms
Animals, Body Weight, Breeding, Diabetes Mellitus, Eating, Feeding Behavior, Female, Genetics, Hyperglycemia, Hypoglycemia, Male, Obesity, Parabiosis, Satiation, Starvation, Mice

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