Exercise and the regulation of skeletal muscle hypertrophy

mTOR Signaling in Growth Control and Disease

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TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling

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Progression Models in Resistance Training for Healthy Adults

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Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo

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mTOR signaling at a glance

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Exercise Metabolism and the Molecular Regulation of Skeletal Muscle Adaptation

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Rheb GTPase is a direct target of TSC2 GAP activity and regulates mTOR signaling

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Hormonal Responses and Adaptations to Resistance Exercise and Training

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Regulation of 4E-BP1 phosphorylation: a novel two-step mechanismPreprint

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Insulin signalling to mTOR mediated by the Akt/PKB substrate PRAS40

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Skeletal muscle atrophy and the E3 ubiquitin ligases MuRF1 and MAFbx/atrogin-1

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TRANSLATIONAL CONTROL IN MAMMALIAN CELLS

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Ingested protein dose response of muscle and albumin protein synthesis after resistance exercise in young men

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Regulation of elongation factor 2 kinase by p90RSK1 and p70 S6 kinase

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Protein supplementation augments the adaptive response of skeletal muscle to resistance-type exercise training: a meta-analysis

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Rapamycin differentially inhibits S6Ks and 4E-BP1 to mediate cell-type-specific repression of mRNA translation

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Protein Ingestion to Stimulate Myofibrillar Protein Synthesis Requires Greater Relative Protein Intakes in Healthy Older Versus Younger Men

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Age‐related differences in the dose–response relationship of muscle protein synthesis to resistance exercise in young and old men

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Phosphorylation of p70^S6kcorrelates with increased skeletal muscle mass following resistance exercise

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Regulation of mTORC1 and its impact on gene expression at a glance

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Resistance exercise load does not determine training-mediated hypertrophic gains in young men

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Postexercise net protein synthesis in human muscle from orally administered amino acids

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Mammalian Target of Rapamycin (mTOR): Conducting the Cellular Signaling Symphony

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Consumption of fat-free fluid milk after resistance exercise promotes greater lean mass accretion than does consumption of soy or carbohydrate in young, novice, male weightlifters

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Timing and distribution of protein ingestion during prolonged recovery from resistance exercise alters myofibrillar protein synthesis

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Disassociation between the effects of amino acids and insulin on signaling, ubiquitin ligases, and protein turnover in human muscle

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Dissecting the role of mTOR: Lessons from mTOR inhibitors

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Rapamycin administration in humans blocks the contraction‐induced increase in skeletal muscle protein synthesis

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Immobilization induces anabolic resistance in human myofibrillar protein synthesis with low and high dose amino acid infusion

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Effects of aging on human skeletal muscle after immobilization and retraining

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Skeletal muscle atrophy during short-term disuse: Implications for age-related sarcopenia

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Two Weeks of Reduced Activity Decreases Leg Lean Mass and Induces “Anabolic Resistance” of Myofibrillar Protein Synthesis in Healthy Elderly

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Untitled work

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Aging impairs contraction-induced human skeletal muscle mtorc1 signaling and protein synthesis

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Variability in Muscle Size and Strength Gain After Unilateral Resistance Training

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The temporal responses of protein synthesis, gene expression and cell signalling in human quadriceps muscle and patellar tendon to disuse

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Enhanced Amino Acid Sensitivity of Myofibrillar Protein Synthesis Persists for up to 24 h after Resistance Exercise in Young Men1–3

Burd, N. A., West, D. W. D., Moore, D. R., Atherton, P. J., Staples, A. W., Prior, T., Tang, J. E., Rennie, M. J., Baker, S. K., & Phillips, S. M. (2011) · The Journal of Nutrition · doi:10.3945/jn.110.135038 · Cited by 325 · see citing works

Alterations of protein turnover underlying disuse atrophy in human skeletal muscle

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Supplementation of a suboptimal protein dose with leucine or essential amino acids: effects on myofibrillar protein synthesis at rest and following resistance exercise in men

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Resistance exercise volume affects myofibrillar protein synthesis and anabolic signalling molecule phosphorylation in young men

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The role of phospholipase D and phosphatidic acid in the mechanical activation of mTOR signaling in skeletal muscle

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A housekeeper with power of attorney: the rRNA genes in ribosome biogenesis

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The role of skeletal muscle mTOR in the regulation of mechanical load‐induced growth

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Elevations in ostensibly anabolic hormones with resistance exercise enhance neither training-induced muscle hypertrophy nor strength of the elbow flexors

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Resistance exercise‐induced increases in putative anabolic hormones do not enhance muscle protein synthesis or intracellular signalling in young men

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The involvement of the ubiquitin proteasome system in human skeletal muscle remodelling and atrophy

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A functional insulin‐like growth factor receptor is not necessary for load‐induced skeletal muscle hypertrophy

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Muscular adaptations in low- versus high-load resistance training: a meta-analysis.

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Cluster analysis tests the importance of myogenic gene expression during myofiber hypertrophy in humans

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Acute Post-Exercise Myofibrillar Protein Synthesis Is Not Correlated with Resistance Training-Induced Muscle Hypertrophy in Young Men

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Ribosome biogenesis: Emerging evidence for a central role in the regulation of skeletal muscle mass

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Growth hormone stimulates the collagen synthesis in human tendon and skeletal muscle without affecting myofibrillar protein synthesis

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Molecular Networks of Human Muscle Adaptation to Exercise and Age

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SKAR Is a Specific Target of S6 Kinase 1 in Cell Growth Control

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Calpains in muscle wasting

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Early activation of mTORC1 signalling in response to mechanical overload is independent of phosphoinositide 3‐kinase/Akt signalling

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The Epidemiology of Sarcopenia

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Muscular and Systemic Correlates of Resistance Training-Induced Muscle Hypertrophy

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Evolution of sarcopenia research

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Disuse Impairs the Muscle Protein Synthetic Response to Protein Ingestion in Healthy Men

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Suppression of endogenous testosterone production attenuates the response to strength training: a randomized, placebo-controlled, and blinded intervention study

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Focal adhesion proteins FAK and paxillin increase in hypertrophied skeletal muscle

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Technics of progressive resistance exercise.

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The Role of Diacylglycerol Kinase ζ and Phosphatidic Acid in the Mechanical Activation of Mammalian Target of Rapamycin (mTOR) Signaling and Skeletal Muscle Hypertrophy

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Sex-based comparisons of myofibrillar protein synthesis after resistance exercise in the fed state

West, D. W. D., Burd, N. A., Churchward-Venne, T. A., Camera, D. M., Mitchell, C. J., Baker, S. K., Hawley, J. A., Coffey, V. G., & Phillips, S. M. (2012) · Journal of Applied Physiology · doi:10.1152/japplphysiol.00170.2012 · Cited by 143 · see citing works

Activation of mTORC1 signaling and protein synthesis in human muscle following blood flow restriction exercise is inhibited by rapamycin

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Skeletal muscle disuse atrophy is not attenuated by dietary protein supplementation in healthy older men

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Claims for the anabolic effects of growth hormone: a case of the Emperor’s new clothes?

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Selected Contribution: Skeletal muscle focal adhesion kinase, paxillin, and serum response factor are loading dependent

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Physiologic and molecular bases of muscle hypertrophy and atrophy: impact of resistance exercise on human skeletal muscle (protein and exercise dose effects).

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The Translation Regulatory Subunit eIF3f Controls the Kinase-Dependent mTOR Signaling Required for Muscle Differentiation and Hypertrophy in Mouse

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CrossTalk proposal: The dominant mechanism causing disuse muscle atrophy is decreased protein synthesis

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MAFbx/Atrogin-1 Controls the Activity of the Initiation Factor eIF3-f in Skeletal Muscle Atrophy by Targeting Multiple C-terminal Lysines

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Hypercatabolic Syndrome: Molecular Basis and Effects of Nutritional Supplements with Amino Acids

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Focal adhesion kinase is required for IGF-I-mediated growth of skeletal muscle cells via a TSC2/mTOR/S6K1-associated pathway

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Mechanical overload and skeletal muscle fiber hyperplasia: a meta-analysis

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Resistance Exercise Increases Postprandial Muscle Protein Synthesis in Humans

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Chapter 2 Cell Signaling in Protein Synthesis

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Translational Control Mechanisms in Long-lasting Synaptic Plasticity and Memory

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A Limited Role for PI(3,4,5)P3 Regulation in Controlling Skeletal Muscle Mass in Response to Resistance Exercise

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CrossTalk opposing view: The dominant mechanism causing disuse muscle atrophy is proteolysis

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