Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease

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Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins

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Ibuprofen Suppresses Plaque Pathology and Inflammation in a Mouse Model for Alzheimer's Disease

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Aβ Immunotherapy Leads to Clearance of Early, but Not Late, Hyperphosphorylated Tau Aggregates via the Proteasome

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Temporal control of gene expression in transgenic mice by a tetracycline-responsive promoter.

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Chronic Treatment with the γ-Secretase Inhibitor LY-411,575 Inhibits β-Amyloid Peptide Production and Alters Lymphopoiesis and Intestinal Cell Differentiation

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Amyloid β Protein (Aβ) in Alzheimeri's Disease Brain

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NSAIDs and enantiomers of flurbiprofen target γ-secretase and lower Aβ42 in vivo

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Anti-Inflammatory Drug Therapy Alters β-Amyloid Processing and Deposition in an Animal Model of Alzheimer's Disease

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Imaging of amyloid-β deposits in brains of living mice permits direct observation of clearance of plaques with immunotherapy

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Neprilysin Gene Transfer Reduces Human Amyloid Pathology in Transgenic Mice

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A vector for expressing foreign genes in the brains and hearts of transgenic mice

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Microglial Activation and β-Amyloid Deposit Reduction Caused by a Nitric Oxide-Releasing Nonsteroidal Anti-Inflammatory Drug in Amyloid Precursor Protein Plus Presenilin-1 Transgenic Mice

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A COMPARATIVE STUDY OF MODIFIED BIELSCHOWSKY, BODIAN AND THIOFLAVIN S STAINS ON ALZHEIMER'S NEUROFIBRILLARY TANGLES

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Passive Amyloid Immunotherapy Clears Amyloid and Transiently Activates Microglia in a Transgenic Mouse Model of Amyloid Deposition

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ApoE-Directed Therapeutics Rapidly Clear β-Amyloid and Reverse Deficits in AD Mouse Models

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Relevance of transgenic mouse models for Alzheimer's disease

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Forebrain overexpression of α-synuclein leads to early postnatal hippocampal neuron loss and synaptic disruption

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In VivoOlfactory Model of APP-Induced Neurodegeneration Reveals a Reversible Cell-Autonomous Function

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Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease-0

Joanna L Jankowsky, H. H. S. (2011) · Figshare · doi:10.6084/m9.figshare.52080

Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease-1

Joanna L Jankowsky, H. H. S. (2011) · Figshare · doi:10.6084/m9.figshare.34517

Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease-4

Joanna L Jankowsky, H. H. S. (2011) · Figshare · doi:10.6084/m9.figshare.52084

Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease-2

Joanna L Jankowsky, H. H. S. (2011) · Figshare · doi:10.6084/m9.figshare.52082

Persistent Amyloidosis following Suppression of Aβ Production in a Transgenic Model of Alzheimer Disease-3

Joanna L Jankowsky, H. H. S. (2011) · Figshare · doi:10.6084/m9.figshare.52083