Cited by 427Endometriosis Research and TreatmentGynecological conditions and treatmentsEctopic Pregnancy Diagnosis and Management

The DOI matches its Crossref record; 267 of the 290 references it cites were found in a registry or an index; none of the 83 sources its claims cite is checked for support yet.

Pathogenesis of endometriosis: the genetic/epigenetic theory

Open access

Abstract

OBJECTIVE To study the pathophysiology of endometriosis. DESIGN Overview of observations on endometriosis. SETTING Not applicable. PATIENT(S) None. INTERVENTIONS(S) None. MAIN OUTCOME MEASURE(S) The hypothesis is compatible with all observations. RESULT(S) Endometriosis, endometrium-like tissue outside the uterus, has a variable macroscopic appearance and a poorly understood natural history. It is a hereditary and heterogeneous disease with many biochemical changes in the lesions, which are clonal in origin. It is associated with pain, infertility, adenomyosis, and changes in the junctional zone, placentation, immunology, plasma, peritoneal fluid, and chronic inflammation of the peritoneal cavity. The Sampson hypothesis of implanted endometrial cells following retrograde menstruation, angiogenic spread, lymphogenic spread, or the metaplasia theory cannot explain all observations if metaplasia is defined as cells with reversible changes and an abnormal behavior/morphology due to the abnormal environment. We propose a polygenetic/polyepigenetic mechanism. The set of genetic and epigenetic incidents transmitted at birth could explain the hereditary aspects, the predisposition, and the endometriosis-associated changes in the endometrium, immunology, and placentation. To develop typical, cystic ovarian or deep endometriosis lesions, a variable series of additional transmissible genetic and epigenetic incidents are required to occur in a cell which may vary from endometrial to stem cells. Subtle lesions are viewed as endometrium in a different environment until additional incidents occur. Typical cystic ovarian or deep endometriosis lesions are heterogeneous and represent three different diseases. CONCLUSION(S) The genetic epigenetic theory is compatible with all observations on endometriosis. Implications for treatment and prevention are discussed.

Overview

AI snapshot

Written from the paper’s full text.

TL;DR

Koninckx proposes a polygenetic/polyepigenetic theory in which inherited and acquired genetic/epigenetic incidents explain endometriosis predisposition, heterogeneity, and progression from subtle lesions to typical, cystic, or deep disease. The theory is argued to be compatible with all observations, but it remains speculative and untested.

Key findings

  • Endometriosis is hereditary: risk is 6%–9% higher in first-degree relatives and 15% higher when relatives had severe disease; hereditary factors are estimated to account for 50% of endometriosis. GWAS identified 10 or 15 loci, and a meta-analysis identified five loci regulating sex steroid hormone pathways, five secondary signals, and 19 SNPs robustly associated with endometriosis.
  • The genetic/epigenetic theory holds that genetic and epigenetic incidents transmitted at birth explain predisposition and endometriosis-associated changes in endometrium, immunology, and placentation; additional transmissible incidents in a cell—ranging from endometrial to stem cells—are required for typical, cystic ovarian, or deep lesions. Subtle lesions are viewed as endometrium in a different environment until additional incidents occur.
  • Typical cystic ovarian and deep endometriosis lesions are heterogeneous and represent three different diseases; lesions are clonal in origin. Deep endometriosis lesions are generally unique, larger than 1 cm, with some 20% lymph node involvement; depth distribution shows two populations overlapping at 6 mm, and a >5 mm definition was later suggested.
  • Natural history is described as mostly non-progressive: most subtle lesions do not progress, most typical/cystic/deep lesions are not progressive after diagnosis, and most are not recurrent after surgery. Cystic ovarian endometriosis recurrence after stripping is less than 20% within 6 months; recurrence of deep endometriosis after complete excision is rare.

Methods

  • Design: overview of observations on endometriosis; setting not applicable; no patients and no interventions. Main outcome measure: the hypothesis is compatible with all observations.
  • Data: clinical, histologic, and biochemical observations and research on endometriotic tissues; the paper notes there is no nonhuman animal model with sufficient similarity to human myometrium, junctional zone, endometrium, placentation, and pregnancy disorders.
  • Analysis: narrative synthesis of observations and historical theories—Sampson’s retrograde menstruation/implantation, metaplasia, and the endometriotic disease theory—into a polygenetic/polyepigenetic mechanism, with figures and a table summarizing clinical observations.

Limitations

  • The paper states the theory “will remain a theory until disproven by new observations”; it can only speculate what combination of genetic and epigenetic incidents leads to typical, cystic, deep, or extragenital endometriosis, and redundancy of biologic processes can mask phenotypic effects.
  • The paper states that absence of experimentation limits views to clinical, histologic, and biochemical observations; natural history is unclear, progression of subtle lesions or from typical to cystic/deep lesions has not been observed directly, epidemiology is unclear, and recurrence data are limited. Some cited observations are personal or unpublished.
  • Evident from the text: this is a narrative hypothesis paper with no new primary data, no systematic review or meta-analysis, and no formal experimental test of the proposed mechanism.

Model-written summary. Check it against the paper before relying on it.

Integrity signals

Registry

Crossref · 10.1016/j.fertnstert.2018.10.013

Cites

267 of 267 references verified0 citation errors · 0 not checkable

Statistics

Nothing to recompute.

Verification

Claims the paper makes

0 of 83 cited sources checked · 0 supported · 0 flagged

Show all 37 claims

  • Probably not a substantial claim. 20
  • It may state two separate claims as one. 24
  • The quote only partly states this claim. 1
40 of 40
  • The paper relies on this without support.

    There is no nonhuman animal model with sufficient similarity to the human myometrium, junctional zone, endometrium, placentation, and pregnancy disorders such as preeclampsia.

    “There is no nonhuman animal model with sufficient similarity to the human myometrium, junctional zone (JZ), endometrium, placentation, and pregnancy disorders, such as preeclampsia.”
  • The paper relies on this without support.

    Functional redundancy is a characteristic of many cellular processes.

    “Second, functional redundancy is a characteristic of many cellular processes.”
  • The authors state this as an assumption.

    The original cell can be an endometrial cell, a stem cell, or a bone marrow cell with inherited genetic and epigenetic defects.

    “The original cell can be an endometrial cell, a stem cell, or a bone marrow cell with their inherited genetic and epigenetic defects.”
  • The paper relies on this without support.

    Many words in the endometriosis literature are not clear.

    “Many words in the endometriosis literature are not clear.”
  • The paper relies on this without support.

    The meanings of words often change over time, especially after new clinical and molecular-biologic observations are added to the initial clinical, macroscopic, and microscopic descriptions.

    “This confusion stems from the fact that the meanings of words often change over time, especially after new clinical and molecular-biologic observations are added to the initial clinical, macroscopic, and microscopic descriptions.”
5 of 5

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

Each contradiction between two passages listed here was confirmed by an independent check.

  • Twin-sister claim cites a study of nontwin sisters

    Minor

    The paper cites reference 44 for a claim about age of onset in twin sisters, but reference 44 is explicitly about nontwin sisters.

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Citations

Cites

Flagged first, then by citations

Show all 290 references

23 of the 290 references couldn’t be matched to a known paper.

Research works printed but not linked: 9

  1. Rokitansky, 1860, Über Uterusdrüsen-Neubildung in Uterus- und Ovarial-Sarcomen, Zeitschr Ges Aerzte Wien, 16, 577
  2. Cullen, 1896, Adeno-myoma of the round ligament, Johns Hopkins Hosp Bull, 7, 112
  3. Cullen, 1896, Adenoma-myoma uteri diffusum benignum, J Hopkins Hosp Bull, 6, 133
  4. Cullen, 1919, The distribution of adenomyomata containing uterine mucosa, Am J Obstet Gynecol, 80, 130
  5. Batt, 2007, Mullerianosis, Histol Histopathol, 22, 1161
  6. Sampson, 1924, Benign and malignant endometrial implants in the peritoneal cavity, and their relation to certain ovarian tumors, Surg Gynecol Obstet, 38, 287
  7. Fallon, 1950, Endometriois: a report of 400 cases, R I Med J, 33
  8. Asencio FA, Ribeiro HA, Ribeiro PA, Mario M, Adamyan L, Ussia A, et al. Case reports and systematic review of estrogen independent symptomatic postmenopausal endometriosis. Gynecol Surgery 2018. In press.
  9. Meyer, 1923, Zur frage der Urnieren-genese van Adenomyomen, Zentralbl Gynakol, 15, 577

Cited by

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Items 401–419 of 419

Metadata

Authors
Koninckx, P. R., Ussia, A., Adamyan, L., Wattiez, A., Gomel, V., & Martin, D. C.
Affiliations
  • Università Cattolica del Sacro Cuore (IT)
  • Russian University of Medicine (RU)
  • Latifa Hospital (AE)
  • University of British Columbia (CA)
  • University of Tennessee Health Science Center (US)
Corresponding author
Koninckx, Philippe R.
Venue
Fertility and Sterility
Publisher
Elsevier BV
Published
2019-02
Volume
111
Issue
2
Pages
327-340
ISSN
0015-0282 (ISSN-L)
OpenAlex
W2903882793
Cited by
427
References
290
Citations by year
2026: 35 · 2025: 60 · 2024: 52 · 2023: 80 · 2022: 61 · 2021: 76 · 2020: 44 · 2019: 19
FWCI
5.26
Type
journal article
Language
en
Open access
Locations
  • Fertility and Sterility (publishedVersion)
  • PubMed (publishedVersion)
  • Lirias (KU Leuven) (submittedVersion, other-oa)
Topics
  • Health Sciences › Medicine › Reproductive Medicine › Endometriosis Research and Treatment
  • Health Sciences › Medicine › Obstetrics and Gynecology › Gynecological conditions and treatments
  • Health Sciences › Medicine › Obstetrics and Gynecology › Ectopic Pregnancy Diagnosis and Management
Keywords
endometriosis pathogenesis, genetic theory, epigenetic theory, retrograde menstruation, metaplasia, cystic ovarian lesions, deep infiltrating endometriosis, infertility, adenomyosis, peritonitis, placentation, stem cells
MeSH terms
Animals, Endometriosis, Endometrium, Female, Genetic Markers, Humans, Phenotype, Prognosis, Risk Factors, Genetic Predisposition to Disease, Heredity, Epigenesis, Genetic

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