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Hypoxia‐inhibited dual‐specificity phosphatase‐2 expression in endometriotic cells regulates cyclooxygenase‐2 expression

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Estrogen receptor β and matrix metalloproteinase 1 are coexpressed in uterine endometrium and endometriotic lesions of patients with endometriosis

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Leptin receptor is induced in endometriosis and leptin stimulates the growth of endometriotic epithelial cells through the JAK2/STAT3 and ERK pathways

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ORIGINAL ARTICLE: Endometrial Osteopontin mRNA Expression and Plasma Osteopontin Levels are Increased in Patients with Endometriosis

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DNA methylation patterns of steroid receptor genes ESR1, ESR2 and PGR in deep endometriosis compromising the rectum

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Hypoxia and estrogen are functionally equivalent in breast cancer-endothelial cell interdependence

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Suppression of annexin A2 by prostaglandin E2 impairs phagocytic ability of peritoneal macrophages in women with endometriosis

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The Mammalian Target of Rapamycin-p70 Ribosomal S6 Kinase but Not Phosphatidylinositol 3-Kinase-Akt Signaling Is Responsible for Fibroblast Growth Factor-9-induced Cell Proliferation

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Premenstrual Regulation of the Pro-Angiogenic Factor CYR61 in Human Endometrium

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Identification of multiple and distinct defects in prostaglandin biosynthetic pathways in eutopic and ectopic endometrium of women with endometriosis

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Estrogen receptor expression affected by hypoxia inducible factor-1α in stromal cells from patients with endometriosis

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Effect of oxygen tensions on the proliferation and angiogenesis of endometriosis heterograft in severe combined immunodeficiency mice

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Expression of Inducible Microsomal Prostaglandin E Synthase in Local Lesions of Endometriosis Patients

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Peer Review #3 of "TET1 may contribute to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis (v0.2)"

(2020) · PeerJ · doi:10.7287/peerj.9950v0.2/reviews/3

Peer Review #1 of "TET1 may contribute to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis (v0.1)"

(2020) · PeerJ · doi:10.7287/peerj.9950v0.1/reviews/1

Peer Review #3 of "TET1 may contribute to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis (v0.1)"

(2020) · PeerJ · doi:10.7287/peerj.9950v0.1/reviews/3

Peer Review #2 of "TET1 may contribute to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis (v0.2)"

(2020) · PeerJ · doi:10.7287/peerj.9950v0.2/reviews/2

Peer Review #2 of "TET1 may contribute to hypoxia-induced epithelial to mesenchymal transition of endometrial epithelial cells in endometriosis (v0.1)"

(2020) · PeerJ · doi:10.7287/peerj.9950v0.1/reviews/2

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Endometriosis is the survivor of reflux endometrium by hypoxia selection—As the phoenix nirvana, a regeneration story of endometrial cells

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