New fundamental resistance exercise determinants of molecular and cellular muscle adaptations

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The Molecular Basis of Skeletal Muscle Atrophy

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Myostatin Inhibits Myoblast Differentiation by Down-regulating MyoD Expression

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Myostatin negatively regulates satellite cell activation and self-renewal

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Signalling pathways that mediate skeletal muscle hypertrophy and atrophy

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HGF/SF Is Present in Normal Adult Skeletal Muscle and Is Capable of Activating Satellite Cells

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Regulation of mitochondrial biogenesis in skeletal muscle by CaMK

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Muscle size and strength are increased following walk training with restricted venous blood flow from the leg muscle, Kaatsu-walk training

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The Kinase Domain of Titin Controls Muscle Gene Expression and Protein Turnover

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Mechanisms of Exercise-Induced Muscle Fibre Injury

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Insulin-like Growth Factor-1 (IGF-1) Inversely Regulates Atrophy-induced Genes via the Phosphatidylinositol 3-Kinase/Akt/Mammalian Target of Rapamycin (PI3K/Akt/mTOR) Pathway*

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The effects of heavy resistance training and detraining on satellite cells in human skeletal muscles

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Patterns of gene expression in atrophying skeletal muscles: response to food deprivation

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Response of Skeletal Muscle Mitochondria to Hypoxia

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A Discussion on Different Approaches for Prescribing Physical Interventions – Four Roads Lead to Rome, but Which One Should We Choose?

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Trainingsrelevante Determinanten der molekularen und zellulären Skelettmuskeladaptation. Teil 1: Einleitung und Längenadaptation

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Untitled work

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