Cited by 1,546Schizophrenia research and treatmentMental Health and PsychiatryHallucinations in medical conditions

A cognitive model of the positive symptoms of psychosis

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GARETY, P. A., KUIPERS, E., FOWLER, D., FREEMAN, D., BEBBINGTON, P. E.·2001·Psychological Medicine

Abstract

In the last 10 years a consensus has developed that the symptoms of psychosis may be better understood by linking the steps between the phenomenological experiences and social, psychological and neurobiological levels of explanation. Cognitive models of psychosis are an important link in this chain. They provide a psychological description of the phenomena from which hypotheses concerning causal processes can be derived and tested; social, individual, and neurobiological factors can then be integrated via their impact on these cognitive processes. In this paper, we set out the cognitive processes that we think lead to the formation and maintenance of the positive symptoms of psychosis and we attempt to integrate into our model research in social factors. If this model proves useful, a fuller integration with the findings of biological research will be required (Frith, 1992).

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Crossref · 10.1017/s0033291701003312

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  • Stress-vulnerability and integrated cognitive models posit a role for social, environmental and psychological factors in the aetiology of psychosis, in addition to genetic and neurodevelopmental features such as a family history of psychosis and low IQ.

    “Stress-vulnerability and integrated cognitive models 21,22 posit a role for social, environmental and psychological factors in the aetiology of psychosis, in addition to genetic and neurodevelopmental features such as a family history of psychosis and low intelligence quotient (IQ).”

    From Clinical, socio‐demographic and psychological characteristics in individuals with persistent psychotic experiences with and without a “need for care” (2016) · cites this paper as [21]

    Split into 2 atoms — 1 confirmed, 1 partially supported, 0 disputed, 0 misattributed, 0 unverifiable. Expand the claim to see per-atom verdicts.

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  • The study's results support biopsychosocial models that emphasize the importance of environmental and psychological factors in the aetiology of psychosis and need for care.

    “These results support biopsychosocial models 21,22,37 that emphasize the importance of environmental and psychological factors in the aetiology of psychosis and need for care.”

    From Clinical, socio‐demographic and psychological characteristics in individuals with persistent psychotic experiences with and without a “need for care” (2016) · cites this paper as [21]

    The Introduction (page unavailable) integrates social, psychological and neurobiological explanations of psychotic symptoms, and ‘A cognitive model of psychosis’ describes how adverse experiences may create cognitive vulnerability. The paper also discusses treatment outcomes, but the cited passages do not establish a role for these factors in the need for care. (LLM, conf=0.84)

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  • Persistent psychotic experiences only become problematic in the context of pre-existent vulnerabilities.

    “it is likely that persistent PEs only become problematic in the context of pre-existent vulnerabilities, as suggested by contemporary aetiological models of psychosis 21,22,37”

    From Clinical, socio‐demographic and psychological characteristics in individuals with persistent psychotic experiences with and without a “need for care” (2016) · cites this paper as [21]

    In “A cognitive model of psychosis” (page not available), the paper says psychosis occurs in people with a vulnerable predisposition and suggests that earlier adversity can create enduring cognitive vulnerability. It does not establish that persistent psychotic experiences become problematic only in the presence of pre-existing vulnerabilities; it also discusses appraisal, emotion and social isolation as influences on symptoms and their maintenance. (LLM, conf=0.84)

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  • According to the cognitive model of positive symptoms of psychosis and the Bio-Psychosocial Model of Transition to Psychosis, avoidance and social withdrawal, as well as psychosocial stress and interpersonal sensitivity, contribute to the onset and maintenance of psychosis symptoms.

    “According to the cognitive model of positive symptoms of psychosis (Garety et al. 2001) and the Bio‐Psychosocial Model of Transition to Psychosis (Georgiades et al. 2023), avoidance and social withdrawal, as well as psychosocial stress and interpersonal sensitivity, contribute to the onset and maintenance of psychosis symptoms.”

    From Social Functioning Interventions in Psychosis: A Systematic Review. (2025) · cites this paper as Garety et al. 2001

    In “A cognitive model of psychosis” (page unavailable), the paper links onset to periods of isolation and suggests that social isolation can maintain reasoning biases associated with symptoms. In “Dysfunctional schemas and adverse social environments” (page unavailable), it also links chronic stress to vulnerability and relapse, and safety behaviours to the maintenance of psychotic beliefs. It does not clearly identify interpersonal sensitivity as a contributor to symptom onset or maintenance, so that part of the joint claim is not supported by this source. (LLM, conf=0.83)

    Experimental — accuracy not yet measured
  • Cognitive models of psychosis emphasise the role of emotional processes, particularly anxiety, in the onset and maintenance cycle of psychotic symptoms.

    “Likewise, cognitive models of psychosis [23] emphasise the role of emotional processes, particularly anxiety, in the onset and maintenance cycle of psychotic symptoms [5, 24].”

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Cites

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2 of the 46 references couldn’t be matched to a known paper.

Research works printed but not linked: 11

  1. Birchwood, M. & Iqbal, Z. (1998). Depression and suicidal thinking in psychosis : a cognitive approach. In Outcome and Innovation in Psychological Treatment of Schizophrenia (ed. T. Wykes, N. Tarrier & S. Lewis), pp. 81-100. Wiley : Chichester.
  2. David, A. S. (1998). The clinical importance of insight. In Insight and Psychosis (ed. X. F. Amador and A. S. David), pp. 332-351. OUP : New York.
  3. Fowler, D., Garety, P. & Kuipers, E. (1998). Understanding the inexplicable : an individually formulated cognitive approach to delusional beliefs. In Cognitive Psychotherapy of Psychotic and Personality Disorders (ed. C. Perris and P. D. McGorry), pp. 129-146. Wiley : Chichester.
  4. Freeman, D., Garety, P., Fowler, D., Kuipers, E., Dunn, G., Bebbington, P. & Hadley, C. (1998). The London-East Anglia randomised controlled trial of cognitive behaviour therapy for psychosis IV : self-esteem & persecutory delusions. British Journal of Clinical Psychology 37, 415-430.
  5. Jaspers, K. (1913). General Psychopathology (translated by J. Hoenig and M. W. Hamilton, 1959). Manchester University Press : Manchester.
  6. Kuipers, E., Fowler, D., Garety, P. A., Chisholm, D., Freeman, D., Dunn, G., Bebbington, P. E. & Hadley, C. (1998). The London- East Anglia Randomised Controlled Trial of Cognitive Behaviour Therapy for Psychosis III : follow-up and economic evaluation at 18 months. British Journal of Psychiatry 173, 61-68.
  7. Maher, B. A. (1988). Anomalous experience and delusional thinking : the logic of explanations. In Delusional Beliefs (ed. T. F. Oltmanns and B. A. Maher), pp. 15-33. Wiley : New York.
  8. Mueser, K. T., Goodman, L. B., Trumbetta, S. L., Rosenberg, S. D., Osher, F. C., Vidaver, R., Auciello, P. & Foy, D. W. (1998). Trauma and post traumatic stress disorder in psychosis. Journal of Consulting and Clinical Psychology 66, 493-499.
  9. Peters, E. R., Joseph, S. A. & Garety, P. A. (1999). Measurement of delusional ideation in the normal population : introducing the PDI. Schizophrenia Bulletin 25, 553-576.
  10. Pharoah, F. M., Mari, J. J. & Streiner, D. (1999). Family intervention for schizophrenia (Cochrane Review). Cochrane Library, Issue 2. Update Software : Oxford.
  11. Tarrier, N., Wittkowski, A., Kinney, C., McCarthy, E., Morris, J. & Humphreys, L. (1999). Durability of the effects of cognitive- behavioural therapy in the treatment of chronic schizophrenia. British Journal of Psychiatry 174, 500-504.

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Authors
GARETY, P. A., KUIPERS, E., FOWLER, D., FREEMAN, D., & BEBBINGTON, P. E.
Affiliations
University of East Anglia (GB)
Venue
Psychological Medicine
Published
2001-02
Volume
31
Issue
2
Pages
189-195
ISSN
  • 0033-2917 (ISSN-L)
  • 1469-8978
OpenAlex
W2162176680
Cited by
1,546
References
46
Citations by year
2026: 62 · 2025: 68 · 2024: 48 · 2023: 66 · 2022: 62 · 2021: 77 · 2020: 72 · 2019: 77 · 2018: 76 · 2017: 87 · 2016: 67 · 2015: 74 · 2014: 86 · 2013: 90 · 2012: 65
FWCI
16.11
Type
journal article
Language
en
Open access
Locations
  • Psychological Medicine (publishedVersion)
  • PubMed (publishedVersion)
  • UCL Discovery (University College London) (submittedVersion)
  • Research Portal (King's College London) (submittedVersion)
  • UEA Digital Repository (University of East Anglia) (submittedVersion)
Topics
  • Health Sciences › Medicine › Psychiatry and Mental health › Schizophrenia research and treatment
  • Social Sciences › Arts and Humanities › Philosophy › Mental Health and Psychiatry
  • Life Sciences › Neuroscience › Cognitive Neuroscience › Hallucinations in medical conditions
Keywords
psychosis, positive symptoms, cognitive models, cognitive processes, symptom maintenance, social factors, neurobiological factors, levels of explanation, phenomenology
MeSH terms
Cognition Disorders, Humans, Psychological Theory, Psychotic Disorders, Self Concept, Social Environment

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