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Cited by 631

The DOI matches its Crossref record; 35 of the 35 references it cites were found in a registry or an index.

Obesity and polycystic ovary syndrome

Open access
Barber, T. M., McCarthy, M. I., Wass, J. A. H., Franks, S.·2006·Clinical Endocrinology
DOI 10.1111/j.1365-2265.2006.02587.x
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Abstract

Summary: The aetiology of Polycystic Ovary Syndrome (PCOS) is complex and multifactorial. There is much evidence, however, to suggest that adipose tissue plays an important role in the development and maintenance of PCOS pathology. There is a close correlation between adiposity and symptom severity in women with PCOS, and even modest reductions in weight generally translate into significant improvements in menstrual regularity, fertility and hyperandrogenic features. This review article considers the various mechanisms that might underlie this link between excess adiposity and PCOS – including the effects of differential insulin sensitivity, abnormal steroid hormone metabolism and adipocytokine secretion. Greater attention to the therapeutic options available to reduce the impact of excess adiposity on ovarian and metabolic function is essential to the management of PCOS.

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Crossref · 10.1111/j.1365-2265.2006.02587.x

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Metadata

Authors
Barber, T. M., McCarthy, M. I., Wass, J. A. H., & Franks, S.
Venue
Clinical Endocrinology
Publisher
Wiley
Published
2006-07-06
Volume
65
Issue
2
Pages
137-145
ISSN
  • 0300-0664 (ISSN-L)
  • 1365-2265
DOI
10.1111/j.1365-2265.2006.02587.x
OpenAlex
W1977875804
Cited by
631
References
35
FWCI
23.09
Type
journal article
Language
en
Open access
bronze

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Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.