Eating Behavior
Abstract
Overconsumption is a major cause of weight gain and obesity. An understanding of the processes controlling eating behavior can throw light on the causes of overconsumption and provide information to prevent undesirable and nonintentional overeating. Although eating is usually considered a fundamental component of the energy homeostasis system, it can occur in the absence of any strong physiologic need, and persist in the presence of recognized satiety signals. It is, however, important to understand the specific physiologic controls and the limits of their impact on food intake. Particularly important is the identification of episodic and tonic physiologic signals, their potency and time course of action. In addition, the operation of the homeostatic mechanisms must be understood in relation to the hedonic processes influencing eating. The current view is that hedonic responses can overcome regulatory processes of homeostasis and this constitutes a mechanism that drives overconsumption. An important distinction is between liking and wanting, and the extent to which these processes act both separately and conjointly to influence food selection and overconsumption. Taken together, these operations should be interpreted in the face of a huge diversity in the expression of eating behavior – across geographical and cultural zones, and within specific ethnic and social groups. In all situations there exists widespread individual variability which is reflected in a spectrum from susceptibility to resistance to overconsumption and weight gain. In an obesogenic environment, not everyone becomes fat. The identification and characterization of susceptible and resistant phenotypes is an important theoretical and practical objective in the study of eating behavior and its relationship to obesity.
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Oro-sensory, gastric, and intestinal signals interact on their effect on early satiation in humans.
“Taken together, oro-sensory, gastric and intestinal signals, which represent episodic control, interact on their effect on early satiation in humans [10].”
From Homeostatic and Circadian Control of Food Intake: Clinical Strategies to Prevent Overconsumption (2013) · cites this paper as [10]
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Homeostatic and Circadian Control of Food Intake: Clinical Strategies to Prevent Overconsumption
Drapeau, V., & Gallant, A. R. (2013) · Current Obesity Reports · doi:10.1007/s13679-012-0038-3 · Cited by 10 · see citing works
- Authors
- Blundell, J. E., Finlayson, G., & Halford, J. C. G.
- Affiliations
- University of Leeds (GB)
- University of Liverpool (GB)
- Venue
- Clinical Obesity in Adults and Children
- Publisher
- Wiley
- Published
- 2009-07-29
- Pages
- 134-150
- OpenAlex
- W2911701386
- Cited by
- 1
- References
- 133
- Citations by year
- 2012: 1
- Type
- other
- Language
- en
- Open access
- closed
- Topics
- Life Sciences › Neuroscience › Endocrine and Autonomic Systems › Regulation of Appetite and Obesity
- Social Sciences › Psychology › Clinical Psychology › Eating Disorders and Behaviors
- Health Sciences › Nursing › Nutrition and Dietetics › Nutrition, Health and Food Behavior
- Keywords
- dietary habits, overconsumption, obesity, energy homeostasis, satiety signals, hedonic eating, food liking, desire to eat, food choice, obesogenic environment
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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