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Developmental imaging genetics: Linking dopamine function to adolescent behavior
Open accessAbstract
Adolescence is a period of development characterized by numerous neurobiological changes that significantly influence behavior and brain function. Adolescence is of particular interest due to the alarming statistics indicating that mortality rates increase two to three-fold during this time compared to childhood, due largely to a peak in risk-taking behaviors resulting from increased impulsivity and sensation seeking. Furthermore, there exists large unexplained variability in these behaviors that are in part mediated by biological factors. Recent advances in molecular genetics and functional neuroimaging have provided a unique and exciting opportunity to noninvasively study the influence of genetic factors on brain function in humans. While genes do not code for specific behaviors, they do determine the structure and function of proteins that are essential to the neuronal processes that underlie behavior. Therefore, studying the interaction of genotype with measures of brain function over development could shed light on critical time points when biologically mediated individual differences in complex behaviors emerge. Here we review animal and human literature examining the neurobiological basis of adolescent development related to dopamine neurotransmission. Dopamine is of critical importance because of (1) its role in cognitive and affective behaviors, (2) its role in the pathogenesis of major psychopathology, and (3) the protracted development of dopamine signaling pathways over adolescence. We will then focus on current research examining the role of dopamine-related genes on brain function. We propose the use of imaging genetics to examine the influence of genetically mediated dopamine variability on brain function during adolescence, keeping in mind the limitations of this approach.
Overview
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TL;DR
This review argues that developmental imaging genetics can link genetically mediated dopamine variability to adolescent frontostriatal function and incentive-driven behavior. It synthesizes animal and human literature on dopamine maturation and dopamine-related gene variants, while emphasizing that gene effects likely change across development and require longitudinal, adequately powered designs.
Key findings
- Adolescence is marked by increased risk-taking and mortality rates that increase two- to three-fold compared with childhood, alongside peaks in sensation/novelty seeking and immature cognitive control.
- Dopamine signaling is heightened in adolescence via peaks in midbrain DA neuron activity, tonic DA levels, prefrontal innervation, and receptor densities; human post-mortem work finds D1 receptor densities peak around 14–18 years of age.
- Functional dopamine gene variants are associated with frontostriatal function: COMT met allele has approximately one third less enzyme activity and higher synaptic dopamine; DRD4 7-repeat is linked to reduced postsynaptic DA inhibition and increased ventral striatal reward reactivity; DAT1 9-repeat is linked to reduced DAT expression and greater striatal synaptic DA.
- Developmental imaging genetics findings can reverse direction relative to adulthood: DRD2 TaqIA A1 allele is associated with decreased reward reactivity in adolescent ventral/dorsal striatum, whereas in adults it has been associated with increased reward reactivity; DAT1 9-repeat shows reduced prefrontal/striatal activation in adolescents during inhibitory control and reward prediction, opposite to adult associations.
Methods
- Narrative review of animal and human literature on adolescent frontostriatal development, dopamine neurotransmission, and imaging genetics studies of dopamine-related polymorphisms.
- Focuses on functional and structural MRI/fMRI studies of DRD1, DRD2, DRD4, COMT, and DAT1, plus gene-gene interaction and multilocus composite-score approaches.
- No primary sample or new empirical analysis is reported; the paper proposes developmental imaging genetics as a future research framework.
Limitations
- The paper states that imaging genetics mainly studies common high-frequency alleles with small effects, cannot establish causal genetic links, and requires well-defined phenotypes, larger samples, multimodal measures, and translational animal models.
- It notes that many dopamine developmental findings are inconsistent, largely from non-human animal models, and have not been directly mapped onto adolescent behavior; initial developmental COMT and DAT1 studies are mixed and need replication.
- Evident from the text: this is a narrative review rather than a systematic review or meta-analysis, and it repeatedly identifies limited developmental imaging genetics studies and absent age-by-genotype tests.
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Claims the paper makes
87 Not checked yet · 22 Own interpretation · 5 No basis given · 2 Partly supported · accuracy not yet measured
None of the sources cited for this claim have been checked yet.
In the paper
“many of the classic risk-taking behaviors observed in adolescence are often in the context of highly emotive and/or reward-seeking states (Casey, Getz et al. 2008; Blakemore and Robbins 2012)”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Introduction
What the sources say
plaincorrelational- Asserted: passes — The authors put it forward as true.
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In the paper
“with continued maturation in the brain systems that underlie both (Casey, Getz et al. 2008; Ernst and Fudge 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
plain- Asserted: passes — The authors put it forward as true.
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In the paper
“adolescents are biased towards potential rewards (Steinberg 2004)”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
- Not checked yetcites Steinberg 2004 source
plain- Asserted: passes — The authors put it forward as true.
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In the paper
“display immature cognitive control (Yurgelun-Todd 2007)”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
- Not checked yetcites Yurgelun-Todd 2007 source
plain- Asserted: passes — The authors put it forward as true.
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In the paper
“Frontostriatal circuits subserving affective, cognitive, and motor processes are significantly modulated by the neurotransmitter dopamine (DA) (for reviews see (Schultz 2002; Wise 2004; Cools 2008), through facilitation of the direct pathway via the action of excitatory DA receptors (D1-like) and inhibition of the indirect pathway via the action of inhibitory DA receptors (D2-like).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
plaindirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
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A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“These changes alter the excitatory-inhibitory balance in neuronal signaling that refine controlled processing into adulthood.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
plaindirect- Asserted: passes — The authors put it forward as true.
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A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Taken together, the current literature highlights that immaturities in the function of and integration between frontal and striatal regions at multiple levels of organization contribute to a distinct adolescent brain (and subsequently behavioral) phenotype.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
plaindirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: passes — The paper’s conclusions depend on it.
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In the paper
“In human post-mortem studies, DA levels in the striatum increase until adolescence and then decrease or remain the same (Haycock, Becker et al. 2003).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Haycock, Becker et al. 2003
plaindirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
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In the paper
“A post mortem human research study found that D1 receptor densities peak around 14–18 years of age (Weickert, Webster et al. 2007), declining thereafter.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Weickert, Webster et al. 2007 source
plaindirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
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In the paper
“Non-human primate studies show that the highest concentrations of DA during adolescence are in the PFC before dropping down in adulthood (Goldman-Rakic and Brown 1982).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Goldman-Rakic and Brown 1982
plaincorrelational- Asserted: passes — The authors put it forward as true.
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- It may state two separate claims as one. 55
- Probably not a substantial claim. 29
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In the paper
“functional connectivity studies suggest that the integration and coordination between brain regions, including subcortical to cortical connections, become more refined and efficient over adolescence, leading to reduced task-irrelevant connections, strengthening of connections supporting goal-directed actions, and elimination of redundant connections (Durston, Davidson et al. 2006; Liston, Watts et”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
- Not checked yetcites Durston, Davidson et al. 2006 source
- Partly supportedcites Liston, Watts et al. 2006 sourceExperimental — accuracy not yet measured
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- Partly supportedcites Fair, Cohen et al. 2009 sourceExperimental — accuracy not yet measured
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hedgeddirect- Asserted: passes — The authors put it forward as true.
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It may state two separate claims as one.
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In the paper
“Adding to this complexity, maturational changes in DA function have not been mapped directly onto behaviors in adolescence suggesting that a comprehensive examination of the interaction of various aspects of the DA system (e.g. receptors, clearance, innervation) and their direct effects on behavior is warranted (Spear 2011; Luciana, Wahlstrom et al. 2012).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Partly supportedcites Spear 2011 sourceExperimental — accuracy not yet measured
Split into 2 atoms — 0 confirmed, 2 partially supported, 0 disputed, 0 misattributed, 0 unverifiable. Expand the claim to see per-atom verdicts.
- Not checked yetcites Luciana, Wahlstrom et al. 2012
hedged- Asserted: passes — The authors put it forward as true.
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Probably not a substantial claim. It may state two separate claims as one.
- Partly supported
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In the paper
“adolescence is often a time when various mental illnesses emerge such as mood disorders, drug abuse disorders, eating disorders, and psychoses (Pine 2002; Chambers, Taylor et al. 2003; Sisk and Zehr 2005; Paus, Keshavan et al. 2008)”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Introduction
What the sources say
plaincorrelational- Asserted: passes — The authors put it forward as true.
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In the paper
“adolescents are capable of mature decision-making (Paus 2005), abstract thinking, and often engage rational behavior (Steinberg, Cauffman et al. 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Introduction
What the sources say
plain- Asserted: passes — The authors put it forward as true.
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It may state two separate claims as one.
In the paper
“Across species and cultures, there are characteristic behaviors during adolescence including peaks in sensation/novelty seeking coupled with diminished levels of harm avoidance, leading to an increase in risky behaviors (Laviola, Macri et al. 2003).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Introduction
What the sources say
- Not checked yetcites Laviola, Macri et al. 2003
plaindirect- Asserted: passes — The authors put it forward as true.
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In the paper
“Evidence suggests that adolescents tend to both process incentives differently than adults (for reviews see: Geier and Luna (2009; Ernst, Daniele et al. 2011)), leading to suboptimal and often risky decision-making.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
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It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“neurobiological models of adolescent development suggest that an over active adolescent incentive system, driven by the striatum, with a still maturing cognitive system, driven by the PFC, may create a functional imbalance in optimal behavioral regulation (i.e. suppressing a potentially rewarding, but inappropriate behavior) thereby enhancing risk taking behavior in adolescence ((Nelson, Leibenlu”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
hedgedconditional- Asserted: passes — The authors put it forward as true.
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In the paper
“This approach, known as ‘imaging genetics’, is grounded in the idea that brain function and structure can serve as intermediate phenotypes between genes and behavior, given the relative proximity of brain function to the genotype (Hariri and Weinberger 2003).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Introduction
What the sources say
- Not checked yetcites Hariri and Weinberger 2003
plaincorrelational- Asserted: passes — The authors put it forward as true.
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Probably not a substantial claim.
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In the paper
“The human striatum is recognized as a core node for incentive processing and resulting behaviors, specifically in the ability to synthesize changing environmental cues and appropriately update behaviors through integration with the prefrontal cortex (PFC) by way of overlapping but functionally segregated pathways (Alexander, DeLong et al. 1986; Postuma and Dagher 2006; Di Martino, Scheres et al. 2”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
plaindirect- Asserted: passes — The authors put it forward as true.
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In the paper
“functional neuroimaging studies of incentive processing demonstrate differential striatal and PFC activation in adolescence relative to adulthood (Bjork, Knutson et al. 2004; Ernst, Nelson et al. 2005; Galvan, Hare et al. 2006; Bjork, Smith et al. 2010; van Leijenhorst, Moor et al. 2010; Padmanabhan 2011), with the majority of studies reporting an increase in striatal activation, coupled with”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
boostedcorrelational- Asserted: passes — The authors put it forward as true.
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In the paper
“Slow changes in tonic levels of DA may be a preparatory mechanism for an organism to respond to environmental cues associated with reward (Schultz 1998).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Schultz 1998 source
hedgedconditional- Asserted: passes — The authors put it forward as true.
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- Load-bearing: fails — The paper’s conclusions may not depend on it.
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Probably not a substantial claim. The paper’s conclusions may not depend on it.
In the paper
“These two mechanisms of DA signaling have been found to lead to distinct behaviors (Floresco, West et al. 2003) and are regulated by reuptake and degradation enzymes.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Floresco, West et al. 2003
plaindirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
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It may state two separate claims as one.
In the paper
“Fast phasic events occur in response to reward-related events, which may serve as important teaching signals for error detection and modulate behavioral changes in response to the environment (Schultz 1998).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Schultz 1998 source
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
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Probably not a substantial claim. The paper’s conclusions may not depend on it. It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Animal and post-mortem human literature suggests an overexpression of receptors for serotonin, dopamine, adenergic, and endocannabinoids (Lidow and Rakic 1992), a peak in the density of interneurons (Anderson, Classey et al. 1995; Lewis 1997; Erickson and Lewis 2002), and an increase in levels of GABA (Hedner, Iversen et al. 1984).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
hedged- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“DA neurons in the midbrain project to medium spiny neurons in the NAcc as well as pyramidal neurons in the PFC, thereby modulating the firing rates of these neurons and establishing a strong reciprocal relationship between striatum and PFC (Grace, Floresco et al. 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Grace, Floresco et al. 2007 source
boosteddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“DA levels are modulated by two dissociable processes of DA discharge that interact, (1) a constant background tonicity regulated by baseline firing of DA neurons and glutamatergic afferents from cortical to striatal regions, and (2) a burst firing high-amplitude phasic release (Grace, Floresco et al. 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Grace, Floresco et al. 2007 source
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
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It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“increased myelination in cortical to subcortical axons, changes in axon caliber, pruning of synapses and receptors, cell shrinkage, and glial changes (Yakovlev and Lecours 1967; Rakic, Bourgeois et al. 1986; Benes, Turtle et al. 1994; Andersen 2003) refine the developing brain and strengthen and consolidate highly used connections, while weakening or eliminating redundant or weakly used”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Incentive driven behaviors and frontostriatal circuits in adolescence
What the sources say
- Not checked yetcites Yakovlev and Lecours 1967
- Not checked yetcites Rakic, Bourgeois et al. 1986 source
- Not checked yetcites Benes, Turtle et al. 1994
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Research on DA transporters has been inconsistent in the midbrain suggesting no consistent developmental change (Moll, Mehnert et al. 2000), increases over adolescence (Galineau, Kodas et al. 2004), and peaks in late childhood (Coulter, Happe et al. 1996).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Moll, Mehnert et al. 2000 source
- Not checked yetcites Galineau, Kodas et al. 2004
- Not checked yetcites Coulter, Happe et al. 1996
hedgeddirect- Asserted: passes — The authors put it forward as true.
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- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“A peak in activity of midbrain DA neurons has been documented in the rat model (McCutcheon, White et al. 2009), suggesting an overall increase in DA levels.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites McCutcheon, White et al. 2009
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“These systems also interact as tonic DA activity regulates phasic signaling in an inhibitory fashion and phasic DA has been shown to enhance tonic activity (Niv, Daw et al. 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Niv, Daw et al. 2007 source
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Other studies have noted a peak in tonic DA concentrations in late adolescence with a subsequent decline in adulthood ((Badanich, Adler et al. 2006; Philpot, Wecker et al. 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Badanich, Adler et al. 2006 source
- Not checked yetcites Philpot, Wecker et al. 2009
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it.
None of the sources cited for this claim have been checked yet.
In the paper
“Dopaminergic innervation to the PFC peaks in adolescence (Rosenberg and Lewis 1995; Benes, Taylor et al. 2000), with the largest increase being in cortical layer III, a region that that is highly implicated in cognitive processing (Lewis and Gonzalez-Burgos 2000).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Rosenberg and Lewis 1995 source
- Not checked yetcites Benes, Taylor et al. 2000
- Not checked yetcites Lewis and Gonzalez-Burgos 2000
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“In the striatum, peaks in both D1 and D2 receptors occur in childhood and begin to decline in adolescence, evident in both animal and human work (Seeman, Bzowej et al. 1987; Lidow and Rakic 1992; Montague, Lawler et al. 1999; Andersen, Thompson et al. 2002).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“non-human primate research suggests that the density of D1 and D2 receptors in PFC increase at different rates, with D1 receptor density demonstrating earlier peaks than D2, which peaks in late-adolescence/early adulthood (Tseng and O'Donnell 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Tseng and O'Donnell 2007
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“abnormalities in DA signaling are implicated in the pathophysiology of neuropsychiatric disorders that often emerge in adolescence (e.g. schizophrenia, drug abuse).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds.
- Rests on an assumption: The risk factors for mental illnesses that emerge in adolescence are not fully characterized.
None of the sources cited for this claim have been checked yet.
In the paper
“Adolescents also show decreased aversive response to substances of abuse (i.e. milder withdrawal responses, reduced psychomotor effects) (Spear 2002; Doremus, Brunell et al. 2003; Levin, Rezvani et al. 2003) and increased sensitivity to DA receptor antagonists (Spear, Shalaby et al. 1980; Spear and Brake 1983; Teicher, Barber et al. 1993).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Spear 2002
- Not checked yetcites Doremus, Brunell et al. 2003
- Not checked yetcites Levin, Rezvani et al. 2003 source
plain- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“DA signaling in adolescence may also influence and be influenced by differences in rates of maturation of subcortical systems relative to cortical, and an functional imbalance in the adolescent brain that is driven by striatal signaling with immaturities in PFC-driven regulation (Chambers, Taylor et al. 2003; Ernst, Pine et al. 2006).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“The adult literature suggests that increasing DA signaling through administration of DA or DA agonists increases novelty-seeking and exploration behaviors, whereas reducing DA signaling with antagonists halts such behaviors (Pijnenburg, Honig et al. 1976; Fouriezos, Hansson et al. 1978; Le Moal and Simon 1991).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Pijnenburg, Honig et al. 1976
- Not checked yetcites Fouriezos, Hansson et al. 1978
- Not checked yetcites Le Moal and Simon 1991
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Other research has suggested that in the striatum, DA transporter levels increase into late childhood and remain stable through adolescence (Coulter, Happe et al. 1996; Tarazi, Tomasini et al. 1998; Galineau, Kodas et al. 2004).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Coulter, Happe et al. 1996
- Not checked yetcites Tarazi, Tomasini et al. 1998
- Not checked yetcites Galineau, Kodas et al. 2004
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it. It may state two separate claims as one.
In the paper
“It is posited that the DA system is at a “functional ceiling” in adolescence relative to childhood or adulthood (Chambers, Taylor et al. 2003), due to peaks in midbrain DA cell firing, overall tonic levels, innervation, as well as increased receptor densities.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Chambers, Taylor et al. 2003 source
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“These early findings point to a hypothesized model of adolescent DA function whereby overall heightened DA signaling leads to heightened motivation, or approach-like behaviors-due to increased activation of the direct pathway and inhibition of the indirect pathway.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
hedgeddirect- Asserted: fails — The authors may not put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The authors may not put it forward as true.
In the paper
“For example, the elevation of tonic DA during adolescence may impact regulation of the phasic response in response to salient or rewarding information (for review see (Luciana, Wahlstrom et al. 2012)), but this has not been empirically tested.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Luciana, Wahlstrom et al. 2012
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it. It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Increases in both D1 and D2 receptors, as seen in adolescence thus may have an overall excitatory effect on the brain, which could result in an increase in behaviors that are DA dependent (such as reward and novelty seeking).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: unclear — It is unclear why the paper says it holds.
It may state two separate claims as one.
- Rests on an assumption: D1 receptors are more abundant in the direct pathway, exciting GABAergic neurons in response to preferred behaviors, and D2 in the indirect pathway, which inhibit GABAergic neurons and reduce the inhibitory effect of the indirect pathway.
None of the sources cited for this claim have been checked yet.
In the paper
“For example, increasing DA levels in individuals who have high baseline DA levels impairs cognitive performance, (perhaps pushing them over the peak of the inverted U curve) whereas improvements are noted in individuals with lower baseline levels (pushing them closer to the apex of the curve) (Mattay, Goldberg et al. 2003; Apud, Mattay et al. 2007; Cools, Frank et al. 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Mattay, Goldberg et al. 2003
- Not checked yetcites Apud, Mattay et al. 2007 source
- Not checked yetcites Cools, Frank et al. 2009
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“D1 receptors stimulation allows for maintenance of information online and stabilization of functional states, and D2 receptor binding is involved in flexible updating of information and allowing for the transition between functional states (Seamans, Durstewitz et al. 2001; Durstewitz and Seamans 2002; Seamans and Yang 2004).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Seamans, Durstewitz et al. 2001
- Not checked yetcites Durstewitz and Seamans 2002
- Not checked yetcites Seamans and Yang 2004 source
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Indeed, the study of the influence of genetic polymorphisms on brain function or “imaging genetics” has already provided considerable insight on the influence of genetically driven variability on brain physiology (e.g. (Hariri and Weinberger 2003; Brown and Hariri 2006; Drabant, Hariri et al. 2006; Hariri and Lewis 2006)).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Developmental Imaging Genetics
What the sources say
- Not checked yetcites Hariri and Weinberger 2003
- Not checked yetcites Brown and Hariri 2006
- Not checked yetcites Drabant, Hariri et al. 2006
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Following this model, it is possible that baseline inter-individual differences in adolescence would be unique relative to differences in adulthood due to maturation in the DA system.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds.
None of the sources cited for this claim have been checked yet.
In the paper
“Despite an overall peak in DA signaling and general maturational processes that occur in adolescence, there is considerable individual variability both in DA signaling, as well as DA-influenced behaviors, likely due to a combination of genetic and environmental factors (Depue and Collins 1999; Frank and Hutchison 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Dopamine
What the sources say
- Not checked yetcites Depue and Collins 1999 source
- Not checked yetcites Frank and Hutchison 2009
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“AG heterozygotes, who have increased D1 receptors, showed increased recruitment of DLPFC relative to AA homozygotes, who engaged a more widely distributed set of brain regions.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Tura, Turner et al. 2008
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“The D2 receptor, which is expressed more abundantly in striatum relative to PFC, exerts a strong influence on frontostriatal connectivity through both inhibition of excitatory and disinhib tion of inhibitory pathways (Cepeda and Levine 1998; Goto and Grace 2005).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Cepeda and Levine 1998
- Not checked yetcites Goto and Grace 2005
boosteddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“These findings are in line with other work suggesting that increased prefrontal DA tone results in improved cognitive performance and more efficient prefrontal signaling (e.g. (Egan, Goldberg et al. 2001; Mattay, Goldberg et al. 2003)).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Egan, Goldberg et al. 2001
- Not checked yetcites Mattay, Goldberg et al. 2003
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it. It may state two separate claims as one.
In the paper
“Simultaneously, D1 receptor activation on local GABAergic (inhibitory) interneurons serves to inhibit irrelevant glutamatergic inputs (Durstewitz, Seamans et al. 2000).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Durstewitz, Seamans et al. 2000
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it.
None of the sources cited for this claim have been checked yet.
In the paper
“In adolescence, when there is a higher density of D2 receptors, the relationship between brain activation and D2 receptor availability might parallel previous findings using pharmacological interventions that target D2 receptors (Kirsch, Reuter et al. 2006; van der Schaaf, van Schouwenburg et al. 2012), suggesting an age by genotype interaction that is yet to be empirically tested.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Kirsch, Reuter et al. 2006
- Not checked yetcites van der Schaaf, van Schouwenburg et al. 2012
hedgedcorrelational- Asserted: fails — The authors may not put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The authors may not put it forward as true. The paper’s conclusions may not depend on it. It may state two separate claims as one.
In the paper
“D2 receptors have two distinct isoforms, the short isoform (D2-S) acts mainly as a presynaptic autoreceptor, inhibiting DA release, whereas the long isoform (D2-L) primarily functions to inhibit the post synaptic cell (Centonze, Grande et al. 2003).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Centonze, Grande et al. 2003
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Decreased D2 autoreceptor function increases DA release and individuals with decreased D2-S demonstrate increased novelty-seeking and reward reactivity (Zald, Cowan et al. 2008; Pecina, Mickey et al. 2012).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Zald, Cowan et al. 2008
- Not checked yetcites Pecina, Mickey et al. 2012
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“It is thus possible that inter-individual differences are expressed differentially as a function of relative DA across development based on genotype (e.g. the val allele may confer a relative advantage for cognitive function earlier in development, when DA levels are higher than in adulthood).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds.
None of the sources cited for this claim have been checked yet.
In the paper
“Furthermore, increasing DA levels interacts with the COMT val158met SNP consistent with the putative inverted U model with met individuals demonstrating diminished cortical efficiency during tasks of cognitive control and val individuals demonstrating improvements (Mattay, Goldberg et al. 2003; Apud, Mattay et al. 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
What the sources say
- Not checked yetcites Mattay, Goldberg et al. 2003
- Not checked yetcites Apud, Mattay et al. 2007 source
hedgeddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“This is adolescents carrying the met allele may surpass optimal thresholds, which could result in less efficient cortical function, relative to val (Wahlstrom, Collins et al. 2010; Wahlstrom, White et al. 2010).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
What the sources say
- Not checked yetcites Wahlstrom, Collins et al. 2010
- Not checked yetcites Wahlstrom, White et al. 2010
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“The DA transporter (DAT) is mainly expressed in the striatum and is responsible for DA reuptake, clearing DA from the extracellular space after release (Jaber, Bloch et al. 1998).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
What the sources say
- Not checked yetcites Jaber, Bloch et al. 1998
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“The DAT binding site density for the most common repeat alleles (9-repeat and 10-repeat) is significantly less for the 9-repeat allele than the 10-repeat allele, linking the 9-repeat allele with reduced DAT expression and greater striatal synaptic DA (Fuke, Suo et al. 2001; Mill, Asherson et al. 2002; VanNess, Owens et al. 2005), although some studies have suggested the opposite (Mill, Asherson et”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
What the sources say
- Not checked yetcites Fuke, Suo et al. 2001
- Not checked yetcites Mill, Asherson et al. 2002 source
- Not checked yetcites VanNess, Owens et al. 2005
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
The paper states this without a source or data behind it here, so there is nothing to check it against. The quote shows it is in the paper.
In the paper
“However, no studies to date have examined the influence of these polymorphisms across development.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
plain- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds.
In the paper
“The COMT val allele is associated with high enzymatic activity and consequently low synaptic dopamine levels, whereas the COMT met allele results in approximately one third less enzyme activity and consequently high synaptic dopamine (Chen, Lipska et al. 2004).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
What the sources say
- Not checked yetcites Chen, Lipska et al. 2004 source
plaincorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
In the paper
“During a visuo-spatial working memory task in individuals between the ages of 6 and 20,Dumontheil et al. (2011), demonstrated that activation in frontal and parietal regions increased across development in individuals homozygous for the val allele, but not met carriers, suggesting delayed development of cognitive function in individuals with the val allele.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
What the sources say
- Not checked yetcites Dumontheil et al. (2011) source
boosteddirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Following the inverted U model, increased D1 and D2 receptor availability may result in increased competition between the direct and indirect pathways which may be more exacerbated in adolescents with higher receptor availability at baseline, leading to a generally more disorganized processing system.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it. The paper may give no reason why it holds. It may state two separate claims as one.
- Rests on an assumption: D1 and D2 receptor densities peak in late childhood, so receptor density is higher in adolescence than in adulthood.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Based on this evidence, it is posited that adolescents, who have increased DA levels relative to adults, may follow a similar pattern as a function of COMT genotype as the pharmacological studies in adults.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the COMT Gene
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim.
None of the sources cited for this claim have been checked yet.
In the paper
“A SNP in the DRD4 gene (rs6277, −521 SNP) results in a 40% reduction in RNA transcription for the T-allele relative to the C-allele (Okuyama, Ishiguro et al. 1999), although another study found no differences (Kereszturi, Kiraly et al. 2006).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA Receptor Genes (DRD1, DRD2, and DRD4)
What the sources say
- Not checked yetcites Okuyama, Ishiguro et al. 1999
- Not checked yetcites Kereszturi, Kiraly et al. 2006
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Lower DAT expression reduces synaptic DA clearance thereby increasing DA levels (Cagniard, Balsam et al. 2006; Cagniard, Beeler et al. 2006).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
What the sources say
- Not checked yetcites Cagniard, Balsam et al. 2006
- Not checked yetcites Cagniard, Beeler et al. 2006
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“Developmental studies using the DAT1 polymorphism suggest that typically developing adolescents with the 9-repeat allele demonstrate reduced activation of prefrontal and striatal regions during inhibitory control (Braet, Johnson et al. 2011), and reward prediction (Paloyelis, Mehta et al. 2012).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
What the sources say
- Not checked yetcites Braet, Johnson et al. 2011
- Not checked yetcites Paloyelis, Mehta et al. 2012
hedgedcorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
None of the sources cited for this claim have been checked yet.
In the paper
“For example, prior studies have shown additive effects of the COMT val158met SNP and the DAT1 3’VNTR during the reward anticipation and outcome stages of reward processing in both PFC and striatum, reporting increased activation associated with genotypes that have increased DA availability (Yacubian, Sommer et al. 2007; Dreher, Kohn et al. 2009).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Gene-Gene Interactions
What the sources say
- Not checked yetcites Yacubian, Sommer et al. 2007 source
- Not checked yetcites Dreher, Kohn et al. 2009
boostedcorrelational- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: unclear — It is unclear whether the paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
The paper states this without a source or data behind it here, so there is nothing to check it against. The quote shows it is in the paper.
In the paper
“However, it is also important to keep in mind that meta-analyses tend to be biased, as studies with null findings are generally not published.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it. The paper may give no reason why it holds.
In the paper
“For example,Munafo et al. (2008) conducted a meta-analysis of studies that have reported associations between a VNTR polymorphism in the serotonin transporter gene (5-HTTLPR) and amygdala activation and suggested that an imaging genetics study would require a total sample of about 70 participants to achieve .8 power for an alpha power of .05.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
What the sources say
- Not checked yetcites Munafo et al. (2008) source
hedgedcorrelationaln = 70- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim.
The paper states this without a source or data behind it here, so there is nothing to check it against. The quote shows it is in the paper.
In the paper
“Evidence points to a protracted development of brain systems including PFC and the striatum throughout childhood and adolescence.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
plain- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds.
The paper states this without a source or data behind it here, so there is nothing to check it against. The quote shows it is in the paper.
In the paper
“The majority of imaging genetics research to date has focused on differences between genotypes in adulthood or within discrete age groups, despite growing evidence that brain systems continue to reorganize across the lifespan and that gene effects likely manifest differently at different stages.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
plain- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. The paper may give no reason why it holds. It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“It is likely that sample sizes will have to be increased in order to replicate previous findings and to generate accurate assessments of the effect sizes of different polymorphisms.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: fails — It may be too vague to check against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. It may be too vague to check against evidence. The paper’s conclusions may not depend on it. It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“These results suggest that DAT1 genotype may influence the system differentially in adolescence – with the 9-repeat allele resulting in decreased striatal and cortical reactivity-than in adulthood – when the 9-repeat allele has been associated with increased activation.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“It is possible that in adolescence, when excess DA levels are present, individuals carrying the 9-repeat allele have an overabundance of synaptic DA availability, which may have opposite effects on brain function than in adulthood.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > DA inactivation genes (COMT, DAT1) > Functional Polymorphism in the DAT1 Gene
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“However, exaggerated DA levels in both striatum and PFC in adolescence may result in an increased sensitivity to rewards coupled with poor executive regulation of impulse driven behaviors, thereby increasing vulnerability for risk-taking behaviors.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: unclear — It is unclear why the paper says it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“The inability to consistently control behavior concurrent with increased sensation seeking persists in adolescence, leading to increases in risk taking behaviors.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: unclear — It is unclear why the paper says it holds.
It may state two separate claims as one.
In the paper
“Similarly, others have suggested that sample sizes of over 25 subjects in each group are necessary for general fMRI studies in order to have adequate reliability (Thirion, Pinel et al. 2007).”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
What the sources say
- Not checked yetcites Thirion, Pinel et al. 2007 source
hedgedn = 25- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Given evidence that various aspects of the DA system are heightened or changed in adolescence and that single gene effects may manifest differently in the adolescent brain, it is also possible that gene interactions differ in the adolescent brain than in the adult brain.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Gene-Gene Interactions
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“DA availability and signaling is heightened during the adolescent period and may promote novelty seeking in an adaptive fashion in order to gain skills that support adult survival.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
hedgedconditional- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: unclear — It is unclear why the paper says it holds.
It may state two separate claims as one.
The paper states this without a source or data behind it here, so there is nothing to check it against. The quote shows it is in the paper.
In the paper
“Gene expression is one of the primary sources of variability, acting through cellular and system-level neural processes to produce complex phenomena that manifest in behavioral function and dysfunction.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Summary/Conclusions
plaindirect- Asserted: passes — The authors put it forward as true.
- Checkable: fails — It may be too vague to check against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: fails — The paper may give no reason why it holds.
Probably not a substantial claim. It may be too vague to check against evidence. The paper may give no reason why it holds.
In the paper
“fMRI is one the most common and reliable methods of measuring brain function at decent spatial and temporal resolutions, but given that it is an indirect measure of brain activity, reflecting a paradigm related change in metabolic consumption (Logothetis, Pauls et al. 2001), interpretation of gene effects is limited.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
What the sources say
- Not checked yetcites Logothetis, Pauls et al. 2001 source
plain- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: passes — The paper’s conclusions depend on it.
- Based: passes — The paper gives a reason why it holds.
It may state two separate claims as one.
A conclusion the paper draws from its own results — it cites no other source for it, so there is none to check. The quote shows it is in the paper.
In the paper
“Assuming a relatively even distribution of the alleles, this would result in approximately 30–35 participants per group.”
§ Developmental imaging genetics: linking dopamine function to adolescent behavior > Considerations and Future Directions for Imaging Genetics studies
hedgedcorrelationaln = 70- Asserted: passes — The authors put it forward as true.
- Checkable: passes — It can be checked against evidence.
- Load-bearing: fails — The paper’s conclusions may not depend on it.
- Based: passes — The paper gives a reason why it holds.
Probably not a substantial claim. The paper’s conclusions may not depend on it.
- The authors state this as an assumption: The alleles are relatively evenly distributed.
The paper relies on this without support.
The risk factors for mental illnesses that emerge in adolescence are not fully characterized.
“the risk factors for which are not fully characterized.”
The authors state this as an assumption.
Each individual is at their own peak in sensation and novelty seeking.
“with the assumption that each individual is at their own peak in sensation and novelty seeking”
The paper relies on this without support.
DA signaling supports reinforcement learning by tuning the strength of synapses, thereby influencing plasticity.
“First, DA signaling supports reinforcement learning as it tunes the strength of synapses, thereby influencing plasticity.”
The paper relies on this without support.
Characterizing the nature of neurochemical systems in human development is challenging, as pharmacological and other invasive procedures (i.e. PET) typically cannot be used to study developing populations.
“Methodologically, characterizing the nature of neurochemical systems in human development is challenging, as pharmacological and other invasive procedures (i.e. PET) typically cannot be used to study developing populations.”
The paper relies on this without support.
Genetic influences on behavior are mediated by changes in cellular and systems levels of functioning in the brain.
“This approach is grounded in the notion that genetic influences on behavior are mediated by changes in cellular and systems levels of functioning in the brain.”
The paper relies on this without support.
D1 receptors are more abundant in the direct pathway, exciting GABAergic neurons in response to preferred behaviors, and D2 in the indirect pathway, which inhibit GABAergic neurons and reduce the inhibitory effect of the indirect pathway.
“D1 receptors are more abundant in the direct pathway, exciting GABAergic neurons in response to preferred behaviors, and D2 in the indirect pathway, which inhibit GABAergic neurons and reduce the inhibitory effect of the indirect pathway.”
The paper relies on this without support.
Limited research has examined polymorphisms of the D1-receptor gene (DRD1) in relation to brain structure/function.
“Limited research has examined polymorphisms of the D1-receptor gene (DRD1) in relation to brain structure/function.”
The paper relies on this without support.
D1 and D2 receptor densities peak in late childhood, so receptor density is higher in adolescence than in adulthood.
“Current research suggests that D1 and D2 receptor densities peak in late childhood, suggesting that receptor density is higher in adolescence relative to adulthood.”
The authors state this as an assumption.
Each polymorphism has equal effect sizes.
“Assuming equal effect sizes of each polymorphism”
The paper relies on this without support.
The genetic basis for complex behavioral traits is likely a result of allelic variation across many genes/polymorphisms and their interactions with each other and the environment.
“The genetic basis for complex behavioral traits is likely a result of allelic variation across many genes/polymorphisms and their interactions with each other and the environment.”
The authors state this as an assumption.
The alleles are relatively evenly distributed.
“Assuming a relatively even distribution of the alleles”
Claims other papers make about this one
Each is checked against this paper’s own text: does it say what the citing paper says it does?
The source backs part of the claim, not all of it — or backs it for a different population, condition or comparison.
In the citing paper
“These changes include increases in synaptic pruning and myelination107,171, as well as increases in the availability of dopamine in the limbic circuitry172,173.”
From Rhythms of life: circadian disruption and brain disorders across the lifespan (2019) · cites this paper as [173]
Experimental — accuracy not yet measuredThe cited source says what the claim attributes to it.
In the citing paper
“Dopamine availability increases during adolescence (11,12)”
From Impact of Sleep and Circadian Rhythms on Addiction Vulnerability in Adolescents (2018) · cites this paper as [12]
In the citing paper
“DA signaling supports reinforcement learning, and DA modulation of striatal and prefrontal function influences affective and motivated behaviors that are altered in adolescence (Padmanabhan and Luna, 2014).”
From Dopaminergic reward sensitivity can promote adolescent health: a new perspective on the mechanism of ventral striatum activation (2016) · cites this paper as Padmanabhan and Luna, 2014
In the citing paper
“The ventral striatum, and the nucleus accumbens in particular, has been recognized as a core node for incentive, reward driven behaviors (see Padmanabhan and Luna, 2014, Galván, 2014).”
From Dopaminergic reward sensitivity can promote adolescent health: a new perspective on the mechanism of ventral striatum activation (2016) · cites this paper as Padmanabhan and Luna, 2014
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Metadata
- Authors
- Padmanabhan, A., & Luna, B.
- Affiliations
- University of Pittsburgh (US)
- Corresponding author
- Padmanabhan, Aarthi
- ORCID
- Padmanabhan, Aarthi
- Venue
- Brain and Cognition
- Publisher
- Elsevier BV
- Published
- 2014-08
- Volume
- 89
- Pages
- 27-38
- ISSN
- 0278-2626 (ISSN-L)
- PMC
- PMC4226044
- OpenAlex
- W2024479703
- Semantic Scholar
- 4d8986c66c17a1fd67454a0f74b5d6b4169b6359
- Cited by
- 87
- References
- 206
- Citations by year
- 2026: 4 · 2025: 4 · 2024: 6 · 2023: 5 · 2022: 12 · 2021: 4 · 2020: 8 · 2019: 6 · 2018: 4 · 2017: 7 · 2016: 12 · 2015: 10 · 2014: 4
- FWCI
- 0.92
- Type
- journal article
- Language
- en
- Open access
- green
- www.ncbi.nlm.nih.gov
- Locations
- Brain and Cognition (publishedVersion)
- PubMed (publishedVersion)
- PubMed Central (acceptedVersion)
- Funders
- National Institute of Mental Health (R01MH080243)
- Topics
- Life Sciences › Neuroscience › Cellular and Molecular Neuroscience › Neurotransmitter Receptor Influence on Behavior
- Life Sciences › Neuroscience › Cognitive Neuroscience › Neural and Behavioral Psychology Studies
- Health Sciences › Medicine › Psychiatry and Mental health › Schizophrenia research and treatment
- Keywords
- imaging genetics, dopamine, adolescent development, dopamine signaling, risk-taking, impulsivity, sensation seeking, individual differences in behavior, psychopathology
- MeSH terms
- Adolescent, Adolescent Behavior, Animals, Brain, Brain Mapping, Dopamine, Genotype, Humans, Magnetic Resonance Imaging, Polymorphism, Genetic, Receptors, Dopamine, Adolescent Development, Genetic Association Studies
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